C. R. Bayer

发表

A novel murine model of Clostridium sordellii myonecrosis: Insights into the pathogenesis of disease.

D. Stevens, A. Bryant, M. J. Aldape, 2016, Anaerobe.

Muscle injury, vimentin expression, and nonsteroidal anti-inflammatory drugs predispose to cryptic group A streptococcal necrotizing infection.

R. Lieber, D. Stevens, A. Bryant, 2008, The Journal of infectious diseases.

Vascular dysfunction and ischemic destruction of tissue in Streptococcus pyogenes infection: the role of streptolysin O-induced platelet/neutrophil complexes.

D. Stevens, A. Bryant, P. Guth, 2005, The Journal of infectious diseases.

Group A streptococcal myonecrosis: increased vimentin expression after skeletal-muscle injury mediates the binding of Streptococcus pyogenes.

D. Stevens, A. Bryant, C. R. Bayer, 2006, The Journal of infectious diseases.

The roles of injury and nonsteroidal anti-inflammatory drugs in the development and outcomes of severe group A streptococcal soft tissue infections

D. Stevens, A. Bryant, M. J. Aldape, 2015, Current opinion in infectious diseases.

Effects of selective and nonselective nonsteroidal anti-inflammatory drugs on antibiotic efficacy of experimental group A streptococcal myonecrosis.

D. Stevens, A. Bryant, S. Hamilton, 2014, The Journal of infectious diseases.

Effects of delayed NSAID administration after experimental eccentric contraction injury – A cellular and proteomics study

Thomas L. Fillmore, B. Webb-Robertson, T. Clauss, 2017, PloS one.

Streptococcus pyogenes NAD+-Glycohydrolase Reduces Skeletal Muscle βNAD+ Levels Independently of Streptolysin O

D. Stevens, A. Bryant, Sarah E. Hobdey, 2022, Microorganisms.

Staphylococcus aureus α-hemolysin promotes platelet-neutrophil aggregate formation.

D. Stevens, A. Bryant, Devin D. Bolz, 2013, The Journal of infectious diseases.

Clostridium perfringens phospholipase C-induced platelet/leukocyte interactions impede neutrophil diapedesis.

D. Stevens, A. Bryant, R. Titball, 2006, Journal of medical microbiology.

Immunization with the C-Domain of alpha -Toxin prevents lethal infection, localizes tissue injury, and promotes host response to challenge with Clostridium perfringens.

D. Stevens, A. Bryant, R. Titball, 2004, The Journal of infectious diseases.

Activation of platelet gpIIbIIIa by phospholipase C from Clostridium perfringens involves store-operated calcium entry.

D. Stevens, A. Bryant, C. R. Bayer, 2003, The Journal of infectious diseases.