A. Ruchon

Cellular Localization of Neprilysin in Mouse Bone Tissue and Putative Role in Hydrolysis of Osteogenic Peptides

J. Aubin, M. Marcinkiewicz, A. Basak, 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Frequency and phenotypic consequences of the 3199del6 CFTR mutation in French Canadians

R. Rozen, Raouf Fetni, Patrick Scott, 2005, Genetics in Medicine.

Role of abnormal neutral endopeptidase-like activities in Hyp mouse bone cells in renal phosphate transport.

D. Lajeunesse, A. Delalandre, G. Boileau, 2002, American journal of physiology. Cell physiology.

Molecular cloning and biochemical characterization of a new mouse testis soluble-zinc-metallopeptidase of the neprilysin family.

N. Seidah, M. Marcinkiewicz, L. DesGroseillers, 2000, The Biochemical journal.

A new brain metalloendopeptidase which degrades the Alzheimer beta-amyloid 1-40 peptide producing soluble fragments without neurotoxic effects.

K. Carvalho, A. Ruchon, M. França, 1997, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.

Developmental Expression and Tissue Distribution of Phex Protein: Effect of the Hyp Mutation and Relationship to Bone Markers

J. Aubin, M. Marcinkiewicz, L. DesGroseillers, 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Report on the p.Ser489X (p.Ser489*) CFTR mutation, a variant with severe associated phenotype and high prevalence in a Quebec French-Canadian cystic fibrosis patient population

I. D. Bie, R. Agatep, A. Ruchon, 2012, Genetics in Medicine.