S. Grubb

发表

Comparison of Cultured Human Cardiomyocyte Clusters Obtained from Embryos/Fetuses or Derived from Human Embryonic Stem Cells.

K. Calloe, K. Møllgård, S. Christensen, 2019, Stem cells and development.

Multiple arrhythmic syndromes in a newborn, owing to a novel mutation in SCN5A.

C. Antzelevitch, J. Cordeiro, K. Calloe, 2011, Canadian journal of physiology and pharmacology.

Ventricular repolarization time, location of pacing stimulus and current pulse amplitude conspire to determine arrhythmogenicity in mice

K. Calloe, S. Olesen, M. Thomsen, 2017, Acta physiologica.

Anoctamin 6 differs from VRAC and VSOAC but is involved in apoptosis and supports volume regulation in the presence of Ca2+

E. Hoffmann, I. H. Lambert, E. Larsen, 2014, Pflügers Archiv - European Journal of Physiology.

Precapillary sphincters maintain perfusion in the cerebral cortex

M. Lauritzen, Changsi Cai, R. Murmu, 2020, Nature Communications.

Precapillary sphincters control cerebral blood flow

M. Lauritzen, Changsi Cai, R. Murmu, 2019, bioRxiv.

Precapillary sphincters maintain perfusion in the cerebral cortex

M. Lauritzen, Changsi Cai, R. Murmu, 2020, Nature Communications.

Impaired dynamics of precapillary sphincters and pericytes at first-order capillaries predict reduced neurovascular function in the aging mouse brain

A. Devor, Changsi Cai, Kirsten Thomsen, 2023, Nature Aging.

Impaired dynamics of brain precapillary sphincters and pericytes at first order capillaries explains reduced neurovascular functions in aging

M. Lauritzen, Changsi Cai, K. Thomsen, 2021 .

Ultrastructure of precapillary sphincters and the neurovascular unit

S. Grubb, 2022, bioRxiv.

Brain capillary pericytes and neurovascular coupling.

M. Lauritzen, C. Aalkjær, S. Grubb, 2021, Comparative biochemistry and physiology. Part A, Molecular & integrative physiology.

Impact of KChIP2 on Cardiac Electrophysiology and the Progression of Heart Failure

K. Calloe, M. Thomsen, S. Grubb, 2012, Front. Physio..

Factors Associated With Large Improvements in Health-Related Quality of Life in Patients With Atrial Fibrillation

D. Singer, G. Fonarow, L. Allen, 2020, Circulation. Arrhythmia and electrophysiology.

TMEM16F (Anoctamin 6), an anion channel of delayed Ca2+ activation

E. Hoffmann, E. Larsen, T. K. Klausen, 2013, The Journal of general physiology.

TMEM 16 F ( Anoctamin 6 ) , an anion channel of delayed Ca 2 + activation

E. Hoffmann, E. Larsen, T. K. Klausen, 2022 .

Development of heart failure is independent of K+ channel‐interacting protein 2 expression

K. Calloe, S. Olesen, M. Thomsen, 2013, The Journal of physiology.

A novel KCND 3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation

J. Svendsen, M. Olesen, A. Holst, 2013 .

A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation.

J. Svendsen, M. Olesen, A. Holst, 2013, Cardiovascular research.

Confocal Imaging of Intracellular Calcium Cycling in Isolated Cardiac Myocytes

G. Aistrup, S. Grubb, J. Wasserstrom, 2013 .

Ultrastructure of the brain waste-clearance pathway

S. Grubb, 2023, bioRxiv.

Deep sleep drives brain fluid oscillations

M. Lauritzen, S. Grubb, 2019, Science.

Anoctamin 6 differs from VRAC and VSOAC but is involved in apoptosis and supports volume regulation in the presence of Ca2+

E. Hoffmann, I. H. Lambert, E. Larsen, 2014, Pflügers Archiv: European Journal of Physiology.

Triggered intracellular calcium waves in dog and human left atrial myocytes from normal and failing hearts

Francisco J. Alvarado, J. Cordeiro, Y. Shiferaw, 2017, Cardiovascular research.

Human Embryonic Stem Cell-Derived Cardiomyocytes Self-Arrange with Areas of Different Subtypes During Differentiation.

K. Calloe, K. Møllgård, S. Christensen, 2017, Stem cells and development.

Characterization and Mechanisms of Action of Novel NaV1.5 Channel Mutations Associated With Brugada Syndrome

R. Nussbaum, M. Scheinman, K. Calloe, 2013, Circulation. Arrhythmia and electrophysiology.

Preservation of cardiac function by prolonged action potentials in mice deficient of KChIP2.

Jussi T. Koivumäki, K. Calloe, S. Olesen, 2015, American journal of physiology. Heart and circulatory physiology.

Anoctamin 6 differs from VRAC and VSOAC but is involved in apoptosis and supports volume regulation in the presence of Ca2+

E. Hoffmann, I. H. Lambert, E. Larsen, 2014, Pflügers Archiv: European Journal of Physiology.

ION CHANNELS , RECEPTORS AND TRANSPORTERS Anoctamin 6 differs from VRAC and VSOAC but is involved in apoptosis and supports volume regulation in the presence of Ca 2 +

E. Hoffmann, I. H. Lambert, K. Poulsen, 2018 .

Anoctamin 6 differs from VRAC and VSOAC but is involved in apoptosis and supports volume regulation in the presence of Ca

E. Hoffmann, I. H. Lambert, K. Poulsen, 2014 .

Loss of K+ Currents in Heart Failure Is Accentuated in KChIP2 Deficient Mice

C. Fiset, K. Calloe, S. Olesen, 2014, Journal of cardiovascular electrophysiology.

Molecular Cloning and Functional Expression of the Equine K+ Channel KV11.1 (Ether à Go-Go-Related/KCNH2 Gene) and the Regulatory Subunit KCNE2 from Equine Myocardium

F. Hauser, K. Calloe, R. Buhl, 2015, PloS one.

A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation.

J. Svendsen, M. Olesen, A. Holst, 2013, Cardiovascular research.