Nathalie L'heureux

Distinct genomic replacements from Lewis correct diastolic dysfunction, attenuate hypertension, and reduce left ventricular hypertrophy in Dahl salt-sensitive rats

S. Nattel, J. Tardif, Yanfen Shi, 2008, Journal of hypertension.

Unique quantitative trait loci in synergy permanently improve diastolic dysfunction.

S. Nattel, J. Tardif, Yanfen Shi, 2013, The Canadian journal of cardiology.

Combining distinctive and novel loci doubles BP reduction, reverses diastolic dysfunction and mitigates LV hypertrophy

S. Nattel, J. Tardif, Yanfen Shi, 2013, Journal of hypertension.

Cardiac pathways distinguish two epistatic modules enacting BP quantitative trait loci and candidate gene analysis

S. Nattel, J. Tardif, J. Tremblay, 2009, Hypertension Research.

Comparison of Atrial Remodeling Caused by Sustained Atrial Flutter Versus Atrial Fibrillation.

S. Nattel, J. Tardif, R. Cartier, 2020, Journal of the American College of Cardiology.

Role of atrial arrhythmia and ventricular response in atrial fibrillation induced atrial remodeling.

Jiening Xiao, S. Nattel, J. Tardif, 2020, Cardiovascular research.

Sinus node dysfunction and hyperpolarization-activated (HCN) channel subunit remodeling in a canine heart failure model.

S. Nattel, M. Fernández-Velasco, Nathalie L'heureux, 2005, Cardiovascular research.