M. Vos

发表

The Use of Vitamin K2 in Patients With Parkinson's Disease and Mitochondrial Dysfunction (PD-K2): A Theranostic Pilot Study in a Placebo-Controlled Parallel Group Design

I. König, N. Brüggemann, S. Schmid, 2021, Frontiers in Neurology.

Mitochondrial Complex I deficiency: guilty in Parkinson’s disease

M. Vos, 2022, Signal Transduction and Targeted Therapy.

De Novo Variants in TAOK1 Cause Neurodevelopmental Disorders.

Maximilian E. R. Weiss, K. Kandaswamy, K. Lohmann, 2019, American journal of human genetics.

This contribution is part of the special series of Inaugural Articles by members of the National Academy of Sciences elected on April 29 , 1997 . In vivo functions of the Saccharomyces cerevisiae Hsp 90 chaperone

S. Lindquist, D. Nathan, M. Vos, 1997 .

Reduced penetrance of Parkinson’s disease models

M. Vos, V. A. Morais, 2022, Medizinische Genetik.

Mutant WDR45 Leads to Altered Ferritinophagy and Ferroptosis in β-Propeller Protein-Associated Neurodegeneration

K. Lohmann, S. Zittel, A. Rakovic, 2022, International journal of molecular sciences.

Frontiers in Synaptic Neuroscience Synaptic Neuroscience

Elsa Lauwers, P. Verstreken, Patrik Verstreken, 2022 .

Vitamin K2 Is a Mitochondrial Electron Carrier That Rescues Pink1 Deficiency

Janaka N. Edirisinghe, P. Verstreken, B. de Strooper, 2012, Science.

Characterization of A-425619 at native TRPV1 receptors: a comparison between dorsal root ganglia and trigeminal ganglia.

T. Neelands, R. Moreland, H. McDonald, 2008, European journal of pharmacology.

Capsaicin causes protein synthesis inhibition and microtubule disassembly through TRPV1 activities both on the plasma membrane and intracellular membranes.

R. Moreland, H. McDonald, M. Jarvis, 2007, Biochemical pharmacology.

In vivo functions of the Saccharomyces cerevisiae Hsp90 chaperone.

S. Lindquist, D. Nathan, M. Vos, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Cardiolipin promotes electron transport between ubiquinone and complex I to rescue PINK1 deficiency

P. Verstreken, B. de Strooper, K. Craessaerts, 2017, The Journal of cell biology.

JCB_201511044 1..14

Klein, Böhm, P. Verstreken, 2017 .

CEP89 is required for mitochondrial metabolism and neuronal function in man and fly.

B. V. van Bon, N. de Leeuw, B. D. de Vries, 2013, Human molecular genetics.

Near-Infrared 808 nm Light Boosts Complex IV-Dependent Respiration and Rescues a Parkinson-Related pink1 Model

P. Verstreken, B. de Strooper, Y. Tardy, 2013, PloS one.

Annals of the New York Academy of Sciences Therapeutic Strategies in Parkinson's Disease: What We Have Learned from Animal Models

P. Verstreken, J. S. Valadas, M. Vos, 2022 .

(R)-(5-tert-Butyl-2,3-dihydro-1H-inden-1-yl)-3-(1H-indazol-4-yl)-urea (ABT-102) Blocks Polymodal Activation of Transient Receptor Potential Vanilloid 1 Receptors in Vitro and Heat-Evoked Firing of Spinal Dorsal Horn Neurons in Vivo

A. Gomtsyan, J. Sullivan, Chih-Hung Lee, 2008, Journal of Pharmacology and Experimental Therapeutics.

Identification of SSF1, CNS1, and HCH1 as multicopy suppressors of a Saccharomyces cerevisiae Hsp90 loss-of-function mutation.

S. Lindquist, D. Nathan, M. Vos, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Neurodegenerative Disorders: Spotlight on Sphingolipids

M. Vos, Frida Mandik, 2021, International journal of molecular sciences.

Ceramide accumulation induces mitophagy and impairs β-oxidation in PINK1 deficiency

I. König, M. Vos, T. Lohnau, 2021, Proceedings of the National Academy of Sciences.

The Importance of Drosophila melanogaster Research to UnCover Cellular Pathways Underlying Parkinson’s Disease

C. Klein, M. Vos, 2021, Cells.

Quantification of TRPV1 Protein Levels in Rat Tissues to Understand its Physiological Roles

M. Chiu, R. Moreland, C. Faltynek, 2013, Journal of Molecular Neuroscience.

Aconitase Causes Iron Toxicity in Drosophila pink1 Mutants

P. Verstreken, G. Esposito, Jef Swerts, 2013, PLoS genetics.

TRPV1b overexpression negatively regulates TRPV1 responsiveness to capsaicin, heat and low pH in HEK293 cells

T. Neelands, R. Moreland, P. Kroeger, 2006, Journal of neurochemistry.

A new model for fatty acid hydroxylase-associated neurodegeneration reveals mitochondrial and autophagy abnormalities

Bernd Wilken, M. Vos, C. Klein, 2022, Frontiers in Cell and Developmental Biology.

The Yeast Complex I Equivalent NADH Dehydrogenase Rescues pink1 Mutants

P. Verstreken, B. de Strooper, G. Esposito, 2012, PLoS genetics.

Mutations in the intellectual disability gene Ube2a cause neuronal dysfunction and impair parkin-dependent mitophagy.

P. Verstreken, B. de Strooper, H. Van Esch, 2013, Molecular cell.

Role of ceramides and sphingolipidsin Parkinson's disease.

Andrew A. Hicks, M. Vos, C. Klein, 2023, Journal of molecular biology.

Stimulation of electron transport as potential novel therapy in Parkinson's disease with mitochondrial dysfunction.

P. Verstreken, C. Klein, M. Vos, 2015, Biochemical Society transactions.

Sleep dysfunction in Parkinson's disease: Novel molecular mechanism and implications for therapy

M. Dulović, M. Vos, 2018, Movement disorders : official journal of the Movement Disorder Society.

Ceramide-induced mitophagy impairs ß-oxidation-linked energy production in PINK1 deficiency

M. Vos, C. Klein, 2022, Autophagy.

Synaptic Mitochondria in Synaptic Transmission and Organization of Vesicle Pools in Health and Disease

P. Verstreken, Elsa Lauwers, M. Vos, 2010, Front. Syn. Neurosci..

Cardiolipin promotes electron transport between ubiquinone and complex I to rescue PINK1 deficiency

Klein, Böhm, Rakovic, 2017 .

Cardiolipin promotes electron transport between ubiquinone and complex I to rescue PINK1 deficiency

Böhm, Klein, Rakovic, 2017 .

Therapeutic strategies in Parkinson's disease: what we have learned from animal models

P. Verstreken, J. S. Valadas, M. Vos, 2015, Annals of the New York Academy of Sciences.

A. Gomtsyan, J. Sullivan, Chih-Hung Lee, 2008, Journal of Pharmacology and Experimental Therapeutics.