S. Leung

发表

Point mutational analysis of the human c-fos serum response factor binding site.

S. Leung, N. Miyamoto, Stewart Leung, 1989, Nucleic acids research.

Structure of the two promoters of the human lck gene: differential accumulation of two classes of lck transcripts in T cells.

T. Mak, Y. Takihara, T. Takadera, 1989, Molecular and cellular biology.

Catalytically Active TYK2 Is Essential for Interferon-β-mediated Phosphorylation of STAT3 and Interferon-α Receptor-1 (IFNAR-1) but Not for Activation of Phosphoinositol 3-Kinase*

R. Ransohoff, A. Wolfman, E. Fish, 1999, The Journal of Biological Chemistry.

Benefical Role of the GPR30 agonist G-1 in an Animal Model of Multiple Sclerosis

E. Prossnitz, W. Karpus, R. Horuk, 2010 .

Beneficial role of the GPR30 agonist G-1 in an animal model of multiple sclerosis

M. Halks-Miller, E. Prossnitz, W. Karpus, 2009, Journal of Neuroimmunology.

Requirement of an ETS-binding element for transcription of the human lck type I promoter.

J. Ghysdael, S. Leung, N. Miyamoto, 1993, Oncogene.

Characterization of beta-R1, a gene that is selectively induced by interferon beta (IFN-beta) compared with IFN-alpha.

G. Stark, R. Ransohoff, M. Rani, 1996, The Journal of biological chemistry.

Characterization of a teleost insulin-like growth factor II (IGF-II) gene: evidence for promoter CCAAT/enhancer-binding protein (C/EBP) sites, and the presence of hepatic C/EBP.

T. T. Chen, M. Shamblott, S. Leung, 1998, Molecular marine biology and biotechnology.

Selective loss of type I interferon-induced STAT4 activation caused by a minisatellite insertion in mouse Stat2

G. Stark, J. Farrar, K. Murphy, 2000, Nature Immunology.

SIRT1 Activators Suppress Inflammatory Responses through Promotion of p65 Deacetylation and Inhibition of NF-κB Activity

Zhong Zhong, T. Riera, S. Sweitzer, 2012, PloS one.

Regulation of STAT‐dependent pathways by growth factors and cytokines

G. Stark, D. Leaman, Xiaoxia Li, 1996, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Activation of Phosphatidylinositol 3-Kinase in Response to Interleukin-1 Leads to Phosphorylation and Activation of the NF-κB p65/RelA Subunit

G. Stark, S. Leung, Nywana Sizemore, 1999, Molecular and Cellular Biology.

Differential expression of two classes of lck transcripts upon phorbol ester treatment of human leukemic T cells

S. Leung, N. Miyamoto, 1991, Journal of cellular physiology.

Formation of STAT1-STAT2 Heterodimers and Their Role in the Activation of IRF-1 Gene Transcription by Interferon- (*)

G R Stark, J E Darnell, J. Darnell, 1996, The Journal of Biological Chemistry.

J. Ghysdael, R. Bosselut, S. Leung, 1994, Oncogene.

Role of STAT2 in the alpha interferon signaling pathway

J. Darnell, G. Stark, I. Kerr, 1995, Molecular and cellular biology.

Characterization of β-R1, a Gene That Is Selectively Induced by Interferon β (IFN-β) Compared with IFN-α*

G. Stark, R. Ransohoff, M. Rani, 1996, The Journal of Biological Chemistry.

Characterization of b-R1, a Gene That Is Selectively Induced by Interferon b (IFN-b) Compared with IFN-a*

G. Stark, R. Ransohoff, G. Foster, 1996 .

Leukemia inhibitory factor inhibits T helper 17 cell differentiation and confers treatment effects of neural progenitor cell therapy in autoimmune disease.

Jingwu Z. Zhang, W. Cao, Yufang Shi, 2011, Immunity.

Crucial role of interleukin-7 in T helper type 17 survival and expansion in autoimmune disease

Jingwu Z. Zhang, Runsheng Li, T. Guo, 2010, Nature Medicine.

Function of Stat2 protein in transcriptional activation by alpha interferon

J. Darnell, G. Stark, I. Kerr, 1996, Molecular and cellular biology.

Interplay of Pathogenic TH1/TH17 Cells and Regulatory T Cells in Auto-immune Disease: A Tale of Yin and Yang

Jingwu Z. Zhang, T. Guo, L. Fang, 2011 .

The cytokine milieu in the interplay of pathogenic Th1/Th17 cells and regulatory T cells in autoimmune disease

X. Chen, Jingwu Z. Zhang, T. Guo, 2010, Cellular and Molecular Immunology.

Repression of IL-4-Induced Gene Expression by IFN-γ Requires Stat1 Activation

H. Mano, U. Schindler, S. Leung, 1999, The Journal of Immunology.

Interferon Signaling Is Dependent on Specific Tyrosines Located within the Intracellular Domain of IFNAR2c

G. Stark, R. Ransohoff, M. Rani, 2002, The Journal of Biological Chemistry.

Functional subdomains of STAT2 required for preassociation with the alpha interferon receptor and for signaling

G. Stark, I. Kerr, S. Leung, 1997, Molecular and cellular biology.

Cooperative binding of Stat1-2 heterodimers and ISGF3 to tandem DNA elements.

G. Stark, Xiaoxia Li, S. Leung, 1998, Biochimie.

STATs Find That Hanging Together Can Be Stimulating

Xiaoxia Li, S. Leung, George R. Stark, 1996, Science.

Combinatorial association and abundance of components of interferon-stimulated gene factor 3 dictate the selectivity of interferon responses.

D. Levy, G. Stark, I. Kerr, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Crucial role of interleukin-7 in T helper type 17 survival and expansion in autoimmune disease.

Jingwu Z. Zhang, Runsheng Li, T. Guo, 2010, Nature medicine.

Differential Inhibition of Inducible T Cell Cytokine Secretion by Potent Iron Chelators

Hong-tao Lu, M. Berger, D. Schilling, 2005, Journal of biomolecular screening.

Catalytically active TYK2 is essential for interferon-beta-mediated phosphorylation of STAT3 and interferon-alpha receptor-1 (IFNAR-1) but not for activation of phosphoinositol 3-kinase.

R. Ransohoff, A. Wolfman, E. Fish, 1999, Journal of Biological Chemistry.

Activation of Phosphatidylinositol 3-Kinase in Response to Interleukin-1 Leads to Phosphorylation and Activation of the NF-κB p65/RelA Subunit

G. Stark, S. Leung, Nywana Sizemore, 1999, Molecular and Cellular Biology.