L. Kaufman

发表

Rap1 signaling contributes to podocyte dysfunction in congenital nephrotic syndrome caused by mutations in MAGI2. Kidney International, 96(3), 642-

V. D’Agati, C. Sawyers, J. Wong, 2019 .

Disruption of MAGI2-RapGEF2-Rap1 signaling contributes to podocyte dysfunction in congenital nephrotic syndrome caused by mutations in MAGI2.

V. D’Agati, C. Sawyers, J. Wong, 2019, Kidney international.

SHROOM3-FYN Interaction Regulates Nephrin Phosphorylation and Affects Albuminuria in Allografts.

J. Wong, J. He, P. Cravedi, 2018, Journal of the American Society of Nephrology : JASN.

Role of ubiquitin-like protein FAT10 in epithelial apoptosis in renal disease.

V. D’Agati, P. Wilson, M. Ross, 2006, Journal of the American Society of Nephrology : JASN.

The pathogenesis of HIV-associated nephropathy.

P. Klotman, S. Collins, L. Kaufman, 2010, Advances in chronic kidney disease.

Up-regulation of the Homophilic Adhesion Molecule Sidekick-1 in Podocytes Contributes to Glomerulosclerosis*

Y. Hata, V. D’Agati, J. He, 2010, The Journal of Biological Chemistry.

Podocyte-specific deletion of signal transducer and activator of transcription 3 attenuates nephrotoxic serum-induced glomerulonephritis

D. Salant, J. He, P. Chuang, 2013, Kidney international.

Proximal Tubules Forget "Self- Eating" When They Meet Western Meals

B. Luxon, M. Kane, L. Denner, 2013 .

(2019). Disruption of MAGI2-RapGEF2-Rap1 signaling contributes to podocyte dysfunction in congenital nephrotic syndrome caused by mutations in MAGI2. Kidney (3), 642-655.

V. D’Agati, C. Sawyers, F. Hildebrandt, 2019 .

SARS-CoV-2 viral protein ORF3A injures renal tubules by interacting with TRIM59 to induce STAT3 activation

A. García-Sastre, Weibin Zhou, Kyung Lee, 2022, Molecular Therapy.

Role of the retinoic acid receptor-α in HIV-associated nephropathy.

V. D’Agati, N. Chen, J. He, 2011, Kidney international.

AMPK mediates regulation of glomerular volume and podocyte survival

L. Cantley, S. Ishibe, J. He, 2021, JCI insight.

AMP-Kinase mediates regulation of glomerular volume and podocyte survival

L. Cantley, S. Ishibe, J. He, 2021, bioRxiv.

Definition of the critical domains required for homophilic targeting of mouse sidekick molecules

P. Klotman, M. D. Ross, L. Kaufman, 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Transcriptome-wide association analysis identifies DACH1 as a kidney disease risk gene that contributes to fibrosis.

Christopher D. Brown, A. Hung, Chengxiang Qiu, 2021, The Journal of clinical investigation.

Mutations in six nephrosis genes delineate a pathogenic pathway amenable to treatment

S. Mane, D. Lowy, K. Nakayama, 2018, Nature Communications.

The Hippo pathway regulator KIBRA promotes podocyte injury by inhibiting YAP signaling and disrupting actin cytoskeletal dynamics

J. He, E. Azeloglu, R. Gordon, 2017, The Journal of Biological Chemistry.

DACH1 protects podocytes from experimental diabetic injury and modulates PTIP-H3K4Me3 activity.

K. Zen, V. D’Agati, Zhihong Liu, 2021, The Journal of clinical investigation.

HIV-1 Infection Initiates an Inflammatory Cascade in Human Renal Tubular Epithelial Cells

P. Klotman, Yueyue Shi, Weijia Zhang, 2006, Journal of acquired immune deficiency syndromes.

Expression of HIV transgene aggravates kidney injury in diabetic mice

Edward Y. Chen, E. Chen, Avi Ma’ayan, 2013, Kidney international.

Deletion of podocyte STAT3 mitigates the entire spectrum of HIV-1-associated nephropathy

J. He, P. Klotman, P. Chuang, 2013, AIDS.

The homophilic adhesion molecule sidekick‐1 contributes to augmented podocyte aggregation in HIV‐associated nephropathy

James R. Ashby, P. Klotman, M. Klotman, 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Sidekick-1 is upregulated in glomeruli in HIV-associated nephropathy.

P. Klotman, M. D. Ross, M. Ross, 2004, Journal of the American Society of Nephrology : JASN.

Incidence of first unprovoked seizure among children in Washington Heights, New York City, 1990-1994

W. Hauser, D. Hesdorffer, L. Kaufman, 1996 .

Acute Kidney Injury in Hospitalized Patients with COVID-19

Benjamin S. Glicksberg, A. Just, M. Danieletto, 2020, medRxiv.

Essential role of poly(ADP-ribosyl)ation in cocaine action

Clarisse H. Panganiban, E. Nestler, R. Neve, 2014, Proceedings of the National Academy of Sciences.

Lack of response to suramin in patients with AIDS and AIDS-related complex.

P. Volberding, J. Levy, M. Gottlieb, 1987, The American journal of medicine.

MAGI-1 Interacts with Nephrin to Maintain Slit Diaphragm Structure through Enhanced Rap1 Activation in Podocytes*

V. D’Agati, J. He, F. Grahammer, 2016, The Journal of Biological Chemistry.

Outcomes of Patients on Maintenance Dialysis Hospitalized with COVID-19.

Benjamin S. Glicksberg, Z. Fayad, E. Bottinger, 2020, Clinical journal of the American Society of Nephrology : CJASN.

Podocyte-specific RAP1GAP expression contributes to focal segmental glomerulosclerosis-associated glomerular injury.

V. D’Agati, J. He, P. Klotman, 2014, The Journal of clinical investigation.

Podocyte-specific RAP1GAP expression contributes to focal segmental glomerulosclerosis-associated glomerular injury.

V. D’Agati, J. He, P. Klotman, 2014, The Journal of clinical investigation.