C. Soria-Valles

发表

Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence‐accelerated mice

I. Vega-Naredo, M. Pallàs, A. Camins, 2008, Journal of pineal research.

1 Matrix Proteases and the Degradome

C. Soria-Valles, 2012 .

NF-κB activation impairs somatic cell reprogramming in ageing

G. Daley, J. Martín-Subero, C. López-Otín, 2015, Nature Cell Biology.

Antioxidant responses to variations of oxygen by the Harderian gland of different species of the superspecies Spalax ehrenbergi

A. Avivi, A. Joel, I. Vega-Naredo, 2010 .

Melatonin alters cell death processes in response to age‐related oxidative stress in the brain of senescence‐accelerated mice

I. Vega-Naredo, M. Pallàs, A. Camins, 2009, Journal of pineal research.

Sexual autophagic differences in the androgen-dependent flank organ of Syrian hamsters.

I. Vega-Naredo, B. Caballero, A. Coto-Montes, 2008, Journal of andrology.

MMP-25 Metalloprotease Regulates Innate Immune Response through NF-κB Signaling

M. S. Fernández-García, A. Ferrando, E. Colado, 2016, The Journal of Immunology.

Reprogramming aging through DOT1L inhibition

C. López-Otín, F. Osorio, C. Soria-Valles, 2015, Cell cycle.

Hallmarks of progeroid syndromes: lessons from mice and reprogrammed cells

C. López-Otín, C. Soria-Valles, Dido Carrero, 2016, Disease Models & Mechanisms.

Haematopoietic stem and progenitor cells from human pluripotent stem cells

A. Engelman, Yi-fen Lu, Patricia Sousa, 2017, Nature.

NF-κB activation impairs somatic cell reprogramming in ageing

G. Daley, J. Martín-Subero, C. López-Otín, 2015, Nature Cell Biology.

Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response.

C. López-Otín, J. Freije, A. Ramsay, 2012, Genes & development.

Analysis of the disintegrin‐metalloproteinases family reveals ADAM29 and ADAM7 are often mutated in melanoma

Jimmy Lin, C. López-Otín, Xiaomu Wei, 2011, Human mutation.

NF-κB signaling as a driver of ageing.

C. López-Otín, J. Freije, F. Osorio, 2016, International review of cell and molecular biology.

iPSCs: On the Road to Reprogramming Aging.

C. López-Otín, C. Soria-Valles, 2016, Trends in molecular medicine.

Matrix Proteases and the Degradome

C. López-Otín, A. Gutiérrez-Fernández, C. Soria-Valles, 2012 .

Loss of MT1‐MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid

M. S. Fernández-García, X. Puente, J. Gutiérrez-Abril, 2015, The EMBO journal.

Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation

V. Quesada, Diana A. Puente, C. López-Otín, 2016, Journal of Medical Genetics.

link accelerated aging to a systemic inflammatory response B activation and κ Nuclear lamina defects cause ATM-dependent NF-Material Supplemental

C. López-Otín, J. Freije, A. Ramsay, 2012 .

Effects of azacitidine on matrix metalloproteinase-9 in acute myeloid leukemia and myelodysplasia.

A. Gutiérrez-Fernández, Á. Moncada-Pazos, T. Bernal, 2013, Experimental hematology.

Loss of the proteostasis factor AIRAPL causes myeloid transformation by deregulating IGF-1 signaling

E. Colado, C. López-Otín, J. Freije, 2015, Nature Medicine.

The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21

C. López-Otín, A. Gutiérrez-Fernández, R. Gomis, 2014, Oncogene.

Immune and inflammatory responses to DNA damage in cancer and aging

C. López-Otín, F. Osorio, A. López-Soto, 2017, Mechanisms of Ageing and Development.

NF-κB signaling as a driver of ageing.

C. López-Otín, J. Freije, F. Osorio, 2016, International review of cell and molecular biology.