Wonil Kim

发表

Selective Inhibition of Hdac1 and Hdac2 as a Potential Therapeutic Option for B-all Short Title: B-alls Are Sensitive to an Hdac1/hdac2 Inhibitor

Tina N. Davis, S. Armstrong, J. Qi, 2022 .

Selective Inhibition of HDAC1 and HDAC2 as a Potential Therapeutic Option for B-ALL

Tina N. Davis, S. Armstrong, J. Qi, 2015, Clinical Cancer Research.

Immune Responses of a Novel Bi-Cistronic SARS-CoV-2 DNA Vaccine Following Intradermal Immunization With Suction Delivery

J. Shan, L. Montaner, J. Zahn, 2022, Frontiers in Virology.

Novel Targets in Myelogenous Leukemia: The Id Family of Proteins

A. Satyanarayana, M. Ji, K. Klarmann, 2011 .

Selective Inhibition of HDAC1 and HDAC2 Is a Potential Therapeutic Option for B-All

Tina N. Davis, S. Armstrong, J. Qi, 2010 .

Gfi-1 regulates the erythroid transcription factor network through Id2 repression in murine hematopoietic progenitor cells.

K. Klarmann, J. Keller, Wonil Kim, 2014, Blood.

repression in murine hematopoietic progenitor cells Id2 Gfi-1 regulates the erythroid transcription factor network through

K. Klarmann, J. Keller, Wonil Kim, 2014 .

Proteasome inhibition targets the KMT2A transcriptional complex in acute lymphoblastic leukemia

Nathaniel R. Twarog, Ji-Hoon Cho, Jing Ma, 2023, Nature Communications.

KMT2A-Rearranged Infant Acute Lymphoblastic Leukemia Cells Undergo ER-Stress-Induced Apoptosis Following Exposure to Proteasome Inhibitors

T. Gruber, Wonil Kim, C. Koss, 2019, Blood.

Selective Inhibition of HDAC1 and HDAC2 as a Potential Therapeutic Option for B-ALL

Tina N. Davis, S. Armstrong, J. Qi, 2015, Clinical Cancer Research.