Weihua Cui

发表

FMS-LIKE TYROSINE KINASE-3 LIGAND ALTERS ANTIGEN-SPECIFIC RESPONSES TO INFECTIONS AFTER SEVERE BURN INJURY

E. Sherwood, J. Bohannon, Geping Fang, 2009, Shock.

Neuron‐specific caveolin‐1 overexpression improves motor function and preserves memory in mice subjected to brain trauma

C. Mandyam, H. Patel, B. Head, 2017, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Insulin increases resistance to burn wound infection-associated sepsis

D. Herndon, M. Jeschke, G. Gauglitz, 2010, Critical care medicine.

Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice

H. Patel, I. Niesman, Jacqueline A. Bonds, 2014, Journal of Neuroinflammation.

Cafeteria Diet Impacts the Body Weight and Energy Expenditure of Brown Norway Rats in an Apparent Age Dependent Manner, but Has no Effect on Muscle Anabolic Sensitivity to Nutrition

N. Bhattarai, A. Murton, Amina El Ayadi, 2021, Frontiers in Nutrition.

Systemic administration of lidocaine reduces morphine requirements and postoperative pain of patients undergoing thoracic surgery after propofol–remifentanil-based anaesthesia

Junfa Li, Shuren Li, Weihua Cui, 2010, European journal of anaesthesiology.

Fms-Like Tyrosine Kinase-3 Ligand Attenuates Local and Systemic Infection in a Model of Post-Burn Pneumonia

R. Cox, T. Toliver-Kinsky, Weihua Cui, 2017, Shock.

Fms-like tyrosine kinase-3 ligand increases resistance to burn wound infection through effects on plasmacytoid dendritic cells

J. Bohannon, T. Toliver-Kinsky, Weihua Cui, 2017, BMC Immunology.

Dendritic Cell Modification of Neutrophil Responses to Infection after Burn Injury

E. Sherwood, J. Bohannon, T. Toliver-Kinsky, 2010, The Journal of Immunology.

Responses to Infection after Burn Injury Dendritic Cell Modification of Neutrophil

E. Sherwood, J. Bohannon, T. Toliver-Kinsky, 2010 .

H2S, a Bacterial Defense Mechanism against the Host Immune Response

E. Nudler, Konstantin Shatalin, C. Szabó, 2018, Infection and Immunity.

Enhancement of Dendritic Cell Production by Fms-Like Tyrosine Kinase-3 Ligand Increases the Resistance of Mice to a Burn Wound Infection1

E. Sherwood, E. Murphey, T. Toliver-Kinsky, 2005, The Journal of Immunology.

Wound Infection Increases the Resistance of Mice to a Burn Fms-Like Tyrosine Kinase-3 Ligand Enhancement of Dendritic Cell Production

E. Sherwood, E. Murphey, Weihua Cui, 2004 .

Enhances Global Immune Responses to Tyrosine Kinase-3 Ligand after Burn Injury Prophylactic Treatment with Fms-Like

R. Cox, E. Sherwood, J. Bohannon, 2017 .

Prophylactic Treatment with Fms-Like Tyrosine Kinase-3 Ligand after Burn Injury Enhances Global Immune Responses to Infection1

R. Cox, E. Sherwood, J. Bohannon, 2008, The Journal of Immunology.

CaMKII Phosphorylation in Primary Somatosensory Cortical Neurons is Involved in the Inhibition of Remifentanil-induced Hyperalgesia by Lidocaine in Male Sprague-Dawley Rats

Junfa Li, R. Han, Shanshan Wang, 2016, Journal of neurosurgical anesthesiology.

Endotoxin Priming Improves Clearance of Pseudomonas aeruginosa in Wild-Type and Interleukin-10 Knockout Mice

M. Durham, Zhiyu Huang, E. Sherwood, 2005, Infection and Immunity.

GLUCAN PHOSPHATE TREATMENT ATTENUATES BURN-INDUCED INFLAMMATION AND IMPROVES RESISTANCE TO PSEUDOMONAS AERUGINOSA BURN WOUND INFECTION

David L. Williams, E. Sherwood, E. Murphey, 2005, Shock.

Dendritic cells modulate burn wound healing by enhancing early proliferation

R. Cox, T. Toliver-Kinsky, Weihua Cui, 2016, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

Endogenous Fms-like Tyrosine Kinase-3 Ligand levels are not altered in mice after a severe burn and infection

J. Bohannon, T. Toliver-Kinsky, Weihua Cui, 2009, BMC Immunology.

Systemic Lidocaine Inhibits Remifentanil-induced Hyperalgesia via the Inhibition of cPKCgamma Membrane Translocation in Spinal Dorsal Horn of Rats

Junfa Li, Shuren Li, Song Han, 2009, Journal of neurosurgical anesthesiology.

Long-term atorvastatin treatment leads to alterations in behavior, cognition, and hippocampal biochemistry

H. Patel, I. Niesman, J. Drummond, 2014, Behavioural Brain Research.

The role of caveolin-1 in morphine-induced structural plasticity in primary cultured mouse cerebral cortical neurons

Junfa Li, Song Han, R. Han, 2018, Neuroscience Letters.

Neuron-Targeted Caveolin-1 Improves Molecular Signaling, Plasticity, and Behavior Dependent on the Hippocampus in Adult and Aged Mice

C. Mandyam, H. Patel, I. Niesman, 2017, Biological Psychiatry.

The increased release of amino acid neurotransmitters of the primary somatosensory cortical area in rats contributes to remifentanil-induced hyperalgesia and its inhibition by lidocaine

Junfa Li, Song Han, Shanshan Wang, 2018, Journal of pain research.

In vivo reduction of hippocampal Caveolin-1 by RNA interference alters morphine addiction and neuroplasticity changes in male mice

Junfa Li, Song Han, Shanshan Wang, 2021, Neuroscience Letters.

Neuron-targeted caveolin-1 improves motor function and preserves memory in mice subjected to brain trauma

C. Mandyam, H. Patel, B. Head, 2018 .

Fms-like tyrosine kinase-3 ligand increases resistance to burn wound infection through effects on plasmacytoid dendritic cells

J. Bohannon, T. Toliver-Kinsky, Weihua Cui, 2017, BMC Immunology.