J. Movassat

发表

PDX-1 and cell-cell contact act in synergy to promote delta-cell development in a human pancreatic endocrine precursor cell line.

F. Levine, G. Beattie, A. Hayek, 2000, Molecular endocrinology.

Neonatally Streptozotocin- Induced (n-STZ) Diabetic Rats: A Family of Type 2 Diabetes Models

M. Dolz, B. Portha, P. Serradas, 2007 .

The Goto-Kakizaki rat is a spontaneous prototypical rodent model of polycystic ovary syndrome

D. Monniaux, N. Roblot, D. Farabos, 2021, Nature Communications.

Expression of the kynurenine pathway enzymes in the pancreatic islet cells. Activation by cytokines and glucolipotoxicity.

B. Portha, B. Gausserès, J. Liu, 2015, Biochimica et biophysica acta.

Exendin 4 up-regulates expression of PDX 1 and hastens differentiation and maturation of human fetal pancreatic cells.

Ana D. Lopez, G. Beattie, A. Hayek, 2002, The Journal of clinical endocrinology and metabolism.

Type 2 diabetes – a matter of failing β‐cell neogenesis? Clues from the GK rat model

C. Álvarez, E. Fernández, M. A. Martín, 2007, Diabetes, obesity & metabolism.

A Holistic View of the Goto-Kakizaki Rat Immune System: Decreased Circulating Immune Markers in Non- Obese Type 2 Diabetes

J. D. Turner, M. Darnaudéry, J. Movassat, 2022, Frontiers in Immunology.

Implication of glycogen synthase kinase 3 in diabetes-associated islet inflammation.

P. Cattan, B. Mégarbane, B. Portha, 2020, The Journal of endocrinology.

Diabetic β-Cells Can Achieve Self-Protection against Oxidative Stress through an Adaptive Up-Regulation of Their Antioxidant Defenses

G. Lacraz, A. Galinier, N. Kassis, 2009, PloS one.

Defective functional β-cell mass and Type 2 diabetes in the Goto–Kakizaki rat model

M. Dolz, G. Lacraz, F. Homo-Delarche, 2007, Expert review of endocrinology & metabolism.

beta-cell function and viability in the spontaneously diabetic GK rat: information from the GK/Par colony.

G. Mithieux, F. Rajas, B. Portha, 2001, Diabetes.

Impaired development of pancreatic beta-cell mass is a primary event during the progression to diabetes in the GK rat

B. Portha, P. Serradas, J. Movassat, 1997, Diabetologia.

Disrupted dynamics of F-actin and insulin granule fusion in INS-1 832/13 beta-cells exposed to glucotoxicity: partial restoration by glucagon-like peptide 1.

B. Portha, B. Gausserès, C. Tourrel-Cuzin, 2016, Biochimica et biophysica acta.

Interleukin-7 Regulates Adipose Tissue Mass and Insulin Sensitivity in High-Fat Diet-Fed Mice through Lymphocyte-Dependent and Independent Mechanisms

P. Froguel, J. D. Di Santo, C. Spriet, 2012, PloS one.

Impaired β-cell regeneration after partial pancreatectomy in the adult Goto-Kakizaki rat, a spontaneous model of type II diabetes

B. Portha, C. Plachot, J. Movassat, 2001, Histochemistry and Cell Biology.

A euglycaemic/non-diabetic perinatal environment does not alleviate early beta cell maldevelopment and type 2 diabetes risk in the GK/Par rat model

J. Renard, B. Portha, J. Movassat, 2012, Diabetologia.

Kynurenine-3-monooxygenase expression is activated in the pancreatic endocrine cells by diabetes and its blockade improves glucose-stimulated insulin secretion.

C. Lim, G. Guillemin, T. Domet, 2022, Biochimica et biophysica acta. Molecular basis of disease.

Reduced up-regulation of the nitric oxide pathway and impaired endothelial and smooth muscle functions in the female type 2 diabetic goto-kakizaki rat heart

M. Bernard, M. Desrois, C. Lan, 2017, Nutrition & Metabolism.

Beta-cell growth in the neonatal Goto-Kakisaki rat and regeneration after treatment with streptozotocin at birth

B. Portha, J. Movassat, 1999, Diabetologia.

COUP-TFII Controls Mouse Pancreatic β-Cell Mass through GLP-1-β-Catenin Signaling Pathways

M. Vasseur-Cognet, P. Petit, K. Maedler, 2012, PloS one.

Keratinocyte growth factor and beta-cell differentiation in human fetal pancreatic endocrine precursor cells

G. Beattie, A. Hayek, B. Portha, 2003, Diabetologia.

cAMP-secretion coupling is impaired in diabetic GK/Par rat β-cells: a defect counteracted by GLP-1.

M. Dolz, B. Portha, M. Giroix, 2011, American journal of physiology. Endocrinology and metabolism.

Local in vivo GSK3β knockdown promotes pancreatic β cell and acinar cell regeneration in 90% pancreatectomized rat.

B. Portha, J. Movassat, D. Bailbé, 2012, Molecular therapy : the journal of the American Society of Gene Therapy.

Underlying mechanisms of glucocorticoid-induced β-cell death and dysfunction: a new role for glycogen synthase kinase 3

S. Tolu, P. Cattan, M. Armanet, 2021, Cell Death & Disease.

Involvement of P2Y signaling in the restoration of glucose‐induced insulin exocytosis in pancreatic β cells exposed to glucotoxicity

S. Tolu, C. Tourrel-Cuzin, J. Movassat, 2021, Journal of cellular physiology.

Beta-cell mass depletion precedes the onset of hyperglycaemia in the GK rat, a genetic model of non-insulin-dependent diabetes mellitus.

B. Portha, J. Movassat, C. Saulnier, 1995, Diabete & metabolisme.

Emerging role for kynurenines in metabolic pathologies

B. Portha, J. Movassat, Jun-Jun Liu, 2019, Current opinion in clinical nutrition and metabolic care.

Early administration of keratinocyte growth factor improves {beta}-cell regeneration in rat with streptozotocin-induced diabetes.

B. Portha, J. Movassat, 2007, The Journal of endocrinology.

Impaired beta-cell regeneration after partial pancreatectomy in the adult Goto-Kakizaki rat, a spontaneous model of type II diabetes.

B. Portha, C. Plachot, J. Movassat, 2001, Histochemistry and cell biology.

Early administration of keratinocyt e growth factor improves b-cell regeneration in rat with streptozotocin-induced diabetes

B. Portha, J. Movassat, 2007 .

Type 2 Diabetes Mellitus and Alzheimer’s Disease: Shared Molecular Mechanisms and Potential Common Therapeutic Targets

S. Tolu, N. Janel, J. Movassat, 2022, International journal of molecular sciences.

One-carbon cycle alterations induced by Dyrk1a dosage

J. Delabar, N. Janel, C. Noll, 2014, Molecular genetics and metabolism reports.

Preconception exposure of adult male rats to bisphenol S impairs insulin sensitivity and glucose tolerance in their male offspring.

S. Tolu, P. Gong, J. Movassat, 2022, Chemosphere.

P2-type purinergic signaling in the regulation of pancreatic β-cell functional plasticity as a promising novel therapeutic approach for the treatment of type 2 diabetes?

C. Tourrel-Cuzin, J. Movassat, Nour Mesto, 2022, Frontiers in Endocrinology.

Early environmental factors, alteration of epigenetic marks and metabolic disease susceptibility.

V. Mezger, B. Portha, A. Fournier, 2014, Biochimie.

Mother or Father: Who Is in the Front Line? Mechanisms Underlying the Non-Genomic Transmission of Obesity/Diabetes via the Maternal or the Paternal Line

B. Portha, V. Grandjean, J. Movassat, 2019, Nutrients.

Insulin Administration Enhances Growth of the β-Cell Mass in Streptozotocin-Treated Newborn Rats

B. Portha, J. Movassat, C. Saulnier, 1997, Diabetes.

The GK rat: a prototype for the study of non-overweight type 2 diabetes.

B. Portha, M. Giroix, C. Tourrel-Cuzin, 2012, Methods in molecular biology.

Islet structure and function in the GK rat.

G. Lacraz, F. Homo-Delarche, M. Gangnerau, 2010, Advances in experimental medicine and biology.

The GK rat beta-cell: A prototype for the diseased human beta-cell in type 2 diabetes?

M. Dolz, G. Lacraz, F. Homo-Delarche, 2009, Molecular and Cellular Endocrinology.

Maternal diabetes, programming of beta-cell disorders and intergenerational risk of type 2 diabetes.

B. Portha, J. Movassat, D. Bailbé, 2014, Diabetes & metabolism.

Impact and Mechanisms of Pancreatic Beta-Cell Mass Programming by Maternal Diabetes - Insight from Animal Model Studies

B. Portha, J. Movassat, A. Chavey, 2011 .

Early-Life Origins of Type 2 Diabetes: Fetal Programming of the Beta-Cell Mass

B. Portha, J. Movassat, A. Chavey, 2011, Experimental diabetes research.

Metabolic Diseases and Down Syndrome: How Are They Linked Together?

S. Tolu, N. Janel, F. Vialard, 2021, Biomedicines.

Defective prolactin signaling impairs pancreatic β-cell development during the perinatal period.

M. Lombès, M. Freemark, N. Binart, 2013, American journal of physiology. Endocrinology and metabolism.

Hypothesis and Theory: Circulating Alzheimer's-Related Biomarkers in Type 2 Diabetes. Insight From the Goto-Kakizaki Rat

N. Janel, J. Movassat, Yuchen Gu, 2019, Front. Neurol..

Neonatal growth and regeneration of beta-cells are regulated by the Wnt/beta-catenin signaling in normal and diabetic rats.

B. Portha, B. Uzan, J. Movassat, 2010, American journal of physiology. Endocrinology and metabolism.

Paternal High-Protein Diet Programs Offspring Insulin Sensitivity in a Sex-Specific Manner

M. Stathopoulou, S. Tolu, P. Gong, 2021, Biomolecules.

Activation of the GLP-1 Receptor Signalling Pathway: A Relevant Strategy to Repair a Deficient Beta-Cell Mass

B. Portha, Bernard Portha, Cécile Tourrel-Cuzin, 2011, Experimental diabetes research.

Diabetic GK/Par rat beta-cells are spontaneously protected against H2O2-triggered apoptosis. A cAMP-dependent adaptive response.

G. Lacraz, N. Kassis, B. Portha, 2010, American journal of physiology. Endocrinology and metabolism.

The Constitutive Lack of α7 Nicotinic Receptor Leads to Metabolic Disorders in Mouse

U. Maskos, S. Pons, B. Thorens, 2020, Biomolecules.

Mechanisms of KGF Mediated Signaling in Pancreatic Duct Cell Proliferation and Differentiation

B. Portha, B. Uzan, J. Movassat, 2009, PloS one.

O-GlcNacylation Links TxNIP to Inflammasome Activation in Pancreatic β Cells

F. Pattou, T. Issad, P. Pagésy, 2019, Front. Endocrinol..

Procyanidins modulate microRNA expression in pancreatic islets.

M. Pinent, A. Ardévol, F. Sánchez-Cabo, 2013, Journal of agricultural and food chemistry.

Follow-up of GK rats during prediabetes highlights increased insulin action and fat deposition despite low insulin secretion.

E. Bertin, J. Mourot, B. Portha, 2008, American journal of physiology. Endocrinology and metabolism.

Lithium treatment mitigates the diabetogenic effects of chronic cortico-therapy.

S. Tolu, B. Uzan, J. Movassat, 2023, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.