C. Jones

发表

Loss of retinoic acid receptors in mouse skin and skin tumors is associated with activation of the ras(Ha) oncogene and high risk for premalignant progression.

S. Rutberg, L. D. De Luca, S. Collins, 1996, Cancer research.

Retinoic acid inhibition of cell cycle progression in MCF-7 human breast cancer cells.

L. D. De Luca, A. Kiss, K. Matsukuma, 1997, Experimental cell research.

Dietary retinoic acid inhibits mouse skin carcinogenesis irrespective of age at initiation.

R. Tarone, L. D. De Luca, L. C. Chen, 1996, Nutrition and cancer.

Vitamin A in epithelial differentiation and skin carcinogenesis.

S. Ross, L. D. De Luca, N. Darwiche, 2009, Nutrition reviews.

Retinoic acid increases tyrosine phosphorylation of focal adhesion kinase and paxillin in MCF-7 human breast cancer cells.

L. D. De Luca, R. Chandraratna, K. Matsukuma, 1999, Cancer research.

The H-ras oncogene interferes with retinoic acid signaling and metabolism in NIH3T3 cells.

L. D. De Luca, C. Jones, K. Kósa, 1995, Cancer research.

Retinol and beta-carotene concentrations in skin, papillomas and carcinomas, liver, and serum of mice fed retinoic acid or beta-carotene to suppress skin tumor formation.

L. D. De Luca, U. Lichti, L. C. Chen, 1994, Nutrition and cancer.

Dietary retinoids are essential for skin papilloma formation induced by either the two-stage or the complete tumorigenesis model in female SENCAR mice.

L. D. De Luca, L. C. Chen, C. Jones, 1992, Cancer Letters.

Retinoic acid and methotrexate specifically increase PHA‐E‐lectin binding to a 67‐kDa glycoprotein in LA‐N‐1 human neuroblastoma cells

S. Ross, L. D. De Luca, C. Jones, 1995, International journal of cancer.