J. Berthiaume

发表

Influence of Ischemia-Reperfusion Injury on Cardiac Metabolism

M. Willis, David I. Brown, J. Berthiaume, 2016 .

Mitochondrial NAD+/NADH Redox State and Diabetic Cardiomyopathy

D. Muntean, Mariana G Rosca, J. Berthiaume, 2017, Antioxidants & redox signaling.

Myocardial insulin resistance induced by high fat feeding in heart failure is associated with preserved contractile function.

M. Chandler, R. F. Muzic, C. Croniger, 2010, American journal of physiology. Heart and circulatory physiology.

Tumor necrosis factor receptor-associated factor 6 as a nuclear factor kappa B-modulating therapeutic target in cardiovascular diseases: at the heart of it all.

M. Willis, Muhammad Abdullah, J. Berthiaume, 2017, Translational research : the journal of laboratory and clinical medicine.

Diet-Induced Ketosis Protects Against Focal Cerebral Ischemia in Mouse.

J. LaManna, Kui Xu, M. Puchowicz, 2017, Advances in experimental medicine and biology.

Dietary Fructose Increases the Sensitivity of Proximal Tubules to Angiotensin II in Rats Fed High-Salt Diets

F. Dominici, J. Garvin, Agustin Gonzalez-Vicente, 2018, Nutrients.

Dietary Fructose Enhances the Ability of Low Concentrations of Angiotensin II to Stimulate Proximal Tubule Na+ Reabsorption

F. Dominici, J. Garvin, Agustin Gonzalez-Vicente, 2017, Nutrients.

Catabolism of (2E)-4-Hydroxy-2-nonenal via ω- and ω-1-Oxidation Stimulated by Ketogenic Diet*

Guo-Fang Zhang, M. Puchowicz, Zhong-Jing Huang, 2014, The Journal of Biological Chemistry.

MuRF1 mono-ubiquitinates TRα to inhibit T3-induced cardiac hypertrophy in vivo.

A. Gerdes, Wei-Yu Tang, M. Willis, 2016, Journal of molecular endocrinology.

Dietary vitamin E decreases doxorubicin-induced oxidative stress without preventing mitochondrial dysfunction

P. Oliveira, K. Wallace, J. Berthiaume, 2007, Cardiovascular Toxicology.

The Modulation of Oxygen Radical Production by Nitric Oxide in Mitochondria*

C. Giulivi, S. Elfering, J. Berthiaume, 2001, The Journal of Biological Chemistry.

Methylene blue decreases mitochondrial lysine acetylation in the diabetic heart

M. Miyagi, M. Chandler, Mikayla Depuydt, 2017, Molecular and Cellular Biochemistry.

Identification of Metabolic Changes in Ileum, Jejunum, Skeletal Muscle, Liver, and Lung in a Continuous I.V. Pseudomonas aeruginosa Model of Sepsis Using Nontargeted Metabolomics Analysis.

N. Deutz, J. Bain, M. Muehlbauer, 2019, The American journal of pathology.

MuRF1 mono-ubiquitinates TR[alpha] to inhibit T3 induced cardiac hypertrophy in vivo

A. Gerdes, Wei-Yu Tang, M. Willis, 2017 .

4-Hydroxy-2(E)-nonenal (HNE) catabolism and formation of HNE adducts are modulated by β oxidation of fatty acids in the isolated rat heart.

Guofang Zhang, Gregory P. Tochtrop, L. Nagy, 2013, Free radical biology & medicine.

Novel approach in LC-MS/MS using MRM to generate a full profile of acyl-CoAs: discovery of acyl-dephospho-CoAs[S]

B. Simons, Guo-Fang Zhang, S. Zhang, 2014, Journal of Lipid Research.

Mitochondrial complex I defect and increased fatty acid oxidation enhance protein lysine acetylation in the diabetic heart.

M. Miyagi, M. Chandler, M. Montano, 2015, Cardiovascular research.

Role of Receptor Protein Tyrosine Phosphatase γ in Sensing Extracellular CO2 and HCO3.

W. Boron, M. Chandler, Yuehan Zhou, 2016, Journal of the American Society of Nephrology : JASN.

Role of Receptor Protein Tyrosine Phosphatase g in Sensing Extracellular CO 2 and HCO 3 2

W. Boron, M. Chandler, Yuehan Zhou, 2016 .

Adriamycin-induced oxidative mitochondrial cardiotoxicity

K. Wallace, J. Berthiaume, 2006, Cell Biology and Toxicology.

Pathophysiology of Heart Failure and an Overview of Therapies

V. Rao, B. Hoit, B. Jensen, 2016 .

Hyperglycemia decreases mitochondrial function: the regulatory role of mitochondrial biogenesis.

C. Palmeira, K. Wallace, A. Rolo, 2007, Toxicology and applied pharmacology.

Ischemia promotes acyl-CoAs dephosphorylation and propionyl-CoA accumulation

Guo-Fang Zhang, S. Previs, T. Kasumov, 2023, Metabolomics.

Persistent Alterations to the Gene Expression Profile of the Heart Subsequent to Chronic Doxorubicin Treatment

K. Wallace, J. Berthiaume, 2007, Cardiovascular Toxicology.

Perfluorooctanoate, perflourooctanesulfonate, and N-ethyl perfluorooctanesulfonamido ethanol; peroxisome proliferation and mitochondrial biogenesis.

K. Wallace, Kendall B Wallace, Jessica Berthiaume, 2002, Toxicology letters.

Cardioprotective effects of dietary lipids evident in the time‐dependent alterations of cardiac function and gene expression following myocardial infarction

M. Chandler, B. Hoit, J. Berthiaume, 2014, Physiological reports.

Changes in myofilament proteins, but not Ca²⁺ regulation, are associated with a high-fat diet-induced improvement in contractile function in heart failure.

X Chen, L Castel, M. Chandler, 2011, American journal of physiology. Heart and circulatory physiology.

The myocardial contractile response to physiological stress improves with high saturated fat feeding in heart failure.

M. Chandler, B. Hoit, M. Bray, 2010, American journal of physiology. Heart and circulatory physiology.

Lymphocyte mitochondrial biomarkers in asymptomatic HIV-1-infected individuals treated with nucleoside reverse transcriptase inhibitors.

H. Balfour, A. Erice, K. Henry, 2002, AIDS.

Normalizing the metabolic phenotype after myocardial infarction: impact of subchronic high fat feeding.

Xin Yu, M. Chandler, M. Young, 2012, Journal of molecular and cellular cardiology.

Changes in myoﬁlament proteins, but not Ca 2 (cid:1) regulation, are associated with a high-fat diet-induced improvement in contractile function in heart failure

M. Chandler, L. Castel, X. Yu, 2011 .