A. Denenberg

发表

Protection of the hypertrophied myocardium by crystalloid cardioplegia.

W. Daggett, N. Munfakh, D. O'Keefe, 1991, The Journal of surgical research.

Bacterial induction of inducible nitric oxide synthase in cultured human intestinal epithelial cells.

C. Szabó, A. Salzman, A. Salzman, 1998, Gastroenterology.

Heat Shock Inhibits TNF-Induced ICAM-1 Expression in Human Endothelial Cells Via I Kappa Kinase Inhibition

H. Wong, T. Shanley, Bin Zhao, 2002, Shock.

Theaflavin, a black tea extract, is a novel anti-inflammatory compound

H. Wong, R. Aneja, K. Odoms, 2004, Critical care medicine.

Proinsulin c-peptide exerts beneficial effects in endotoxic shock in mice

G. Piraino, M. O’connor, P. Hake, 2007, Critical care medicine.

Epigallocatechin, a Green Tea Polyphenol, Attenuates Myocardial Ischemia Reperfusion Injury in Rats

Timothy J. Burroughs, H. Wong, P. Hake, 2004, Molecular medicine.

A Green Tea-Derived Polyphenol, Epigallocatechin-3-Gallate, Inhibits IκB Kinase Activation and IL-8 Gene Expression in Respiratory Epithelium

V. Malhotra, H. Wong, D. Wheeler, 2004, Inflammation.

Immunopathology and Infectious Diseases Peroxisome Proliferator-Activated Receptor Regulates Inflammation via NF-B Signaling in Polymicrobial Sepsis

G. Piraino, M. O’connor, P. Hake, 2010 .

Regulation of chemokine expression by IL-10 in lung inflammation.

T. Shanley, A. Denenberg, N. Vasi, 2000, Cytokine.

Phosphorylation of Extracellular Signal-Regulated Kinase (ERK)-1/2 Is Associated with the Downregulation of Peroxisome Proliferator-Activated Receptor (PPAR)-γ during Polymicrobial Sepsis

G. Piraino, P. Hake, B. Zingarelli, 2010, Molecular medicine.

Sesquiterpene Lactone Parthenolide, an Inhibitor of I&kgr;B Kinase Complex and Nuclear Factor-&kgr;B, Exerts Beneficial Effects in Myocardial Reperfusion Injury

H. Wong, P. Hake, B. Zingarelli, 2002, Shock.

Ciglitazone ameliorates lung inflammation by modulating the inhibitor &kgr;B protein kinase/nuclear factor-&kgr;B pathway after hemorrhagic shock

Ranjit S. Chima, G. Piraino, P. Hake, 2008, Critical care medicine.

ABLATION OF THE HEAT SHOCK FACTOR-1 INCREASES SUSCEPTIBILITY TO HYPEROXIA-MEDIATED CELLULAR INJURY

V. Malhotra, H. Wong, K. Dunsmore, 2002, Experimental lung research.

Increased expression of heat shock protein-70 protects A549 cells against hyperoxia.

H. Wong, J. Wispé, M. Ryan, 1998, American journal of physiology. Lung cellular and molecular physiology.

Differential regulation of activator protein-1 and heat shock factor-1 in myocardial ischemia and reperfusion injury: role of poly(ADP-ribose) polymerase-1.

B. Aronow, H. Wong, M. O’connor, 2004, American journal of physiology. Heart and circulatory physiology.

Induction and activity of nitric oxide synthase in cultured human intestinal epithelial monolayers.

M. O’connor, C. Szabó, A. Salzman, 1996, The American journal of physiology.

Recovery after cardioplegia in the hypertrophic rat heart.

W. Daggett, D. Torchiana, C. Bashour, 2000, The Journal of surgical research.

Liver X Receptor &agr; Activation with the Synthetic Ligand T0901317 Reduces Lung Injury and Inflammation After Hemorrhage and Resuscitation Via Inhibition of the Nuclear Factor &kgr;B Pathway

A. Lentsch, G. Piraino, M. O’connor, 2011, Shock.

PP2A REGULATES UPSTREAM MEMBERS OF THE C-JUN N-TERMINAL KINASE MITOGEN-ACTIVATED PROTEIN KINASE SIGNALING PATHWAY

H. Wong, Lei Sun, T. Shanley, 2007, Shock.

THE GREEN TEA POLYPHENOL EPIGALLOCATECHIN-3-GALLATE IMPROVES SYSTEMIC HEMODYNAMICS AND SURVIVAL IN RODENT MODELS OF POLYMICROBIAL SEPSIS

H. Wong, J. Catravas, P. Hake, 2007, Shock.

The Immunomodulatory Effects of Albumin In Vitro and In Vivo

H. Wong, B. Zingarelli, D. Wheeler, 2011, Advances in pharmacological sciences.

Temporal and Mechanistic Effects of Heat Shock on LPS-Mediated Degradation of IκBα in Macrophages

H. Wong, T. Shanley, K. Dunsmore, 2002, Inflammation.

Phosphatase inhibition leads to activation of IκB kinase in murine macrophages

H. Wong, T. Shanley, Bin Zhao, 2002 .

Role of nitric oxide in acute lung inflammation: Lessons learned from the inducible nitric oxide synthase knockout mouse*

P. Ward, T. Shanley, A. Salzman, 2002, Critical care medicine.

Hyperoxia prolongs tumor necrosis factor-alpha-mediated activation of NF-kappaB: role of IkappaB kinase.

H. Wong, T. Shanley, K. Odoms, 2002, Shock.

HYPEROXIA PROLONGS TUMOR NECROSIS FACTOR- -MEDIATED ACTIVATION OF NF- B: ROLE OF I B KINASE

H. Wong, T. Shanley, K. Odoms, 2002 .

Poly(ADP-Ribose) Polymerase-1 Regulates Activation of Activator Protein-1 in Murine Fibroblasts1

M. O’connor, P. Hake, B. Zingarelli, 2003, The Journal of Immunology.

Fibroblasts Activation of Activator Protein-1 in Murine Poly ( ADP-Ribose ) Polymerase-1 Regulates

M. O’connor, P. Hake, B. Zingarelli, 2003 .

Biological activity of truncated C-terminus human heat shock protein 72.

Sue E. Poynter, H. Wong, D. Wheeler, 2011, Immunology letters.

Poly(ADP-ribose) synthetase activation mediates increased permeability induced by peroxynitrite in Caco-2BBe cells.

C. Szabó, A. Salzman, Csaba Szabó, 1998, Gastroenterology.

Temporal and mechanistic effects of heat shock on LPS-mediated degradation of IkappaBalpha in macrophages.

H. Wong, T. Shanley, K. Dunsmore, 2002, Inflammation.

Hepatocyte NF-kappaB activation is hepatoprotective during ischemia-reperfusion injury and is augmented by ischemic hypothermia.

H. Wong, R. Schuster, A. Lentsch, 2007, American journal of physiology. Gastrointestinal and liver physiology.

HEAT SHOCK PROTEIN INDUCTION PROTECTS HUMAN RESPIRATORY EPITHELIUM AGAINST NITRIC OXIDE‐MEDIATED CYTOTOXICITY

J. Wispé, M. Ryan, A. Denenberg, 1997, Shock.

Liver apoptosis is age dependent and is reduced by activation of peroxisome proliferator-activated receptor-gamma in hemorrhagic shock.

G. Piraino, M. O’connor, P. Hake, 2010, American journal of physiology. Gastrointestinal and liver physiology.

Oxygenated cardioplegia: the metabolic and functional effects of glucose and insulin.

D. Hoaglin, W. Daggett, N. Munfakh, 1991, The Annals of thoracic surgery.

Maximal oxygenation of dilute blood Cardioplegic solution

W. Daggett, D. O'Keefe, G. Geffin, 1987 .

Absence of inducible nitric oxide synthase modulates early reperfusion‐induced NF‐κB and AP‐1 activation and enhances myocardial damage

H. Wong, M. O’connor, P. Hake, 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Changes in peroxisome proliferator-activated receptor-gamma activity in children with septic shock

H. Wong, B. Zingarelli, J. Kaplan, 2009, Intensive Care Medicine.

Endotoxin-induced ileal mucosal hyperpermeability in pigs: role of tissue acidosis.

C. Compton, A. Salzman, A. Denenberg, 1994, The American journal of physiology.

CONTRIBUTION OF MKP-1 REGULATION OF p38 TO ENDOTOXIN TOLERANCE

H. Wong, J. Molkentin, O. Bueno, 2005, Shock.

Maximal oxygenation of dilute blood cardioplegic solution.

W. Daggett, D. O'Keefe, G. Geffin, 1994, The Annals of thoracic surgery.

Immunoregulatory effects of regulated, lung-targeted expression of IL-10 in vivo.

T. Shanley, Bin Zhao, S. Wert, 2005, American journal of physiology. Lung cellular and molecular physiology.

K(ATP)-channel inhibition improves hemodynamics and cellular energetics in hemorrhagic shock.

C. Szabó, A. Salzman, Csaba Szabó, 1997, The American journal of physiology.

Activator protein‐1 signalling pathway and apoptosis are modulated by poly(ADP‐ribose) polymerase‐1 in experimental colitis

Timothy J. Burroughs, G. Piraino, M. O’connor, 2004, Immunology.

A novel role for matrix metalloproteinase-8 in sepsis*

H. Wong, B. Zingarelli, K. Dunsmore, 2012, Critical care medicine.

Absence of Poly(ADP-ribose) Polymerase-1 Alters Nuclear Factor-κB Activation and Gene Expression of Apoptosis Regulators after Reperfusion Injury

B. Aronow, M. O’connor, P. Hake, 2003, Molecular medicine.

The A2A receptor mediates an endogenous regulatory pathway of cytokine expression in THP‐1 cells

H. Wong, I. Biaggioni, T. Shanley, 2002, Journal of leukocyte biology.

C-peptide, a novel inhibitor of lung inflammation following hemorrhagic shock.

G. Piraino, P. Hake, B. Zingarelli, 2011, American journal of physiology. Lung cellular and molecular physiology.

Maximal 0 genation of Dilute Bloo 7 Cardioplegic Solution

W. Daggett, D. O'Keefe, G. Geffin, 1987 .

INDUCTION OF ENDOTOXIN TOLERANCE ENHANCES BACTERIAL CLEARANCE AND SURVIVAL IN MURINE POLYMICROBIAL SEPSIS

Sue E. Poynter, H. Wong, B. Zingarelli, 2008, Shock.

Heat shock inhibition of lipopolysaccharide-mediated tumor necrosis factor expression is associated with nuclear induction of MKP-1 and inhibition of mitogen-activated protein kinase activation

H. Wong, T. Shanley, Bin Zhao, 2004, Critical care medicine.

Absence of endogenous interleukin 10 enhances early stress response during post-ischaemic injury in mice intestine

H. Wong, Z. Yang, P. Hake, 2001, Gut.

Mechanism and function of heat shock-dependent IκBα expression

H. Wong, K. Dunsmore, K. Page, 2006, Inflammation Research.

C-Peptide Ameliorates Kidney Injury Following Hemorrhagic Shock

Ranjit S. Chima, G. Piraino, M. O’connor, 2011, Shock.

Peroxisome Proliferator Activator Receptor-γ Ligands, 15-Deoxy-Δ12,14-Prostaglandin J2 and Ciglitazone, Reduce Systemic Inflammation in Polymicrobial Sepsis by Modulation of Signal Transduction Pathways 1

M. O’connor, P. Hake, B. Zingarelli, 2003, The Journal of Immunology.

Superior recovery of hypertrophied rat myocardium after cardioplegic arrest.

W. Daggett, D. Torchiana, G. Geffin, 1998, The Annals of thoracic surgery.

Induction of HSP70 is dispensable for anti-inflammatory action of heat shock or NSAIDs in mast cells.

H. Wong, F. Redegeld, N. Bloksma, 2006, Experimental hematology.

Modulation of Signal Transduction Pathways Inflammation in Polymicrobial Sepsis by and Ciglitazone, Reduce Systemic 2 -Prostaglandin J 12,14 D Ligands, 15-Deoxy-g Peroxisome Proliferator Activator Receptor-O'Connor,

P. Hake, B. Zingarelli, J. Cook, 2003 .

DIVERSE CARDIOPROTECTIVE SIGNALING MECHANISMS OF PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR-γ LIGANDS, 15-DEOXY-Δ12,14-PROSTAGLANDIN J2 AND CIGLITAZONE, IN REPERFUSION INJURY: ROLE OF NUCLEAR FACTOR-κB, HEAT SHOCK FACTOR 1, AND AKT

Timothy J. Burroughs, H. Wong, G. Piraino, 2007, Shock.

Peroxisome proliferator activator receptor-gamma ligands, 15-deoxy-Delta(12,14)-prostaglandin J2 and ciglitazone, reduce systemic inflammation in polymicrobial sepsis by modulation of signal transduction pathways.

M. O’connor, P. Hake, B. Zingarelli, 2003, Journal of immunology.