R. Bouchard

发表

The Bcl-2 Homology-3 Domain (BH3)-Only Proteins, Bid, DP5/Hrk, and BNip3L, Are Upregulated in Reactive Astrocytes of End-Stage Mutant SOD1 Mouse Spinal Cord

D. Linseman, N. Duval, H. Wilkins, 2018, Front. Cell. Neurosci..

C-terminal binding proteins are essential pro-survival factors that undergo caspase-dependent downregulation during neuronal apoptosis

Trisha R. Stankiewicz, D. Linseman, Emily K. Schroeder, 2013, Molecular and Cellular Neuroscience.

Calpain Plays a Central Role in 1-Methyl-4-phenylpyridinium (MPP(+))-Induced Neurotoxicity in Cerebellar Granule Neurons

D. Linseman, H. Wilkins, K. Ryan, 2011, Neurotoxicity Research.

Caspases indirectly regulate cleavage of the mitochondrial fusion GTPase OPA1 in neurons undergoing apoptosis

C. Blackstone, D. Linseman, J. Brewster, 2009, Brain Research.

Green tea epigallocatechin 3-gallate accumulates in mitochondria and displays a selective antiapoptotic effect against inducers of mitochondrial oxidative stress in neurons.

D. Linseman, Elise R Breed, Emily K. Schroeder, 2009, Antioxidants & redox signaling.

Glutathione Binding to the Bcl-2 Homology-3 Domain Groove

K. Tyler, D. Linseman, Emily K. Schroeder, 2007, Journal of Biological Chemistry.

Caspase Inhibition Protects against Reovirus-Induced Myocardial Injury In Vitro and In Vivo

C. Long, R. DeBiasi, K. Tyler, 2004, Journal of Virology.

Fully automated software for quantitative measurements of mitochondrial morphology.

A. Keller, J. Reusch, P. McClatchey, 2016, Mitochondrion.

Dipeptidyl peptidase-4 inhibition in diabetic rats leads to activation of the transcription factor CREB in β-cells.

S. Pugazhenthi, R. Bouchard, Limei Qin, 2015, European journal of pharmacology.

Induction of an Inflammatory Loop by Interleukin-1β and Tumor Necrosis Factor-α Involves NF-kB and STAT-1 in Differentiated Human Neuroprogenitor Cells

S. Pugazhenthi, Yuji Zhang, R. Bouchard, 2013, PloS one.

A MOLECULAR BASIS FOR BCL-2 ANTIOXIDANT FUNCTION AT MITOCHONDRIA*

K. Tyler, D. Linseman, Emily K. Schroeder, 2008 .

Dominant negative mutant forms of the cAMP response element binding protein induce apoptosis and decrease the anti-apoptotic action of growth factors in human islets

J. Reusch, S. Pugazhenthi, R. Gill, 2007, Diabetologia.

Novel Nuclear Herniations Induced by Nuclear Localization of a Viral Protein

K. Tyler, R. Bouchard, C. Hoyt, 2004, Journal of Virology.

Regulation of cyclic AMP response element‐binding protein during neuroglial interactions

S. Pugazhenthi, R. Bouchard, Limei Qin, 2016, Journal of neurochemistry.

A myocyte enhancer factor 2D (MEF2D) kinase activated during neuronal apoptosis is a novel target inhibited by lithium.

M. K. Meintzer, T. Laessig, K. Heidenreich, 2003, Journal of neurochemistry.

Suppression of Death Receptor Signaling in Cerebellar Purkinje Neurons Protects Neighboring Granule Neurons from Apoptosis via an Insulin-like Growth Factor I-dependent Mechanism*

T. Laessig, K. Heidenreich, D. Linseman, 2002, The Journal of Biological Chemistry.

N-terminal cleavage of the mitochondrial fusion GTPase OPA1 occurs via a caspase-independent mechanism in cerebellar granule neurons exposed to oxidative or nitrosative stress

C. Blackstone, D. Linseman, N. Duval, 2013, Brain Research.

The permeability transition pore triggers Bax translocation to mitochondria during neuronal apoptosis

T. Laessig, K. Heidenreich, B. Butts, 2005, Cell Death and Differentiation.

Distinct mechanisms of neuronal apoptosis are triggered by antagonism of Bcl‐2/Bcl‐x(L) versus induction of the BH3‐only protein Bim

K. Heidenreich, B. Butts, D. Linseman, 2005, Journal of neurochemistry.

Varicella zoster virus infection of human fetal lung cells alters mitochondrial morphology

A. Shahzad, R. Cohrs, A. Keller, 2016, Journal of NeuroVirology.

The p75 Neurotrophin Receptor Can Induce Autophagy and Death of Cerebellar Purkinje Neurons

T. Laessig, K. Heidenreich, P. Barker, 2004, The Journal of Neuroscience.

Inactivation of the Myocyte Enhancer Factor-2 Repressor Histone Deacetylase-5 by Endogenous Ca2//Calmodulin-dependent Kinase II Promotes Depolarization-mediated Cerebellar Granule Neuron Survival*

M. K. Meintzer, T. Laessig, K. Heidenreich, 2003, Journal of Biological Chemistry.

Differential Mitochondrial Adaptation in Primary Vascular Smooth Muscle Cells from a Diabetic Rat Model

A. Keller, L. Walker, J. Reusch, 2016, Oxidative medicine and cellular longevity.

Glycogen Synthase Kinase-3β Phosphorylates Bax and Promotes Its Mitochondrial Localization during Neuronal Apoptosis

T. Laessig, K. Heidenreich, B. Butts, 2004, The Journal of Neuroscience.

A myocyte enhancer factor 2D (MEF2D) kinase activated during neuronal apoptosis is a novel target inhibited by lithium

M. K. Meintzer, T. Laessig, K. Heidenreich, 2003 .

K. Heidenreich, B. Varnum, J. Pawlowski, 2007, Endocrinology.

Insulin-Like Growth Factor-I Blocks Bcl-2 Interacting Mediator of Cell Death (Bim) Induction and Intrinsic Death Signaling in Cerebellar Granule Neurons

T. Laessig, K. Heidenreich, D. Linseman, 2002, The Journal of Neuroscience.

Neuroprotective actions of Glucagon‐like peptide‐1 in differentiated human neuroprogenitor cells

S. Pugazhenthi, K. Velmurugan, R. Bouchard, 2012, Journal of neurochemistry.

CREB Downregulation in Vascular Disease: A Common Response to Cardiovascular Risk

A. Chait, P. Watson, J. Reusch, 2010, Arteriosclerosis, thrombosis, and vascular biology.

Inhibition of Rac GTPase triggers a c‐Jun‐ and Bim‐dependent mitochondrial apoptotic cascade in cerebellar granule neurons

E. Kandel, K. Tyler, K. Heidenreich, 2005, Journal of neurochemistry.

Neuronal Apoptosis Induced by Selective Inhibition of Rac GTPase versus Global Suppression of Rho Family GTPases Is Mediated by Alterations in Distinct Mitogen-activated Protein Kinase Signaling Cascades*

Trisha R. Stankiewicz, D. Linseman, K. Aktories, 2015, The Journal of Biological Chemistry.

The pesticide rotenone induces caspase‐3‐mediated apoptosis in ventral mesencephalic dopaminergic neurons

K. Heidenreich, D. Linseman, Susan M. Jones, 2003, Journal of neurochemistry.

Cytokine-mediated Down-regulation of the Transcription Factor cAMP-response Element-binding Protein in Pancreatic β-Cells*

L. Boxer, J. Reusch, S. Pugazhenthi, 2003, Journal of Biological Chemistry.

Endoplasmic reticulum stress and trophic factor withdrawal activate distinct signaling cascades that induce glycogen synthase kinase-3β and a caspase-9-dependent apoptosis in cerebellar granule neurons

K. Heidenreich, D. Linseman, J. Brewster, 2006, Molecular and Cellular Neuroscience.

Signal Transducer and Activator of Transcription-5 Mediates Neuronal Apoptosis Induced by Inhibition of Rac GTPase Activity*

M. Nevalainen, K. Tyler, Trisha R. Stankiewicz, 2012, The Journal of Biological Chemistry.

Proteasome inhibition elicits a biphasic effect on neuronal apoptosis via differential regulation of pro-survival and pro-apoptotic transcription factors

K. Heidenreich, B. Butts, D. Linseman, 2005, Molecular and Cellular Neuroscience.

Dysregulation of Rac or Rho elicits death of motor neurons and activation of these GTPases is altered in the G93A mutant hSOD1 mouse model of amyotrophic lateral sclerosis

Trisha R. Stankiewicz, D. Linseman, R. Bouchard, 2020, Neurobiology of Disease.

Laser capture microdissection of neurons from differentiated human neuroprogenitor cells in culture.

S. Pugazhenthi, R. Bouchard, Thomas Chong, 2013, Journal of visualized experiments : JoVE.

C-terminal binding proteins are essential pro-survival factors that undergo caspase-dependent downregulation during neuronal apoptosis

Trisha R. Stankiewicz, D. Linseman, Emily K. Schroeder, 2013, Molecular and Cellular Neuroscience.

Glycogen synthase kinase-3beta phosphorylates Bax and promotes its mitochondrial localization during neuronal apoptosis.

T. Laessig, D. Linseman, M. Florez-McClure, 2004, Journal of Neuroscience.