Y. Park

发表

The Systemic Autoinflammatory Diseases: Coming of Age with the Human Genome.

E. Remmers, D. Kastner, D. Stone, 2020, The Journal of allergy and clinical immunology.

Biallelic hypomorphic mutations in a linear deubiquitinase define otulipenia, an early-onset autoinflammatory disease

J. Mullikin, E. Remmers, D. Kastner, 2016, Proceedings of the National Academy of Sciences.

Brief Report: Connecting Two Pathways Through Ca2+ Signaling: NLRP3 Inflammasome Activation Induced by a Hypermorphic PLCG2 Mutation

D. Kastner, J. Kehrl, P. Hoffmann, 2015, Arthritis & rheumatology.

The Yersinia Virulence Factor YopM Hijacks Host Kinases to Inhibit Type III Effector-Triggered Activation of the Pyrin Inflammasome.

D. Kastner, P. Hearing, I. Brodsky, 2016, Cell host & microbe.

Aberrant tRNA processing causes an autoinflammatory syndrome responsive to TNF inhibitors

W. C. Owen, M. Hafner, A. Šedivá, 2018, Annals of the rheumatic diseases.

Prostaglandin E2 Inhibits NLRP3 Inflammasome Activation through EP4 Receptor and Intracellular Cyclic AMP in Human Macrophages

J. Shelhamer, D. Kastner, Yueqin Liu, 2015, The Journal of Immunology.

Receptor and Intracellular Cyclic AMP in Inflammasome Activation through EP4 Inhibits NLRP3 2 Prostaglandin E

D. Kastner, Yueqin Liu, M. Sokolowska, 2015 .

Aberrant actin depolymerization triggers the pyrin inflammasome and autoinflammatory disease that is dependent on IL-18, not IL-1β

D. Kastner, A. Roberts, S. Masters, 2015, Journal of Experimental Medicine.

Loss-of-function mutations in TNFAIP3 leading to A20 haploinsufficiency cause an early onset autoinflammatory syndrome

J. Mullikin, Yuan Zhang, D. Kastner, 2015, Nature Genetics.

Pyrin Inflammasome Activation and RhoA Signaling in the Autoinflammatory Diseases FMF and HIDS

D. Kastner, J. Chae, G. Wood, 2016, Nature Immunology.

Brief Report: Deficiency of Complement 1r Subcomponent in Early‐Onset Systemic Lupus Erythematosus: The Role of Disease‐Modifying Alleles in a Monogenic Disease

E. Remmers, D. Kastner, M. Gadina, 2017, Arthritis & rheumatology.

Selenoprotein W enhances skeletal muscle differentiation by inhibiting TAZ binding to 14-3-3 protein.

I. Kim, Y. Park, Y. Jeon, 2014, Biochimica et biophysica acta.

NLRP3 mutation and cochlear autoinflammation cause syndromic and nonsyndromic hearing loss DFNA34 responsive to anakinra therapy

H. Nakanishi, D. Kastner, J. Butman, 2017, Proceedings of the National Academy of Sciences.

Ancient familial Mediterranean fever mutations in human pyrin and resistance to Yersinia pestis

C. Rotimi, D. Shriner, E. Remmers, 2020, Nature Immunology.

Haematopoietic stem cell-dependent Notch transcription is mediated by p53 through the Histone chaperone Supt16h

D. Sahoo, D. Traver, K. Myung, 2020, Nature Cell Biology.

Transcriptional Regulation of Selenoprotein W by MyoD during Early Skeletal Muscle Differentiation*

I. Kim, Youn Wook Chung, Y. Park, 2010, The Journal of Biological Chemistry.

Inhibition of 14-3-3 binding to Rictor of mTORC2 for Akt phosphorylation at Ser473 is regulated by selenoprotein W.

I. Kim, Y. Park, Y. Jeon, 2013, Biochimica et biophysica acta.

Antioxidative role of selenoprotein W in oxidant-induced mouse embryonic neuronal cell death

Min Goo Lee, M. B. Yim, D. Jeong, 2009, Molecules and cells.

Selenoprotein W promotes cell cycle recovery from G2 arrest through the activation of CDC25B.

I. Kim, Y. Park, Y. Jeon, 2012, Biochimica et biophysica acta.

Autoantigen spermatid nuclear transition protein 1 enhances pro-inflammatory cytokine production stimulated by anti-DNA autoantibodies in macrophages

Y. Jang, Y. Park, Laxmi Prasad Uprety, 2022, European Journal of Inflammation.