K. Caldwell

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Prenatal Alcohol Exposure Damages Brain Signal Transduction System

K. Caldwell, 2004 .

Fear conditioning is associated with altered integration of PLC and ERK signaling in the hippocampus

K. Caldwell, Colin T. Buckley, 2004, Pharmacology Biochemistry and Behavior.

Formation of phosphatidylinositol 3-phosphate by isomerization from phosphatidylinositol 4-phosphate.

J. Walsh, P. Majerus, K. Caldwell, 1991, Proceedings of the National Academy of Sciences of the United States of America.

The maternal-placental-fetal interface: Adaptations of the HPA axis and immune mediators following maternal stress and prenatal alcohol exposure

L. Bakhireva, A. Allan, E. Milligan, 2022, Experimental Neurology.

Identification of myotubularin as the lipid phosphatase catalytic subunit associated with the 3-phosphatase adapter protein, 3-PAP

M. Layton, P. Majerus, C. Mitchell, 2003, Proceedings of the National Academy of Sciences of the United States of America.

Chronic ethanol exposure increases the association of hippocampal mu-opioid receptors with G-protein receptor kinase 2.

K. Caldwell, L. Saland, J. B. Chavez, 2008, Alcohol.

Isolation and characterization of two 3-phosphatases that hydrolyze both phosphatidylinositol 3-phosphate and inositol 1,3-bisphosphate.

P. Majerus, V. Bansal, D. Lips, 1991, The Journal of biological chemistry.

Developmental arsenic exposure is associated with sex differences in the epigenetic regulation of stress genes in the adult mouse frontal cortex

A. Allan, K. Caldwell, Elizabeth R. Solomon, 2020, Toxicology and applied pharmacology.

A mouse model of prenatal ethanol exposure using a voluntary drinking paradigm.

A. Allan, K. Caldwell, J. Chynoweth, 2003, Alcoholism, clinical and experimental research.

The inositol polyphosphate 4-phosphatase forms a complex with phosphatidylinositol 3-kinase in human platelet cytosol.

P. Majerus, C. Mitchell, F. Norris, 1999, Proceedings of the National Academy of Sciences of the United States of America.

The isolation and characterization of inositol polyphosphate 4-phosphatase.

P. Majerus, V. Bansal, K. Caldwell, 1990, The Journal of biological chemistry.

Do Class II Major Histocompatibility Molecules Function as Signal Transducers during B Lymphocyte Activation

J. Freed, D. Cooper, M. Newell, 1987 .

Hippocampal N-methyl-D-aspartate receptor subunit expression profiles in a mouse model of prenatal alcohol exposure.

A. Allan, K. Caldwell, S. Sheema, 2010, Alcoholism, clinical and experimental research.

The effects of Ca2+ and calmodulin on adenylyl cyclase activity in plasma membranes derived from neural and non-neural cells.

D. Cooper, K. Caldwell, C. Boyajian, 1992, Cell calcium.

Pathway for the formation of D-3 phosphate containing inositol phospholipids in intact human platelets.

P. Majerus, C. Mitchell, V. Bansal, 1990, Journal of Biological Chemistry.

Two-layer antibody capture of enzymes on the surface of microtiter plates: application to the study of the regulation of phospholipase C-gamma1 catalytic activity.

K. Caldwell, Colin T. Buckley, 2003, Analytical biochemistry.

Prenatal ethanol exposure persistently impairs NMDA receptor‐dependent activation of extracellular signal‐regulated kinase in the mouse dentate gyrus

N. Perrone-Bizzozero, C. Valenzuela, A. Allan, 2009, Journal of neurochemistry.

Prenatal ethanol exposure impairs executive function in mice into adulthood.

J. Brigman, K. Marquardt, K. Caldwell, 2014, Alcoholism, clinical and experimental research.

Prenatal alcohol exposure alters p35, CDK5 and GSK3β in the medial frontal cortex and hippocampus of adolescent mice

A. Allan, L. A. Cunningham, K. Caldwell, 2014, Toxicology reports.

Moderate Prenatal Alcohol Exposure Reduces Plasticity and Alters NMDA Receptor Subunit Composition in the Dentate Gyrus

C. Valenzuela, M. R. Diaz, Julie C Everett, 2013, The Journal of Neuroscience.

Prenatal ethanol exposure reduces mGluR5 receptor number and function in the dentate gyrus of adult offspring.

N. Perrone-Bizzozero, D. Savage, S. Frausto, 2004, Alcoholism, clinical and experimental research.

Effects of prenatal ethanol exposure on phospholipase C-beta 1 and phospholipase A2 in hippocampus and medial frontal cortex of adult rat offspring.

E. Weeber, D. Savage, A. Allan, 1997, Alcoholism, clinical and experimental research.

Signal Transduction Mechanisms for Adenosine

D. Cooper, K. Caldwell, 1990 .

Interactions between neurotransmitters that regulate cAMP and intracellular Ca2+ levels in the CNS.

D. Cooper, W. Schlegel, M. Ahlijanian, 1988, Advances in experimental medicine and biology.

Adenosine A1 receptors inhibit both adenylate cyclase activity and TRH-activated Ca2+ channels by a pertussis toxin-sensitive mechanism in GH3 cells.

D. Cooper, W. Schlegel, K. Caldwell, 1989, Cellular signalling.

Evaluation of methods for the isolation of plasma membranes displaying guanosine 5'-triphosphate-dependence for the regulation of adenylate cyclase activity: potential application to the study of other guanosine 5'-triphosphate-dependent transduction systems.

K. Prasad, D. Cooper, M. Newell, 1988, Analytical biochemistry.

The Phosphatidylinositol Pathway of Platelets and Vascular Cells a

P. Majerus, C. Mitchell, V. Bansal, 1991, Annals of the New York Academy of Sciences.

Arsenic exposure during embryonic development alters the expression of the long noncoding RNA growth arrest specific-5 (Gas5) in a sex-dependent manner.

A. Allan, K. Caldwell, Elizabeth R. Solomon, 2017, Neurotoxicology and teratology.

Wortmannin-Sensitive Phosphorylation, Translocation, and Activation of PLCγ1, but Not PLCγ2, in Antigen-stimulated RBL-2H3 Mast Cells

J. Pfeiffer, S. A. Barker, K. Caldwell, 1998 .

Prenatal Alcohol Exposure Results in Sex-Specific Alterations in Circular RNA Expression in the Developing Mouse Brain

N. Mellios, A. Allan, A. Gardiner, 2020, Frontiers in Neuroscience.

Wortmannin blocks lipid and protein kinase activities associated with PI 3-kinase and inhibits a subset of responses induced by Fc epsilon R1 cross-linking.

J. Oliver, J. Pfeiffer, A. Hall, 1995, Molecular biology of the cell.

A limited access mouse model of prenatal alcohol exposure that produces long-lasting deficits in hippocampal-dependent learning and memory.

A. Allan, K. Caldwell, Megan L. Brady, 2012, Alcoholism, clinical and experimental research.

Fetal alcohol spectrum disorder-associated depression: Evidence for reductions in the levels of brain-derived neurotrophic factor in a mouse model

R. Paz, A. Allan, Sara N. Alcon, 2008, Pharmacology Biochemistry and Behavior.

Characterization of an adapter subunit to a phosphatidylinositol (3)P 3-phosphatase: Identification of a myotubularin-related protein lacking catalytic activity

J. Whisstock, M. Layton, P. Majerus, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Recent insights in phosphatidylinositol signaling

P. Majerus, T. Ross, V. Bansal, 1990, Cell.

Aging Exacerbates Neuroinflammatory Outcomes Induced by Acute Ozone Exposure

E. Milligan, M. Campen, Selita N. Lucas, 2018, Toxicological sciences : an official journal of the Society of Toxicology.

Prenatal alcohol exposure results in brain region- and sex-specific changes in circHomer1 expression in adult mouse brain

N. Mellios, A. Allan, K. Caldwell, 2023, Frontiers in Neuroscience.

Benzo[a]pyrene- and TCDD-induced alterations in tyrosine phosphorylation and insulin-like growth factor signaling pathways in the MCF-10A human mammary epithelial cell line.

S. Burchiel, S. Ethier, S. Tannheimer, 1998, Carcinogenesis.

Identification of mitogen-activated protein kinase docking sites in enzymes that metabolize phosphatidylinositols and inositol phosphates

K. Caldwell, Colin T. Buckley, M. Sosa, 2006, Cell Communication and Signaling.

Chronic Ethanol Exposure Increases the Association of Hippocampal Mu-Opioid Receptors with G-Protein Receptor Kinase 2 (GRK2)

D. C. Lee, K. Caldwell, L. Saland, 2008 .

Effects of a novel cognition-enhancing agent on fetal ethanol-induced learning deficits.

Martina J. Rosenberg, K. Akers, D. Hamilton, 2010, Alcoholism, clinical and experimental research.

Prenatal Alcohol Exposure Is Associated with Altered Subcellular Distribution of Glucocorticoid and Mineralocorticoid Receptors in the Adolescent Mouse Hippocampal Formation

A. Allan, K. Caldwell, Samantha L. Goggin, 2013, Alcoholism, clinical and experimental research.

Sex-Dependent Effects of the Histone Deacetylase Inhibitor, Sodium Valproate, on Reversal Learning After Developmental Arsenic Exposure

A. Allan, K. Caldwell, Elizabeth R. Solomon, 2018, Front. Genet..

Prenatal alcohol exposure alters mRNA expression for stress peptides, glucocorticoid receptor function and immune factors in acutely stressed neonatal brain

C. Valenzuela, D. Savage, A. Allan, 2023, Frontiers in Neuroscience.

Sex-specific deficits in biochemical but not behavioral responses to delay fear conditioning in prenatal alcohol exposure mice

A. Allan, K. Caldwell, Elizabeth R. Solomon, 2018, Neurobiology of Learning and Memory.

Fear Conditioning-Induced Alterations of Phospholipase C-β1a Protein Level and Enzyme Activity in Rat Hippocampal Formation and Medial Frontal Cortex

R. Sutherland, E. Weeber, D. Savage, 2001, Neurobiology of Learning and Memory.

Prenatal Alcohol Exposure Modifies Glucocorticoid Receptor Subcellular Distribution in the Medial Prefrontal Cortex and Impairs Frontal Cortex-Dependent Learning

A. Allan, K. Caldwell, Samantha L. Goggin, 2014, PloS one.

Identification of Phospholipase C-γ1 as a Mitogen-activated Protein Kinase Substrate*

S. Rhee, F. Sekiya, K. Caldwell, 2004, Journal of Biological Chemistry.

Does calmodulin regulate non-neuronal adenylate cyclase?

D. Cooper, K. Caldwell, 1988, The Biochemical journal.

A novel method for the normalization of ChIP-qPCR data

Elizabeth R Solomon, Kevin K Caldwell, Andrea M Allan, 2021, MethodsX.

Prevalence and epidemiologic characteristics of FASD from various research methods with an emphasis on recent in-school studies.

David Buckley, Lisa H. Lu, Suzanne M. Houston, 2009, Developmental disabilities research reviews.

Delay fear conditioning modifies phospholipase C-β1a signaling in the hippocampus and frontal cortex

E. Weeber, K. Caldwell, 2004, Pharmacology Biochemistry and Behavior.

Prenatal ethanol exposure impairs executive function in mice into adulthood.

J. Brigman, K. Marquardt, K. Caldwell, 2014, Alcoholism, clinical and experimental research.

A limited access mouse model of prenatal alcohol exposure that produces long-lasting deficits in hippocampal-dependent learning and memory.

A. Allan, K. Caldwell, Megan L. Brady, 2012, Alcoholism: Clinical and Experimental Research.

The impact of prenatal alcohol exposure on hippocampal-dependent outcome measures is influenced by prenatal and early-life rearing conditions.

A. Allan, M. Labrecque, K. Caldwell, 2015, Alcoholism, clinical and experimental research.