H. Bär

发表

The biology of desmin filaments: how do mutations affect their structure, assembly, and organisation?

U. Aebi, S. Strelkov, H. Herrmann, 2004, Journal of structural biology.

Upregulation of embryonic transcription factors in right ventricular hypertrophy

L. Jahn, J. Kreuzer, H. Bär, 2003, Basic Research in Cardiology.

Unexpected pulsatile intracardiac mass.

H. Katus, D. Mereles, H. Bär, 2009, International journal of cardiology.

Interference of amino-terminal desmin fragments with desmin filament formation.

H. Zentgraf, U. Aebi, H. Katus, 2009, Cell motility and the cytoskeleton.

Disease mutations in the “head” domain of the extra-sarcomeric protein desmin distinctly alter its assembly and network-forming properties

H. Katus, Sarika Sharma, H. Herrmann, 2009, Journal of Molecular Medicine.

Assembly defects of desmin disease mutants carrying deletions in the alpha-helical rod domain are rescued by wild type protein.

U. Aebi, H. Katus, H. Herrmann, 2007, Journal of structural biology.

Impact of disease mutations on the desmin filament assembly process.

S. Müller, U. Aebi, H. Katus, 2006, Journal of molecular biology.

Phosphorylation of cytokeratin 8 and 18 in human vascular smooth muscle cells of atherosclerotic lesions and umbilical cord vessels

W. Kübler, L. Jahn, J. Kreuzer, 2001, Basic Research in Cardiology.

Accessory mitral valve as a potential source of cardioembolism.

H. Katus, D. Mereles, H. Bär, 2009, International journal of cardiology.

Intermediate filaments: from cell architecture to nanomechanics

Ueli Aebi, Laurent Kreplak, Sergei V. Strelkov, 2007, Nature Reviews Molecular Cell Biology.

Severe muscle disease-causing desmin mutations interfere with in vitro filament assembly at distinct stages.

U. Aebi, A. Kostareva, H. Herrmann, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Desmin and vimentin intermediate filament networks: their viscoelastic properties investigated by mechanical rheometry.

Sarika Sharma, N. Willenbacher, H. Herrmann, 2009, Journal of molecular biology.

Clinical, genetic, and cardiac magnetic resonance imaging findings in primary desminopathies

B. Wintersperger, H. Goebel, M. Vorgerd, 2008, Neuromuscular Disorders.

Prevalence of asymptomatic mitral valve malformations

H. Katus, D. Mereles, S. Hardt, 2009, Clinical Research in Cardiology.

Pathogenic effects of a novel heterozygous R350P desmin mutation on the assembly of desmin intermediate filaments in vivo and in vitro.

M. Vorgerd, R. Kley, D. Fischer, 2005, Human molecular genetics.

Cardiac α-actin in smooth muscle cells: detection in umbilical cord vessels and in atherosclerotic lesions

W. Kübler, L. Jahn, J. Kreuzer, 2000, Basic Research in Cardiology.

Conspicuous involvement of desmin tail mutations in diverse cardiac and skeletal myopathies

J. Holton, H. Katus, B. Eymard, 2007, Human mutation.

Forced expression of desmin and desmin mutants in cultured cells: impact of myopathic missense mutations in the central coiled-coil domain on network formation.

T. Sejersen, H. Katus, A. Kostareva, 2006, Experimental cell research.

Mutations in desmin's carboxy-terminal "tail" domain severely modify filament and network mechanics.

Sarika Sharma, N. Willenbacher, H. Herrmann, 2010, Journal of molecular biology.

Severe myopathy mutations modify the nanomechanics of desmin intermediate filaments.

L. Kreplak, L Kreplak, H Bär, 2009, Journal of molecular biology.

Cell cycle regulation and apoptotic cell death

H. Bär, L. Jahn, 2000 .

Cardiomyocyte production in mass suspension culture: embryonic stem cells as a source for great amounts of functional cardiomyocytes.

R. Zweigerdt, J. Lehmann, H. Bär, 2008, Tissue engineering. Part A.

G.O.7 A homozygous desmin deletion causes an Emery-Dreifuss like recessive myopathy with desmin depletion

C. Maurage, A. Ferreiro, D. Fischer, 2009, Neuromuscular Disorders.

Exploring the mechanical behavior of single intermediate filaments.

U Aebi, U. Aebi, L. Kreplak, 2005, Journal of molecular biology.

Smoothelin is an indicator of reversible phenotype modulation of smooth muscle cells in balloon-injured rat carotid arteries

G. V. van Eys, L. Jahn, J. Kreuzer, 2002, Basic Research in Cardiology.

Incomplete nonsense-mediated decay of mutant lamin A/C mRNA provokes dilated cardiomyopathy and ventricular tachycardia

H. Zentgraf, H. Katus, D. Weichenhan, 2008, Journal of Molecular Medicine.

Mutations in desmin's carboxy-terminal "tail" domain severely modify filament and network mechanics.

Sarika Sharma, N. Willenbacher, H. Herrmann, 2010, Journal of molecular biology.

Clinical, genetic, and cardiac magnetic resonance imaging findings in primary desminopathies

B. Wintersperger, H. Goebel, M. Vorgerd, 2008, Neuromuscular Disorders.