I. Lim

发表

Quercetin inhibits fibronectin production by keloid-derived fibroblasts. Implication for the treatment of excessive scars.

B. Bay, S. T. Lee, L. Sun, 2003, Journal of dermatological science.

Complex epithelial–mesenchymal interactions modulate transforming growth factor‐β expression in keloid‐derived cells

M. Longaker, W. Xia, George P. Yang, 2004, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

Evaluation of cell culture on the polyurethane-based membrane (TegadermTM): implication for tissue engineering of skin

B. Bay, T. T. Phan, I. J. Lim, 2004, Cell and Tissue Banking.

Increased transcriptional response to mechanical strain in keloid fibroblasts due to increased focal adhesion complex formation

M. Longaker, K. Fong, George P. Yang, 2006, Journal of cellular physiology.

From scarless fetal wounds to keloids: Molecular studies in wound healing

M. Longaker, H. Lorenz, George P. Yang, 2003, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

Water-soluble betamethasone-loaded poly(lactide-co-glycolide) hollow microparticles as a sustained release dosage form.

Y. Y. Yang, C. Chaw, T. Phan, 2003, Journal of microencapsulation.

Functional cationic derivatives of starch as antimicrobial agents

Yi Yan Yang, J. Tan, Yugen Zhang, 2019, Polymer Chemistry.

The role of the activin system in keloid pathogenesis.

S. Chan, D. Phillips, A. Mukhopadhyay, 2007, American journal of physiology. Cell physiology.

Conditioned medium from keloid keratinocyte/keloid fibroblast coculture induces contraction of fibroblast‐populated collagen lattices

A. Mukhopadhyay, S. Chan, I. Lim, 2005, The British journal of dermatology.

Upregulation of secretory connective tissue growth factor (CTGF) in keratinocyte‐fibroblast coculture contributes to keloid pathogenesis

A. Mukhopadhyay, D. Do, H. Han, 2006, Journal of cellular physiology.

Evaluation of electrospun PCL/gelatin nanofibrous scaffold for wound healing and layered dermal reconstitution.

B. Bay, S. Ramakrishna, Yanzhong Zhang, 2007, Acta biomaterialia.

The role of hepatocyte growth factor/c-Met system in keloid pathogenesis.

A. Mukhopadhyay, I. Lim, V. Dang, 2010, The Journal of trauma.

Synchronous Activation of ERK and Phosphatidylinositol 3-Kinase Pathways Is Required for Collagen and Extracellular Matrix Production in Keloids*

T. T. Nguyen, H. Huynh, E. Tran, 2003, Journal of Biological Chemistry.

mTOR as a potential therapeutic target for treatment of keloids and excessive scars

A. Mukhopadhyay, O. Aalami, D. Do, 2007, Experimental dermatology.

Role of IGF system of mitogens in the induction of fibroblast proliferation by keloid-derived keratinocytes in vitro.

Hung Huynh, Boon Huat Bay, H. Huynh, 2003, American journal of physiology. Cell physiology.

Differences in collagen production between normal and keloid-derived fibroblasts in serum-media co-culture with keloid-derived keratinocytes.

Michael T Longaker, H. Huynh, M. Longaker, 2002, Journal of dermatological science.

Preclinical Evaluation of Tegaderm™ Supported Nanofibrous Wound Matrix Dressing on Porcine Wound Healing Model.

C. Lim, S. Ramakrishna, Yanzhong Zhang, 2015, Advances in wound care.

TECHNOLOGY ADVANCES Preclinical Evaluation of Tegaderm Supported Nanofibrous Wound Matrix Dressing on Porcine Wound Healing Model

S. Ramakrishna, Yanzhong Zhang, Irene H. C. Kee, 2015 .

Stat3 contributes to keloid pathogenesis via promoting collagen production, cell proliferation and migration

Xinmin Cao, I. Lim, T. Phan, 2006, Oncogene.

Investigation of the influence of keloid-derived keratinocytes on fibroblast growth and proliferation in vitro.

M. Longaker, M T Longaker, W. Tan, 2001, Plastic and reconstructive surgery.

Syndecan-2 and decorin: proteoglycans with a difference--implications in keloid pathogenesis.

S. Chan, A. Mukhopadhyay, H. H. Cheong, 2010, The Journal of trauma.

Differential transcriptional responses of keloid and normal keratinocytes to serum stimulation.

M. Longaker, W. Xia, George P. Yang, 2006, The Journal of surgical research.

Fibroblasts cocultured with keloid keratinocytes: normal fibroblasts secrete collagen in a keloidlike manner.

Hung Huynh, H. Huynh, M. Longaker, 2002, American journal of physiology. Cell physiology.

Cytokine profiling and Stat3 phosphorylation in epithelial-mesenchymal interactions between keloid keratinocytes and fibroblasts.

Xinmin Cao, C. P. Lim, I. Lim, 2009, The Journal of investigative dermatology.

Intraperitoneally Delivered Umbilical Cord Lining Mesenchymal Stromal Cells Improve Survival and Kidney Function in Murine Lupus via Myeloid Pathway Targeting

T. Lim, Jinmiao Chen, W. Hwang, 2022, International journal of molecular sciences.

Suppression of transforming growth factor beta/smad signaling in keloid-derived fibroblasts by quercetin: implications for the treatment of excessive scars.

M. Longaker, S. Chan, I. Lim, 2004, The Journal of trauma.

Epithelial and Mesenchymal Stem Cells from the Umbilical Cord Lining Membrane

I. Lim, T. Phan, 2014, Cell transplantation.

Water-soluble betamethasone-loaded poly(lactide-co-glycolide) hollow microparticles as a sustained release dosage form

Y. Y. Yang, C. Chaw, T. Phan, 2003 .

Increased CCN2 Transcription in Keloid Fibroblasts Requires Cooperativity Between AP-1 and SMAD Binding Sites

M. Longaker, W. Xia, George P. Yang, 2007 .

Increased CCN2 transcription in keloid fibroblasts requires cooperativity between AP-1 and SMAD binding sites.

M. Longaker, W. Xia, George P. Yang, 2004, Annals of surgery.

Smad3 signalling plays an important role in keloid pathogenesis via epithelial–mesenchymal interactions

M. Longaker, A. Mukhopadhyay, M. Longaker, 2005, The Journal of pathology.

Functional cationic derivatives of starch as antimicrobial agents

Y. Yang, Yugen Zhang, Guangshun Yi, 2019, Polymer Chemistry.