K. Mykkänen

CADASIL mutations and shRNA silencing of NOTCH3 affect actin organization in cultured vascular smooth muscle cells

M. Viitanen, M. Baumann, M. Pöyhönen, 2012, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Hereditary multi-infarct dementia of the Swedish type is a novel disorder different from NOTCH3 causing CADASIL.

M. Viitanen, C. Morris, D. St. Clair, 2007, Brain : a journal of neurology.

Congruence between NOTCH3 mutations and GOM in 131 CADASIL patients

M. Viitanen, M. Baumann, M. Pöyhönen, 2009, Brain : a journal of neurology.

CADASIL: the most common hereditary subcortical vascular dementia

M. Viitanen, M. Baumann, M. Pöyhönen, 2008 .

Diagnosing Vascular Dementia by Skin Biopsy - Uniqueness of CADASIL

M. Viitanen, M. Baumann, M. Pöyhönen, 2011 .

Proteome Analysis of Cultivated Vascular Smooth Muscle Cells from a CADASIL Patient

M. Viitanen, M. Baumann, M. Pöyhönen, 2007, Molecular medicine.

Different Clinical Phenotypes in Monozygotic CADASIL Twins With a Novel NOTCH3 Mutation

M. Viitanen, M. Pöyhönen, K. Amberla, 2009, Stroke.

Hereditary multi-infarct dementia of the Swedish type is a novel disorder different from NOTCH 3 causing

D. Clair, M. Viitanen, C. Morris, 2007 .

Mitochondrial DNA sequence variation and mutation rate in patients with CADASIL

M. Viitanen, S. Finnilä, K. Majamaa, 2006, Neurogenetics.

Detection of the founder effect in Finnish CADASIL families

M. Viitanen, P. Sistonen, M. Savontaus, 2004, European Journal of Human Genetics.

APOE and AGT in the Finnish p.Arg133Cys CADASIL population

M. Viitanen, S. Rovio, M. Baumann, 2015, Acta neurologica Scandinavica.