F. McGregor

发表

Retinoic Acid Receptor-a Expression in Oral Dysplasias : Correlation with Acquisition of the Immortal Phenotype 1

D. Soutar, P. Harrison, E. Wagner, 2006 .

Senescing oral dysplasias are not immortalized by ectopic expression of hTERT alone without other molecular changes, such as loss of INK4A and/or retinoic acid receptor-β: but p53 mutations are not necessarily required

E. Parkinson, K. Gordon, P. Harrison, 2003, Oncogene.

Acute Inhibition of MEK Suppresses Congenital Melanocytic Nevus Syndrome in a Murine Model Driven by Activated NRAS and Wnt Signaling

Hong Wu, K. Blyth, V. Kinsler, 2015, The Journal of investigative dermatology.

Cucurbit [7] uril encapsulated cisplatin overcomes resistance to cisplatin induced by Rab25 overexpression in an intraperitoneal ovarian cancer model

J. Plumb, N. Gomez-roman, N. Wheate, 2015, Journal of Ovarian Research.

Gastrokine 1 is abundantly and specifically expressed in superficial gastric epithelium, down‐regulated in gastric carcinoma, and shows high evolutionary conservation

K. Oien, I. Downie, W. Keith, 2004, The Journal of pathology.

Identification of blottin: A novel gastric trefoil factor family‐2 binding protein

T. Hunt, R. Poulsom, N. Wright, 2006, Proteomics.

Molecular changes associated with oral dysplasia progression and acquisition of immortality: potential for its reversal by 5-azacytidine.

E. Parkinson, P. Harrison, J. Fleming, 2002, Cancer research.

Selective ablation of human cancer cells by telomerase-specific adenoviral suicide gene therapy vectors expressing bacterial nitroreductase

I. Ganly, A. Bilsland, W. Keith, 2003, Oncogene.

Regulation of herpes simplex virus poly (A) site usage and the action of immediate-early protein IE63 in the early-late switch

A. Phelan, J. Clements, F. McGregor, 1996, Journal of virology.

Inappropriate retinoic acid receptor-beta expression in oral dysplasias: correlation with acquisition of the immortal phenotype.

D. Soutar, P. Harrison, E. Wagner, 1997, Cancer research.

Hypoxia-inducible factor 1 alpha is required for the tumourigenic and aggressive phenotype associated with Rab25 expression in ovarian cancer

J. Cassidy, J. Plumb, A. Chalmers, 2016, Oncotarget.