Haijun Chen

发表

Kir2.1 and K2P1 channels reconstitute two levels of resting membrane potential in cardiomyocytes

Dongchuan Zuo, Kuihao Chen, Min Zhou, 2017, The Journal of physiology.

Kir2.1 channels set two levels of resting membrane potential with inward rectification

Dongchuan Zuo, Kuihao Chen, Zheng Liu, 2018, Pflügers Archiv - European Journal of Physiology.

Functional analysis of a novel potassium channel (KCNA1) mutation in hereditary myokymia

L. Kaczmarek, L. Ment, F. Hisama, 2006, Neurogenetics.

Kir2 inward rectification–controlled precise and dynamic balances between Kir2 and HCN currents initiate pacemaking activity

Sho-Ya Wang, Dongchuan Zuo, Kuihao Chen, 2018, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

A δ-Conotoxin from Conus ermineus Venom Inhibits Inactivation in Vertebrate Neuronal Na+ Channels but Not in Skeletal and Cardiac Muscles*

S. Heinemann, A. Ménez, J. Barbier, 2004, Journal of Biological Chemistry.

K2P1 leak cation channels contribute to ventricular ectopic beats and sudden death under hypokalemia

Kai Tang, Bo Han, Shangwei Hou, 2022, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Interaction of Scorpion α-Toxins with Cardiac Sodium Channels

S. Heinemann, Haijun Chen, 2001, The Journal of General Physiology.

Scorpion α and α‐like toxins differentially interact with sodium channels in mammalian CNS and periphery

S. Heinemann, D. Gordon, G. Nicholson, 2000 .

Functional Analysis of a Novel Potassium Channel (KCNA1) Mutation in Hereditary Mokymia

L. Kaczmarek, L. Ment, F. Hisama, 2007 .

Report Charybdotoxin Binding in the I Ks Pore Demonstrates Two MinK Subunits in Each Channel Complex

S. Goldstein, S. Rajan, Shuhua Xu, 2003 .

De novo gain-of-function KCNT1 channel mutations cause malignant migrating partial seizures of infancy

L. Kaczmarek, A. Munnich, J. Casanova, 2012, Nature Genetics.

Serial perturbation of MinK in IKs implies an alpha-helical transmembrane span traversing the channel corpus.

S. Goldstein, Haijun Chen, 2007, Biophysical journal.

Localization of Kv1.3 channels in presynaptic terminals of brainstem auditory neurons

L. Kaczmarek, C. Rahner, Valeswara-Rao Gazula, 2010, The Journal of comparative neurology.

TWIK-1 Two-Pore Domain Potassium Channels Change Ion Selectivity and Conduct Inward Leak Sodium Currents in Hypokalemia

Haijun Chen, Xuexin Zhang, Liqun Ma, 2011, Science Signaling.

TWIK-1 and TREK-1 Are Potassium Channels Contributing Significantly to Astrocyte Passive Conductance in Rat Hippocampal Slices

H. Kimelberg, M. Xie, G. Schools, 2009, The Journal of Neuroscience.

Cyclosporin A selectively reduces the functional expression of Kir2.1 potassium channels in Xenopus oocytes

S. Heinemann, Y. Kubo, T. Hoshi, 1998, FEBS letters.

Genetic Deletion of TREK-1 or TWIK-1/TREK-1 Potassium Channels does not Alter the Basic Electrophysiological Properties of Mature Hippocampal Astrocytes In Situ

Qi Wang, R. Bryan, Qi Wan, 2016, Front. Cell. Neurosci..

mGluR3 Activation Recruits Cytoplasmic TWIK-1 Channels to Membrane that Enhances Ammonium Uptake in Hippocampal Astrocytes

Min Zhou, Haijun Chen, Wei Wang, 2016, Molecular Neurobiology.

The contribution of TWIK-1 channels to astrocyte K+ current is limited by retention in intracellular compartments

L. Kaczmarek, J. Barhanin, F. Lesage, 2013, Front. Cell. Neurosci..

Pathogenic mechanism of a catecholaminergic polymorphic ventricular tachycardia causing-mutation in cardiac calcium release channel RyR2.

Wenjun Zheng, Z. Yuchi, Qian Zhao, 2018, Journal of molecular and cellular cardiology.

Altered and dynamic ion selectivity of K+ channels in cell development and excitability.

F. Lesage, F. Chatelain, Haijun Chen, 2014, Trends in pharmacological sciences.

Acid-sensitive TWIK and TASK Two-pore Domain Potassium Channels Change Ion Selectivity and Become Permeable to Sodium in Extracellular Acidification*

Min Zhou, Haijun Chen, Xuexin Zhang, 2012, The Journal of Biological Chemistry.

Charybdotoxin Binding in the IKs Pore Demonstrates Two MinK Subunits in Each Channel Complex

S. Goldstein, Shuhua Xu, Haijun Chen, 2003, Neuron.

Pore- and state-dependent cadmium block of I(Ks) channels formed with MinK-55C and wild-type KCNQ1 subunits.

F. Sesti, S. Goldstein, Haijun Chen, 2003, Biophysical journal.

Classification of 2-pore domain potassium channels based on rectification under quasi-physiological ionic conditions

Dongchuan Zuo, Min Zhou, Haijun Chen, 2014, Channels.

Silent TWIK-1 potassium channels conduct monovalent cation currents.

Min Zhou, Haijun Chen, Yu-Ping Xie, 2012, Biophysical journal.

The N-Terminal Domain of Slack Determines the Formation and Trafficking of Slick/Slack Heteromeric Sodium-Activated Potassium Channels

L. Kaczmarek, F. Sigworth, L. Salkoff, 2009, The Journal of Neuroscience.

Kir2.1 channels set two levels of resting membrane potential with inward rectification

Dongchuan Zuo, Kuihao Chen, Zheng Liu, 2017, Pflügers Archiv - European Journal of Physiology.