P. Banin

Opposite Clinical Phenotypes of Glucokinase Disease: Description of a Novel Activating Mutation and Contiguous Inactivating Mutations in Human Glucokinase (GCK) Gene

J. C. Aledo, C. Dionisi-Vici, L. Lenzi, 2009 .

Opposite clinical phenotypes of glucokinase disease: Description of a novel activating mutation and contiguous inactivating mutations in human glucokinase (GCK) gene.

J. C. Aledo, F. Bermúdez-Silva, F. Rodríguez de Fonseca, 2009, Molecular endocrinology.

Type 1 diabetes (T1DM) in children and adolescents of immigrated families in Emilia-Romagna (Italy).

S. Zucchini, G. Chiari, V. De Sanctis, 2010, Acta bio-medica : Atenei Parmensis.

Sublocalization of von Willebrand factor pseudogene to 22q11.22–q11.23 by in situ hybridization in a 46,X,t(X;22)(pter;q11.21) translocation

E. Calzolari, F. Bernardi, G. Marchetti, 1989, Human Genetics.

The continuous glucose monitoring system (CGMS) in patients with beta-thalassemia major and impaired glucose homeostasis: preliminary results.

V. De Sanctis, M. Gamberini, P. Banin, 2008, Pediatric endocrinology reviews : PER.

Opposite clinical phenotypes of glucokinase disease: Description of a novel activating mutation and contiguous inactivating mutations in human glucokinase (GCK) gene.

F. Bermúdez-Silva, F. Rodríguez de Fonseca, C. Dionisi-Vici, 2009, Molecular endocrinology.

Opposite Clinical Phenotypes of “Glucokinase Disease”: Description of a Novel Activating Mutation and Contiguous Inactivating Mutations in Human Glucokinase Gene

C. Dionisi-Vici, L. Lenzi, S. Toni, 2009 .