R. Laybutt

发表

Glucose Transporter (GLUT-4) Is Targeted to Secretory Granules in Rat Atrial Cardiomyocytes

J. Slot, D. James, E. Kraegen, 1997, The Journal of cell biology.

Casitas b-lineage lymphoma-deficient mice are protected against high-fat diet-induced obesity and insulin resistance.

D. James, N. Turner, G. Cooney, 2006, Diabetes.

Low serum PYY is linked to insulin resistance in first-degree relatives of subjects with type 2 diabetes

L. Heilbronn, L. Campbell, H. Herzog, 2006, Neuropeptides.

Chronic Hyperglycemia Triggers Loss of Pancreatic β Cell Differentiation in an Animal Model of Diabetes*

S. Bonner-Weir, Arun J Sharma, W. Hasenkamp, 1999, The Journal of Biological Chemistry.

beta-cell adaptation to hyperglycemia.

S. Bonner-Weir, H. Kaneto, G. Weir, 2001, Diabetes.

Potential role of the early response gene c-myc in beta-cell adaptation to changes in glucose concentration.

G. Steil, G. Weir, J. Henquin, 2001, Diabetes.

Angiotensin Converting Enzyme‐2 Therapy Improves Liver Fibrosis and Glycemic Control in Diabetic Mice With Fatty Liver

P. Angus, I. Alexander, C. Herath, 2021, Hepatology communications.

NPY signalling in early osteoblasts controls glucose homeostasis

P. Baldock, H. Herzog, A. D. Nguyen, 2015, Molecular metabolism.

Translational control is required for the unfolded protein response and in vivo glucose homeostasis.

E McEwen, S. Bonner-Weir, D. Scheuner, 2001, Molecular cell.

MicroRNAs contribute to compensatory β cell expansion during pregnancy and obesity.

M. Prentki, B. Thorens, S. Rome, 2012, The Journal of clinical investigation.

Contribution of Intronic miR-338-3p and Its Hosting Gene AATK to Compensatory β-Cell Mass Expansion.

M. Prentki, E. Ayuso, V. Jiménez, 2015, Molecular endocrinology.

Title : Contribution of Intronic miR-338-3 p and Its Hosting Gene AATK to Compensatory β-Cell Mass Expansion

E. Ayuso, V. Jiménez, F. Bosch, 2022 .

Critical reduction in beta-cell mass results in two distinct outcomes over time. Adaptation with impaired glucose tolerance or decompensated diabetes.

S. Bonner-Weir, D. R. Laybutt, G. Weir, 2003, The Journal of biological chemistry.

Inhibition of Y1 receptor signaling improves islet transplant outcome

S. Grey, C. Ip, D. R. Laybutt, 2017, Nature Communications.

Neuropeptide Y1 receptor antagonism protects β-cells and improves glycemic control in type 2 diabetes

H. Thomas, H. Herzog, Yanchuan Shi, 2021, Molecular metabolism.

Elevated Neuropeptide Y1 Receptor Signaling Contributes to β-cell Dysfunction and Failure in Type 2 Diabetes

H. Thomas, H. Herzog, Yanchuan Shi, 2021, bioRxiv.