B. Mushala

发表

Diet-induced obese mice are resistant to improvements in cardiac function resulting from short-term adropin treatment

I. Scott, M. Jurczak, M. Stoner, 2022, Current research in physiology.

GCN5L1 impairs diastolic function in mice exposed to a high fat diet by restricting cardiac pyruvate oxidation

N. Yates, I. Scott, M. Jurczak, 2022, Physiological reports.

GCN 5 L 1 promotes diastolic dysfunction by inhibiting cardiac pyruvate oxidation 1 2

N. Yates, I. Scott, M. Jurczak, 2022 .

Inhibitory Acetylation of Pyruvate Dehydrogenase Promotes Diastolic Dysfunction in Mice

I. Scott, M. Jurczak, M. Stoner, 2021 .

GCN5L1 promotes diastolic dysfunction by inhibiting cardiac pyruvate oxidation

N. Yates, I. Scott, M. Jurczak, 2022, bioRxiv.

G-PROTEIN COUPLED RECEPTOR 19 (GPR19) KNOCKOUT MICE DISPLAY SEX-DEPENDENT METABOLIC DYSFUNCTION

I. Scott, M. Jurczak, M. Stoner, 2022, bioRxiv.

ADROPIN: A HEPATOKINE MODULATOR OF VASCULAR FUNCTION AND CARDIAC FUEL METABOLISM.

I. Scott, B. Mushala, 2020, American journal of physiology. Heart and circulatory physiology.

Increased fatty acid oxidation enzyme activity in the hearts of mice fed a high fat diet does not correlate with improved cardiac contractile function

I. Scott, Manling Zhang, Dharendra Thapa, 2020, Current research in physiology.

Validation of GCN5L1/BLOC1S1/BLOS1 Antibodies Using Knockout Cells and Tissue

I. Scott, M. Stoner, Dharendra Thapa, 2023, bioRxiv.