Y. Ishii

Tumor necrosis factor-alpha-mediated decrease in glutathione increases the sensitivity of pulmonary vascular endothelial cells to H2O2.

A. Malik, P. del Vecchio, P. Vecchio, 1992, The Journal of clinical investigation.

Rat alveolar macrophage cytokine production and regulation of neutrophil recruitment following acute ozone exposure.

F. Hirata, H. Yang, D. Bassett, 1997, Toxicology and applied pharmacology.

Expression of Th2-skewed pathology mediators in monocyte-derived type 2 of dendritic cells (DC2).

K. Akagawa, K. Nakata, K. Satoh, 2009, Immunology letters.

Neutrophil adhesion to TNFα‐activated endothelial cells potentiates leukotrine B4 production

A. Malik, Y. Ishii, S. Lo, 1992 .

Attenuation of endotoxin-induced acute lung injury by the Rho-associated kinase inhibitor, Y-27632.

A. Ishizaka, S. Tasaka, N. Hasegawa, 2005, American journal of respiratory cell and molecular biology.

Role of Rho-kinase in reexpansion pulmonary edema in rabbits.

A. Ishizaka, S. Tasaka, M. Kohno, 2005, American journal of physiology. Lung cellular and molecular physiology.

Responses of serum and lung angiotensin-converting enzyme activities in the early phase of pulmonary damage induced by oleic acid in dogs.

S. Kira, T. Nukiwa, T. Arai, 1982, The American review of respiratory disease.

Lipopolysaccharide binding protein and CD14 interaction induces tumor necrosis factor-alpha generation and neutrophil sequestration in lungs after intratracheal endotoxin.

A. Malik, S. Goyert, P. del Vecchio, 1993, Circulation research.