E. Tiravanti

In vivo hyperoxic preconditioning protects against rat-heart ischemia/reperfusion injury by inhibiting mitochondrial permeability transition pore opening and cytochrome c release.

G. Paradies, V. Paradies, F. M. Ruggiero, 2011, Free radical biology & medicine.

HYPEROXIA CONFERS MYOCARDIAL PROTECTION IN MECHANICALLY VENTILATED RATS THROUGH THE GENERATION OF FREE RADICALS AND OPENING OF MITOCHONDRIAL ATP‐SENSITIVE POTASSIUM CHANNELS

T. Fiore, R. Rastaldo, G. Colantuono, 2008, Clinical and experimental pharmacology & physiology.

Melatonin protects against heart ischemia-reperfusion injury by inhibiting mitochondrial permeability transition pore opening.

T. Fiore, G. Paradies, F. M. Ruggiero, 2009, American journal of physiology. Heart and circulatory physiology.

Mitochondrial dysfunction associated with cardiac ischemia/reperfusion can be attenuated by oxygen tension control. Role of oxygen-free radicals and cardiolipin.

T. Fiore, G. Paradies, F. M. Ruggiero, 2005, Biochimica et biophysica acta.

Protective effect of melatonin against mitochondrial dysfunction associated with cardiac ischemiareperfusion: role of cardiolipin

G. Paradies, F. M. Ruggiero, G. Petrosillo, 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

EGCG, a green tea polyphenol, improves endothelial function and insulin sensitivity, reduces blood pressure, and protects against myocardial I/R injury in SHR.

M. Quon, G. Colantuono, A. Federici, 2007, American journal of physiology. Endocrinology and metabolism.

Treatment of spontaneously hypertensive rats with rosiglitazone ameliorates cardiovascular pathophysiology via antioxidant mechanisms in the vasculature.

M. Quon, G. Colantuono, A. Federici, 2009, American journal of physiology. Endocrinology and metabolism.

Nitrosyl-Heme Complexes Are Formed in the Ischemic Heart

J. Zweier, A. Samouilov, E. Tiravanti, 2004, Journal of Biological Chemistry.

Treatment of spontaneously hypertensive rats with rosiglitazone ameliorates cardiovascular pathophysiology via antioxidant mechanisms in the vasculature.

M. Quon, G. Colantuono, A. Federici, 2009, American journal of physiology. Endocrinology and metabolism.