C. Madry

发表

The N-Terminal Domains of both NR1 and NR2 Subunits Determine Allosteric Zn2+ Inhibition and Glycine Affinity of N-Methyl-d-aspartate Receptors

H. Betz, B. Laube, C. Madry, 2007, Molecular Pharmacology.

Potentiation of Glycine-Gated NR1/NR3A NMDA Receptors Relieves Ca2+-Dependent Outward Rectification

J. R. Geiger, H. Betz, B. Laube, 2009, Front. Mol. Neurosci..

P2Y13 receptors regulate microglial morphology, surveillance, and resting levels of interleukin 1β release

D. Attwell, I. Arancibia-Cárcamo, B. Robaye, 2019, Glia.

Pannexin 1-a novel regulator of acute hypoxic pulmonary vasoconstriction.

W. Kuebler, K. Connelly, J. Grune, 2021, Cardiovascular research.

Formation of NR1/NR2 and NR1/NR3 Heterodimers Constitutes the Initial Step in N-Methyl-D-aspartate Receptor Assembly*

B. Laube, Ingo Bartholomäus, C. Madry, 2008, Journal of Biological Chemistry.

Principal role of NR3 subunits in NR1/NR3 excitatory glycine receptor function.

A. Nicke, H. Betz, B. Laube, 2007, Biochemical and biophysical research communications.

The N-terminal domains of both NR 1 and NR 2 subunits determine allosteric Zn 2 + inhibition and glycine affinity of NMDA receptors

H. Betz, B. Laube, C. Madry, 2007 .

Supralinear potentiation of NR1/NR3A excitatory glycine receptors by Zn2+ and NR1 antagonist

H. Betz, B. Laube, Jörg R. P. Geiger, 2008, Proceedings of the National Academy of Sciences.

Positive Modulatory Interactions of NMDA Receptor GluN1/2B Ligand Binding Domains Attenuate Antagonists Activity

B. Laube, B. Klein, B. Costa, 2017, Front. Pharmacol..

Characterisation of C101248: A novel selective THIK-1 channel inhibitor for the modulation of microglial NLRP3-inflammasome

Justin J. W. Powell, E. Kavanagh, D. Brough, 2022, Neuropharmacology.

Amyloid β oligomers constrict human capillaries in Alzheimer’s disease via signaling to pericytes

D. Attwell, Hui Gong, S. Brandner, 2019, Science.

Effects of the ecto-ATPase apyrase on microglial ramification and surveillance reflect cell depolarization, not ATP depletion

D. Attwell, N. Hamilton, I. Arancibia-Cárcamo, 2018, Proceedings of the National Academy of Sciences.

Microglial Ramification, Surveillance, and Interleukin-1β Release Are Regulated by the Two-Pore Domain K+ Channel THIK-1

D. Attwell, R. Jolivet, R. Bryan, 2017, Neuron.

The N-terminal domain of the GluN3A subunit determines the efficacy of glycine-activated NMDA receptors

H. Betz, B. Laube, Kirsten Geider, 2016, Neuropharmacology.

Ion Channels and Receptors as Determinants of Microglial Function

D. Attwell, C. Madry, P. Izquierdo, 2019, Trends in Neurosciences.

Receptors, Ion Channels, and Signaling Mechanisms Underlying Microglial Dynamics*

D. Attwell, C. Madry, 2015, The Journal of Biological Chemistry.

Amyloid β oligomers constrict human capillaries in Alzheimer’s disease via signalling to pericytes

D. Attwell, Hui Gong, S. Brandner, 2018, bioRxiv.

The role of pannexin hemichannels in the anoxic depolarization of hippocampal pyramidal cells

D. Attwell, C. Madry, C. Haglerød, 2010, Brain : a journal of neurology.

Characterisation of C101248: A novel selective THIK-1 channel inhibitor for the modulation of microglial NLRP3-inflammasome

Justin J. W. Powell, E. Kavanagh, D. Brough, 2022, Neuropharmacology.

Interaction between mGluR8 and Calcium Channels in Photoreceptors Is Sensitive to Pertussis Toxin and Occurs Via G Protein βγ Subunit Signaling

P. Koulen, Jiyuan Liu, C. Madry, 2005 .

Progesterone Potentiates IP3-Mediated Calcium Signaling Through Akt/PKB

P. Koulen, Meharvan Singh, R. S. Duncan, 2008, Cellular Physiology and Biochemistry.

Differentially distributed IP3 receptors and Ca2+ signaling in rod bipolar cells.

P. Koulen, Jiyuan Liu, C. Madry, 2005, Investigative ophthalmology & visual science.

Progesterone potentiates calcium release through IP3 receptors by an Akt-mediated mechanism in hippocampal neurons.

P. Koulen, Meharvan Singh, R. S. Duncan, 2009, Cell calcium.

Interaction between mGluR8 and calcium channels in photoreceptors is sensitive to pertussis toxin and occurs via G protein betagamma subunit signaling.

P. Koulen, Jiyuan Liu, C. Madry, 2005, Investigative Ophthalmology and Visual Science.

Amyloidosis and ageing have differential effects on microglial Ca2+ activity in the mouse brain

D. Attwell, R. Jolivet, C. Madry, 2023, bioRxiv.

Analysis of Signaling Mechanisms Regulating Microglial Process Movement.

D. Attwell, R. Jolivet, C. Madry, 2019, Methods in molecular biology.

Synaptic Plasticity in Mammalian Brain Resting Microglial Motility Is Independent of

D. Attwell, B. MacVicar, Hyun B Choi, 2015 .

Receptors, Ion Channels, and Signaling Mechanisms Underlying Microglial Dynamics*

D. Attwell, C. Madry, 2015, The Journal of Biological Chemistry.

Microglia shape hippocampal networks but are dispensable for pruning of synapses during development

I. Vida, C. Madry, David A. D. Munro, 2023, bioRxiv.

Amyloid plaques and normal ageing have differential effects on microglial Ca2+ activity in the mouse brain

D. Attwell, C. Madry, P. Izquierdo, 2023, Pflugers Archiv : European journal of physiology.

Differential contribution of THIK-1 K+ channels and P2X7 receptors to ATP-mediated neuroinflammation by human microglia

M. Lizio, P. Fidzinski, B. Ossola, 2024, Journal of neuroinflammation.

Lifelong absence of microglia alters hippocampal glutamatergic networks but not synapse and spine density

C. Madry, David A. D. Munro, Ali Rifat, 2024, EMBO reports.