J. Arranz-Tagarro

发表

The Neuroprotection Exerted by Memantine, Minocycline and Lithium, against Neurotoxicity of CSF from Patients with Amyotrophic Lateral Sclerosis, Is Antagonized by Riluzole

J. Matías‐Guiu, E. Martı́nez-Vila, L. Galán, 2013, Neurodegenerative Diseases.

Stabilizers of neuronal and mitochondrial calcium cycling as a strategy for developing a medicine for Alzheimer's disease.

Antonio G. García, J. Arranz-Tagarro, J. Padín, 2012, ACS chemical neuroscience.

Novel sulfoglycolipid IG20 causes neuroprotection by activating the phase II antioxidant response in rat hippocampal slices

R. León, Manuela G. López, Antonio G. García, 2017, Neuropharmacology.

Cerebrospinal fluid cytotoxicity does not affect survival in amyotrophic lateral sclerosis

J. Matías‐Guiu, V. Pytel, L. Galán, 2017, Acta neurologica Scandinavica.

Cellular changes in motor neuron cell culture produced by cytotoxic cerebrospinal fluid from patients with amyotrophic lateral sclerosis

J. Matías‐Guiu, L. Galán, Antonio G. García, 2014 .

Cellular changes in motor neuron cell culture produced by cytotoxic cerebrospinal fluid from patients with amyotrophic lateral sclerosis.

J. Matías‐Guiu, L. Galán, U. Gómez-Pinedo, 2014, Neurologia.

Histological changes in the rat brain and spinal cord following prolonged intracerebroventricular infusion of cerebrospinal fluid from amyotrophic lateral sclerosis patients are similar to those caused by the disease.

J. Matías‐Guiu, F. López-Sosa, J. Barcia, 2016, Neurologia.

Gramine Derivatives Targeting Ca(2+) Channels and Ser/Thr Phosphatases: A New Dual Strategy for the Treatment of Neurodegenerative Diseases.

L. Gandía, J. Arranz-Tagarro, L. González-Lafuente, 2016, Journal of medicinal chemistry.

Histological changes in the rat brain and spinal cord following prolonged intracerebroventricular infusion of cerebrospinal fluid from amyotrophic lateral sclerosis patients are similar to those caused by the disease

J. Matías‐Guiu, J. Barcia, L. Galán, 2018 .

Hypoxia-elicited catecholamine release is controlled by L-type as well as N/PQ types of calcium channels in rat embryo chromaffin cells.

J. Arranz-Tagarro, J. Padín, J. Fernández‐Morales, 2014, American journal of physiology. Cell physiology.

Plasmalemmal sodium-calcium exchanger shapes the calcium and exocytotic signals of chromaffin cells at physiological temperature.

L. Gandía, R. de Pascual, R. Olivares, 2013, American journal of physiology. Cell physiology.

Recent patents on calcium channel blockers: emphasis on CNS diseases

J. Arranz-Tagarro, C. de los Ríos, J. Padín, 2014, Expert opinion on therapeutic patents.

Function of AT1 and AT2 receptors in atrial contractions from spontaneous hypertensive and diabetic‐induced streptozotocin rats

A. Jurkiewicz, J. Arranz-Tagarro, R. Miranda-Ferreira, 2018, Clinical and experimental pharmacology & physiology.

Altered mitochondrial function, calcium signaling, and catecholamine release in chromaffin cells of diabetic and SHR rats

A. Jurkiewicz, J. Arranz-Tagarro, R. Miranda-Ferreira, 2017, European journal of pharmacology.

Altered mitochondrial function, capacitative calcium entry and contractions in the aorta of hypertensive rats

J. Velasco-Martín, J. Regadera, A. Jurkiewicz, 2017, Journal of hypertension.

Depressed excitability and ion currents linked to slow exocytotic fusion pore in chromaffin cells of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

L. Gandía, R. de Pascual, J. Arranz-Tagarro, 2015, American journal of physiology. Cell physiology.

CALL FOR PAPERS Omic and Systems Biology Approaches in Neurodegenerative

Antonio G. García, L. Gandía, J. Arranz-Tagarro, 2015 .

Functional Upregulation of STIM-1/Orai-1-Mediated Store-Operated Ca2+ Contributing to the Hypertension Development Elicited by Chronic EtOH Consumption.

A. Jurkiewicz, D. Roman-Campos, J. Arranz-Tagarro, 2017, Current Vascular Pharmacology.