A. Stein

发表

Delayed Adaptation of the Heart to Stress: Late Preconditioning

R. Bolli, B. Dawn, Xian-Liang Tang, 2004, Stroke.

Gene therapy with iNOS provides long-term protection against myocardial infarction without adverse functional consequences.

R. Bolli, S. Tiwari, B. Dawn, 2006, American journal of physiology. Heart and circulatory physiology.

GENE THERAPY WITH INDUCIBLE NITRIC OXIDE SYNTHASE PROVIDES LONG-TERM PROTECTION AGAINST MYOCARDIAL INFARCTION WITHOUT ADVERSE FUNCTIONAL CONSEQUENCES

R. Bolli, S. Tiwari, B. Dawn, 2005 .

Design and Evaluation of Tumor-Specific Dendrimer Epigenetic Therapeutics

Rahul Rattan, Sascha N. Goonewardena, Hong Zong, 2015, ChemistryOpen.

Late preconditioning induced by NO donors, adenosine A1 receptor agonists, and delta1-opioid receptor agonists is mediated by iNOS.

R. Bolli, W. Tan, Yiru Guo, 2005, American journal of physiology. Heart and circulatory physiology.

The late phase of ischemic preconditioning induces a prosurvival genetic program that results in marked attenuation of apoptosis.

R. Bolli, W. Tan, Yanqing Zhu, 2007, Journal of molecular and cellular cardiology.

IL-6 plays an obligatory role in late preconditioning via JAK-STAT signaling and upregulation of iNOS and COX-2.

R. Bolli, B. Dawn, W. Tan, 2004, Cardiovascular research.

Administration of a CO-releasing molecule at the time of reperfusion reduces infarct size in vivo.

R. Bolli, B. Dawn, R. Motterlini, 2004, American journal of physiology. Heart and circulatory physiology.

Hypercholesterolemia blunts NO donor-induced late preconditioning against myocardial infarction in conscious rabbits

R. Bolli, Xian-Liang Tang, G. Shirk, 2004, Basic research in cardiology.

Tumor necrosis factor-alpha does not modulate ischemia/reperfusion injury in naïve myocardium but is essential for the development of late preconditioning.

R. Bolli, B. Dawn, W. Tan, 2004, Journal of molecular and cellular cardiology.

Administration of a CO-releasing molecule induces late preconditioning against myocardial infarction.

R. Bolli, B. Dawn, R. Motterlini, 2005, Journal of molecular and cellular cardiology.

Effects of anesthesia on echocardiographic assessment of left ventricular structure and function in rats

R. Bolli, S. Tiwari, B. Dawn, 2006, Basic Research in Cardiology.

A murine model of inducible, cardiac-specific deletion of STAT3: its use to determine the role of STAT3 in the upregulation of cardioprotective proteins by ischemic preconditioning.

R. Bolli, J. Molkentin, B. Dawn, 2011, Journal of molecular and cellular cardiology.

Loss of H3K4 methylation destabilizes gene expression patterns and physiological functions in adult murine cardiomyocytes.

J. Jalife, G. Dressler, T. Herron, 2011, The Journal of clinical investigation.

The chromatin remodeling protein CHD7, mutated in CHARGE syndrome, is necessary for proper craniofacial and tracheal development

D. M. Martin, Elizabeth A. Hurd, Ethan D. Sperry, 2014, Developmental dynamics : an official publication of the American Association of Anatomists.

24 – Epigenetics in Cardiac Rhythm Diseases

A. Stein, 2013 .

Carbon monoxide induces a late preconditioning-mimetic cardioprotective and antiapoptotic milieu in the myocardium.

R. Bolli, B. Dawn, R. Motterlini, 2012, Journal of molecular and cellular cardiology.

The PTIP-Associated Histone Methyltransferase Complex Prevents Stress-Induced Maladaptive Cardiac Remodeling

J. Sadoshima, Sascha N. Goonewardena, A. Stein, 2015, PloS one.

Sirtuin 5 levels are limiting in preserving cardiac function and suppressing fibrosis in response to pressure overload

D. Sinclair, Shaday Michán, C. Lyssiotis, 2022, Scientific Reports.

Sirtuin 5 levels are limiting in preserving cardiac function and suppressing fibrosis in response to pressure overload

D. Sinclair, Shaday Michán, C. Lyssiotis, 2021, bioRxiv.

Cardiac stem cells delivered intravascularly traverse the vessel barrier, regenerate infarcted myocardium, and improve cardiac function.

D. Torella, R. Bolli, M. Rota, 2005, Proceedings of the National Academy of Sciences of the United States of America.

The beneficial effects of postinfarct cytokine combination therapy are sustained during long-term follow-up.

Robert J. Vincent, R. Bolli, S. Tiwari, 2009, Journal of molecular and cellular cardiology.

Monocyte Subsets and Inflammatory Cytokines in Acute Decompensated Heart Failure.

Rahul Rattan, Sascha N. Goonewardena, S. Hummel, 2016, Journal of cardiac failure.

Postinfarct Cytokine Therapy Regenerates Cardiac Tissue and Improves Left Ventricular Function

R. Bolli, P. Anversa, J. Kajstura, 2006, Circulation research.

Original Contribution

Xian-Liang Tang, G. Shirk, A. Stein, 1988 .

THE RELEVANCE OF HLA ANTIBODY AND A DONOR SPECIFIC FLOW CYTOMETRY CROSSMATCH TO REJECTION AND GRAFT SURVIVAL IN CARDIAC ALLOGRAFT RECIPIENTS

R. Bolli, S. Prabhu, B. Dawn, 2004 .

Interstitial remodeling in β1-adrenergic receptor transgenic mice

Xian-Liang Tang, G. Shirk, A. Stein, 2006, Basic Research in Cardiology.

VE-cadherin Role of SCH 79797 in Maintaining Vascular Integrity in Rat Model of Subarachnoid Hemorrhage

Xian-Liang Tang, G. Shirk, A. Stein, 2013 .

Original Contribution

Xian-Liang Tang, F. Manz, H. Kalhoff, 1988 .