K. Okuma

Cortical bone loss due to skeletal unloading in aldehyde dehydrogenase 2 gene knockout mice is associated with decreased PTH receptor expression in osteocytes.

A. Sakai, Yoshiaki Yamanaka, Takafumi Tajima, 2018, Bone.

Disturbance of osteonal bone remodeling and high tensile stresses on the lateral cortex in atypical femoral fracture after long-term treatment with Risedronate and Alfacalcidol for osteoporosis

Hideaki Takahashi, N. Yamamoto, A. Sakai, 2021, Bone reports.

Disturbance of Osteonal Remodeling in Atypical Femoral Fracture: A Short Review of Pathogenesis and a Case Report: Histomorphometric Analysis of Fracture Site

Hideaki Takahashi, N. Yamamoto, A. Sakai, 2021, Osteoporotic Fracture and Systemic Skeletal Disorders.

Disruption of the Aldehyde Dehydrogenase 2 Gene Results in No Increase in Trabecular Bone Mass Due to Skeletal Loading in Association with Impaired Cell Cycle Regulation Through p21 Expression in the Bone Marrow Cells of Mice

T. Kawamoto, Toshiharu Mori, A. Sakai, 2017, Calcified Tissue International.

Disruption of the aldehyde dehydrogenase 2 gene increases the bone anabolic response to intermittent PTH treatment in an ovariectomized mouse model.

A. Sakai, Takafumi Tajima, Manabu Tsukamoto, 2020, Bone.

Disruption of the Aldehyde Dehydrogenase 2 Gene Results in No Increase in Trabecular Bone Mass Due to Skeletal Loading in Association with Impaired Cell Cycle Regulation Through p21 Expression in the Bone Marrow Cells of Mice

T. Kawamoto, Toshiharu Mori, A. Sakai, 2017, Calcified Tissue International.