B. Roy

Cell-type and promoter-context dependent retinoic acid receptor (RAR) redundancies for RAR beta 2 and Hoxa-1 activation in F9 and P19 cells can be artefactually generated by gene knockouts.

P. Chambon, R. Taneja, P. Reczek, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Synergistic activation of retinoic acid (RA)-responsive genes and induction of embryonal carcinoma cell differentiation by an RA receptor alpha (RAR alpha)-, RAR beta-, or RAR gamma-selective ligand in combination with a retinoid X receptor-specific ligand

P. Chambon, R. Taneja, B. Roy, 1995, Molecular and cellular biology.

Activation function 2 (AF‐2) of retinoic acid receptor and 9‐cis retinoic acid receptor: presence of a conserved autonomous constitutive activating domain and influence of the nature of the response element on AF‐2 activity.

P. Chambon, R. Losson, B. Roy, 1994, The EMBO journal.

Reexpression of retinoic acid receptor (RAR) gamma or overexpression of RAR alpha or RAR beta in RAR gamma-null F9 cells reveals a partial functional redundancy between the three RAR types.

P. Chambon, P. Bouillet, R. Taneja, 1995, Proceedings of the National Academy of Sciences of the United States of America.

c-Myc trans-activates the p53 promoter through a required downstream CACGTG motif.

V. Rotter, D. Reisman, J. Beamon, 1993, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Transactivation of the human p53 tumor suppressor gene by c-Myc/Max contributes to elevated mutant p53 expression in some tumors

D. Reisman, J. Beamon, E. Balint, 1994, Molecular and cellular biology.

Far Upstream Element-Binding Protein 1 Binds the 3' Untranslated Region of PKD2 and Suppresses Its Translation.

C. Sergi, J. King, Song-Mei Liu, 2016, Journal of the American Society of Nephrology : JASN.