Chehade N. Karam

发表

Sphingosine-1-Phosphate Receptor Modulator, FTY720, Improves Diastolic Dysfunction and Partially Reverses Atrial Remodeling in a Tm-E180G Mouse Model Linked to Hypertrophic Cardiomyopathy.

Shamim A. K. Chowdhury, C. Liew, B. Wolska, 2019, Circulation. Heart failure.

Effect of insulin and angiotensin II receptor subtype-1 antagonist on myocardial remodelling in rats with insulin-dependent diabetes mellitus

A. Bikhazi, K. Bitar, N. Nuwayri-Salti, 2005, Journal of hypertension.

Absence of physiological Ca2+ transients is an initial trigger for mitochondrial dysfunction in skeletal muscle following denervation

K. Dhakal, Heping Cheng, Jianjie Ma, 2017, Skeletal Muscle.

ROS‐related mitochondrial dysfunction in skeletal muscle of an ALS mouse model during the disease progression

K. Dhakal, Jianjie Ma, J. Yi, 2018, Pharmacological research.

Effect of type-1 diabetes mellitus on the regulation of insulin and endothelin-1 receptors in rat hearts.

A. Bikhazi, K. Bitar, N. Nuwayri-Salti, 2007, Canadian journal of physiology and pharmacology.

Green Tea Catechin Normalizes the Enhanced Ca2+ Sensitivity of Myofilaments Regulated by a Hypertrophic Cardiomyopathy–Associated Mutation in Human Cardiac Troponin I (K206I)

M. Ackerman, P. D. de Tombe, J. Bos, 2015, Circulation. Cardiovascular genetics.

Effect of systemic insulin and angiotensin II receptor subtype-1 antagonist on endothelin-1 receptor subtype(s) regulation and binding in diabetic rat heart.

A. Bikhazi, K. Bitar, N. Nuwayri-Salti, 2005, Endothelium : journal of endothelial cell research.

Expression of tropomyosin-κ induces dilated cardiomyopathy and depresses cardiac myofilament tension by mechanisms involving cross-bridge dependent activation and altered tropomyosin phosphorylation

R. Solaro, C. Warren, S. Rajan, 2011, Journal of Muscle Research and Cell Motility.

Molecular and Functional Characterization of a Novel Cardiac-Specific Human Tropomyosin Isoform

Christian F. Bulcao, G. Boivin, S. Liggett, 2009, Circulation.

Peroxisome proliferator-activated receptor-α expression induces alterations in cardiac myofilaments in a pressure-overload model of hypertrophy.

E. Lewandowski, R. Solaro, M. Henze, 2017, American journal of physiology. Heart and circulatory physiology.

Function Peroxisome proliferator-activated receptor- (cid:2) expression induces alterations in cardiac myoﬁlaments in a pressure-overload model of hypertrophy

E. Lewandowski, M. Henze, R. Solaro, 2017 .