A. Ménard

发表

Distinct genomic replacements from Lewis correct diastolic dysfunction, attenuate hypertension, and reduce left ventricular hypertrophy in Dahl salt-sensitive rats

S. Nattel, J. Tardif, Yanfen Shi, 2008, Journal of hypertension.

A loss of genome buffering capacity of Dahl salt-sensitive model to modulate blood pressure as a cause of hypertension.

S. Charron, A. Deng, A. Ménard, 2005, Human molecular genetics.

Combining distinctive and novel loci doubles BP reduction, reverses diastolic dysfunction and mitigates LV hypertrophy

S. Nattel, J. Tardif, Yanfen Shi, 2013, Journal of hypertension.

Distinct quantitative trait loci for kidney, cardiac, and aortic mass dissociated from and associated with blood pressure in Dahl congenic rats

P. Hamet, S. Charron, C. Xiao, 2006, Mammalian Genome.

Animal Model Studies Reveal that Common Human-Centric Non-Coding Variants from Epidemiology are By-products of Primate Evolution Unrelated to Physiological Control of Blood Pressure

A. Deng, A. Ménard, 2021, Cardiology and Cardiovascular Medicine.

Cardiac pathways distinguish two epistatic modules enacting BP quantitative trait loci and candidate gene analysis

S. Nattel, J. Tardif, J. Tremblay, 2009, Hypertension Research.

Guinea pig liver transglutaminase: A modified purification procedure affording enzyme with superior activity in greater yield.

J. Keillor, A. Ménard, N. Day, 1999, Protein expression and purification.

Pre‐steady‐state kinetic studies of rat kidney γ‐glutamyl transpeptidase confirm its ping‐pong mechanism

R. Castonguay, C. Lherbet, J. Keillor, 2004 .

Nonlinear free energy relationship in the general-acid-catalyzed acylation of rat kidney gamma-glutamyl transpeptidase by a series of gamma-glutamyl anilide substrate analogues.

Y. Roupioz, R. Castonguay, C. Lherbet, 2001, Biochemistry.

Functional Dosage of Muscarinic Cholinergic Receptor 3 Signalling, Not the Gene Dose, Determines Its Hypertension Pathogenesis.

É. Thorin, D. Deblois, A. Deng, 2019, The Canadian journal of cardiology.

Conserved mammalian modularity of quantitative trait loci revealed human functional orthologs in blood pressure control

A. Deng, A. Ménard, 2020, PloS one.

Functional Captures of Multiple Human Quantitative Trait Loci Regulating Blood Pressure with the Use of Orthologs in Genetically Defined Rat Models.

A. Deng, A. Ménard, 2020, The Canadian journal of cardiology.

Submegabase resolution of epistatically interacting quantitative trait loci for blood pressure applicable for essential hypertension

S. Charron, C. Xiao, A. Deng, 2008, Journal of hypertension.

Alterations in Fibronectin Type III Domain Containing 1 Protein Gene Are Associated with Hypertension

A. Deng, A. Ménard, C. Chauvet, 2016, PloS one.

Shifting Paradigm from Gene Expressions to Pathways Reveals Physiological Mechanisms in Blood Pressure Control in Causation

A. Deng, A. Ménard, David W. Deng, 2023, International journal of molecular sciences.

Epistasis, Not Numbers, Regulates Functions of Clustered Dahl Rat Quantitative Trait Loci Applicable to Human Hypertension

S. Charron, A. Deng, A. Ménard, 2005, Hypertension.

α-Kinase 2 is a novel candidate gene for inherited hypertension in Dahl rats

C. Xiao, Yanrui Wu, A. Deng, 2011, Journal of hypertension.

Retinoblastoma-associated protein 140 as a candidate for a novel etiological gene to hypertension

A. Deng, A. Ménard, Kimberley Crespo, 2016, Clinical and experimental hypertension.

Modularization and epistatic hierarchy determine homeostatic actions of multiple blood pressure quantitative trait loci.

Cristina Chauvet, Kimberley Crespo, Annie Ménard, 2013, Human molecular genetics.

Sexual Dimorphism on Hypertension of Quantitative Trait Loci Entrapped in Dahl Congenic Rats

C. Xiao, A. Deng, A. Ménard, 2008, Clinical and experimental hypertension.

Individual QTLs controlling quantitative variation in blood pressure inherited in a Mendelian mode

S. Charron, C. Xiao, C. Xiao, 2007, Heredity.

Novel Pathogenesis of Hypertension and Diastolic Dysfunction Caused by M3R (Muscarinic Cholinergic 3 Receptor) Signaling

J. Tardif, S. Laporte, É. Thorin, 2018, Hypertension.

Normotension in Lewis and Dahl salt-resistant rats is governed by different genes

A. Deng, A. Ménard, J. Roy, 2011, Journal of hypertension.

Two candidate genes for two quantitative trait loci epistatically attenuate hypertension in a novel pathway

A. Deng, A. Ménard, C. Chauvet, 2015, Journal of hypertension.

Hypertension Suppression, Not a Cumulative Thrust of Quantitative Trait Loci, Predisposes Blood Pressure Homeostasis to Normotension

A. Deng, A. Ménard, Kimberley Crespo, 2015, Circulation. Cardiovascular genetics.

Novel genes as primary triggers for polygenic hypertension

C. Xiao, A. Deng, B. Aguila, 2012, Journal of hypertension.