Yotam Kaufman

发表

Reduced Folate Carrier Gene Silencing in Multiple Antifolate-resistant Tumor Cell Lines Is Due to a Simultaneous Loss of Function of Multiple Transcription Factors but Not Promoter Methylation*

Y. Assaraf, Lior Mayo, M. Stark, 2004, Journal of Biological Chemistry.

Resistance to multiple novel antifolates is mediated via defective drug transport resulting from clustered mutations in the reduced folate carrier gene in human leukaemia cell lines.

Y. Assaraf, D. Priest, G. Jansen, 2002, The Biochemical journal.

Coexistence of multiple mechanisms of PT523 resistance in human leukemia cells harboring 3 reduced folate carrier alleles: transcriptional silencing, inactivating mutations, and allele loss.

Y. Assaraf, G. Jansen, I. Ifergan, 2006, Blood.

The evolution of an RNA-based memory of self in the face of genomic conflict

Dominik Handler, J. Brennecke, A. Burga, 2022, bioRxiv.

Reduced folate carrier mutations are not the mechanism underlying methotrexate resistance in childhood acute lymphoblastic leukemia

Y. Assaraf, G. Rechavi, R. Elhasid, 2004, Cancer.

XX ovarian dysgenesis is caused by a PSMC3IP/HOP2 mutation that abolishes coactivation of estrogen-driven transcription.

Ayal B. Gussow, L. Carmel, E. Levy-Lahad, 2011, American journal of human genetics.

Testicular differentiation factor SF-1 is required for human spleen development.

C. Flück, N. Camats, E. Levy-Lahad, 2014, The Journal of clinical investigation.

MeCP2 deficiency in Rett syndrome causes epigenetic aberrations at the PWS/AS imprinting center that affects UBE3A expression.

A. Razin, R. Shemer, Kirill Makedonski, 2005, Human molecular genetics.

MeCP 2 deficiency in Rett syndrome causes epigenetic aberrations at the PWS / AS imprinting center that affects UBE 3 A expression

A. Razin, R. Shemer, Kirill Makedonski, 2005 .

Establishing the epigenetic status of the Prader-Willi/Angelman imprinting center in the gametes and embryo.

A. Razin, R. Shemer, Kirill Makedonski, 2004, Human molecular genetics.

MeCP2 Deficiency in the Brain Decreases BDNF Levels by REST/CoREST-Mediated Repression and Increases TRKB Production

A. Razin, R. Shemer, Kirill Makedonski, 2007, Epigenetics.

An SR protein is essential for activating DNA repair in malaria parasites

B. Singh, M. Goyal, R. Dzikowski, 2021, Journal of cell science.

Mechanisms of activation of the paternally expressed genes by the Prader-Willi imprinting center in the Prader-Willi/Angelman syndromes domains

A. Razin, R. Shemer, Aharon Razin, 2012, Proceedings of the National Academy of Sciences.

Protein-binding elements establish in the oocyte the primary imprint of the Prader-Willi/Angelman syndromes domain

A. Razin, R. Shemer, Maya Heled, 2009, Proceedings of the National Academy of Sciences.

but Not Promoter Methylation Function of Multiple Transcription Factors Lines Is Due to a Simultaneous Loss of Multiple Antifolate-resistant Tumor Cell Reduced Folate Carrier Gene Silencing in Recombination:

Y. Assaraf, M. Stark, L. Rothem, 2014 .

silencing, inactivating mutations, and allele loss leukemia cells harboring 3 reduced folate carrier alleles: transcriptional Coexistence of multiple mechanisms of PT523 resistance in human

Y. Assaraf, G. Jansen, I. Ifergan, 2013 .

An SR protein is essential for the recovery of malaria parasites from DNA damage and exposure to artemisinin

D. Ron, B. Singh, M. Goyal, 2021, bioRxiv.