N. Rust

发表

Mutations in AP2S1 cause familial hypocalciuric hypercalcemia type 3

G. McVean, D. Buck, Lorna Gregory, 2012, Nature Genetics.

Cutting edge: rapid cloning of tumor-specific CTL suitable for adoptive immunotherapy of melanoma.

G. Ogg, P. Romero, V. Cerundolo, 1999, Journal of immunology.

Analysis of Lyn/CD22 double‐deficient B cells in vivo demonstrates Lyn‐ and CD22‐independent pathways affecting BCR regulation and B cell survival

C. Goodnow, H. Ferry, R. Cornall, 2005, European journal of immunology.

A G‐protein Subunit‐α11 Loss‐of‐Function Mutation, Thr54Met, Causes Familial Hypocalciuric Hypercalcemia Type 2 (FHH2)

M. Nesbit, R. Thakker, T. Cranston, 2016, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Cinacalcet for Symptomatic Hypercalcemia Caused by AP2S1 Mutations.

M. Nesbit, R. Thakker, F. Hannan, 2016, The New England journal of medicine.

A monoclonal antibody that recognizes an antigenic determinant shared by HLA A2 and B17.

P. Parham, A. McMichael, F. Brodsky, 1980, Human immunology.

Immunotherapy of Melanoma Tumor-Specific CTL Suitable for Adoptive Cutting Edge: Rapid Cloning of

G. Ogg, P. Romero, Patrick, 1999 .

Structural and functional variability among DQβ alleles of DR2 subtypes

H. Mcdevitt, J. Bell, N. Rust, 2004, Immunogenetics.

Functional and biochemical subtypes of the haplotype HLA-DR3 in patients with celiac disease or idiopathic membranous nephropathy.

D. Jewell, A. McMichael, K. Wood, 1987, Human immunology.

Independent contributions of HLA epitopes and killer inhibitory receptor expression to the functional alloreactive specificity of natural killer cells.

K. Welsh, M. Dallman, V. Cerundolo, 1998, Human immunology.

Monoclonal Antibody to Human Platelet Glycoprotein I I. IMMUNOLOGICAL STUDIES

A. McMichael, D. Mason, G. Tobelem, 1981, British journal of haematology.

Immunoregulation by CD4 T cells in the induction of specific immunological unresponsiveness to alloantigens in vivo: evidence for a reduction in the frequency of alloantigen-specific cytotoxic T cells in vitro.

D. Roelen, K. Wood, P. Morris, 1998, Human immunology.

Steroid-protein interactions. 23. Nonidentity of cortisol-binding globulin and progesterone-binding globulin in guinea pig serum.

U. Westphal, R. Burton, G. Harding, 1971, Steroids.

Simplification of two-dimensional gel patterns of HLA class II antigens.

A. McMichael, N. Rust, 2008, Tissue antigens.

HLA-DR2 subtypes form an additional supertypic family of DR beta alleles.

H. Mcdevitt, A. McMichael, N. Rust, 1987, Proceedings of the National Academy of Sciences of the United States of America.

Mutations affecting G-protein subunit α11 in hypercalcemia and hypocalcemia.

M. Nesbit, R. Thakker, Rajesh V Thakker, 2013, The New England journal of medicine.

High-affinity binding of progesterone, testosterone and cortisol in normal and androgen-treated guinea pigs during various reproductive stages: relationship to masculinization.

U. Westphal, N. Rust, M. Diamond, 1969, Endocrinology.

Identification of 70 calcium-sensing receptor mutations in hyper- and hypo-calcaemic patients: evidence for clustering of extracellular domain mutations at calcium-binding sites.

J. Connell, B. Harding, Jenny J. Yang, 2012, Human molecular genetics.

Analysis of C. elegans intestinal gene expression and polyadenylation by fluorescence-activated nuclei sorting and 3′-end-seq

M. Hengartner, A. Furger, B. Tian, 2012, Nucleic acids research.

Identification of a G‐Protein Subunit‐α11 Gain‐of‐Function Mutation, Val340Met, in a Family With Autosomal Dominant Hypocalcemia Type 2 (ADH2)

A. Pagnamenta, B. Glaser, Jenny C. Taylor, 2016, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Allosteric Modulation of the Calcium-sensing Receptor Rectifies Signaling Abnormalities Associated with G-protein α-11 Mutations Causing Hypercalcemic and Hypocalcemic Disorders*

M. Nesbit, R. Thakker, A. Hanyaloglu, 2016, The Journal of Biological Chemistry.

DR2 (DRw15, 16): 2-D Gel Patterns

S. Fuggle, D. Charron, S. Sacks, 1989 .

DQw1 (DQw5, DQw6): 2-D Gel Patterns

R. Winchester, S. Fuggle, R. Bontrop, 1989 .

DQw3 (DQw7, DQw8, DQw9): 2-D Gel Patterns

D. Monos, R. Winchester, R. Fauchet, 1989 .

Direct isolation, phenotyping and cloning of low-frequency antigen-specific cytotoxic T lymphocytes from peripheral blood

G. Ogg, V. Cerundolo, P. Bruggen, 1998, Current Biology.

Structural and functional variability among DQ beta alleles of DR2 subtypes.

H. Mcdevitt, J. Bell, N. Rust, 1987, Immunogenetics.