Michal A. Levine

发表

Expression of angiotensin-converting enzyme (CD143) identifies and regulates primitive hemangioblasts derived from human pluripotent stem cells.

Elias T. Zambidis, T. Park, E. Zambidis, 2008, Blood.

Challenges and strategies for generating therapeutic patient-specific hemangioblasts and hematopoietic stem cells from human pluripotent stem cells.

Elias T. Zambidis, P. Burridge, Ann Peters, 2010, The International journal of developmental biology.

Identification of a novel mutation of SH3BP2 in cherubism and demonstration that SH3BP2 mutations lead to increased NFAT activation

R. Kohanski, S. Lietman, Michal A. Levine, 2006, Human mutation.

Edinburgh Research Explorer Expression of angiotensin-converting enzyme (CD143) identifies and regulates primitive hemangioblasts derived from human pluripotent stem cells

T. Park, E. Zambidis, M. Pryzhkova, 2008 .

Challenges and strategies for generating therapeutic patient-specific hemangioblasts and hematopoietic stem cells from human pluripotent stem cells.

Elias T. Zambidis, P. Burridge, Ann Peters, 2010, The International journal of developmental biology.

Olfactory dysfunction in humans with deficient guanine nucleotide-binding protein

R. Weinstock, A. Spiegel, A. Moses, 1986, Nature.

Rat cerebral cortex corticotropin‐releasing hormone receptors: evidence for receptor coupling to multiple G‐proteins

H. Randeva, D. Grammatopoulos, E. Hillhouse, 2001, Journal of neurochemistry.

Coupling of the PTH/PTHrP receptor to multiple G-proteins

L. Suva, M. Pines, M. Levine, 1998, Endocrine.

Early identification of a 12-bp tandem duplication in TNFRSF11A encoding receptor activator of nuclear factor-kappa B (RANK): Clinical characterization and response to bisphosphonate therapy.

S. Mumm, M. Whyte, K. Shekdar, 2023, Bone.

Directional memory arises from long-lived cytoskeletal asymmetries in polarized chemotactic cells

L Mahadevan, Daniel Irimia, Yasmine Meroz, 2016, Proceedings of the National Academy of Sciences.

Identification of a mutation in the gene encoding the alpha subunit of the stimulatory G protein of adenylyl cyclase in McCune-Albright syndrome.

C. Francomano, M. Levine, C. Francomano, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Mutation in the gene encoding the stimulatory G protein of adenylate cyclase in Albright's hereditary osteodystrophy.

D. Valle, P. Gruppuso, G. Steele, 1990, The New England journal of medicine.

Heterogeneous mutations in the gene encoding the alpha-subunit of the stimulatory G protein of adenylyl cyclase in Albright hereditary osteodystrophy.

M. Levine, Michal A. Levine, J. D. Vechio, 1993, The Journal of clinical endocrinology and metabolism.

Infantile hypothyroidism in two sibs: an unusual presentation of pseudohypoparathyroidism type Ia.

T. Jap, M. Levine, Michal A. Levine, 1985, The Journal of pediatrics.

Genetic deficiency of the alpha subunit of the guanine nucleotide-binding protein Gs as the molecular basis for Albright hereditary osteodystrophy.

H. Bourne, M. Levine, P. Smallwood, 1988, Proceedings of the National Academy of Sciences of the United States of America.

Activity of the stimulatory guanine nucleotide-binding protein is reduced in erythrocytes from patients with pseudohypoparathyroidism and pseudopseudohypoparathyroidism: biochemical, endocrine, and genetic analysis of Albright's hereditary osteodystrophy in six kindreds.

T. Jap, A. Spiegel, M. Levine, 1986, The Journal of clinical endocrinology and metabolism.

Quantitative reverse transcription-PCR measurement of thyroglobulin mRNA in peripheral blood of healthy subjects.

M. Ringel, G. Francis, R. Tuttle, 1999, Clinical chemistry.

A novel spliced variant of the type 1 corticotropin-releasing hormone receptor with a deletion in the seventh transmembrane domain present in the human pregnant term myometrium and fetal membranes.

H. Randeva, E. Karteris, A. Easton, 1999, Molecular endocrinology.

Pancreatic Glucagon-Like Peptide-1 Receptor Couples to Multiple G Proteins and Activates Mitogen-Activated Protein Kinase Pathways in Chinese Hamster Ovary Cells *

M. Bernier, C. Montrose‐Rafizadeh, M. J. Garant, 1999 .

A cytoskeleton-based memory in chemotactic leukocytes.

Guillaume Charras, Yasmine Meroz, Jagesh V. Shah, 2014 .

Specificity of G protein beta and gamma subunit interactions.

E. Neer, C. J. Schmidt, Michal A. Levine, 1992, The Journal of biological chemistry.

A novel Gs alpha mutant in a patient with Albright hereditary osteodystrophy uncouples cell surface receptors from adenylyl cyclase.

M. Levine, A. Miric, D. Zimmerman, 1994, The Journal of biological chemistry.

Measurement of thyroglobulin mRNA in peripheral blood as an adjunctive test for monitoring thyroid cancer

M. Ringel, I. Brown, M. Levine, 2003, Molecular pathology : MP.

Mutation Screening of the TNFRSF11A Gene Encoding Receptor Activator of NFkB (RANK) in Familial and Sporadic Paget's Disease of Bone and Osteosarcoma

M. Adams, A. Sparks, J. Venter, 2001, Calcified Tissue International.

Pancreatic Glucagon-Like Peptide-1 Receptor Couples to Multiple G Proteins and Activates Mitogen-Activated Protein Kinase Pathways in Chinese Hamster Ovary Cells *

M. Bernier, C. Montrose‐Rafizadeh, M. J. Garant, 1999 .

Targeted disruption of Gnas in embryonic stem cells.

J. Gearhart, K. Reese, M. Levine, 1997, Endocrinology.

Immunochemical analysis of the alpha-subunit of the stimulatory G-protein of adenylyl cyclase in patients with Albright's hereditary osteodystrophy.

M. Levine, Michal A. Levine, J. Patten, 1990, The Journal of clinical endocrinology and metabolism.

Reproductive dysfunction in women with Albright's hereditary osteodystrophy.

G. Merriam, A. Moses, M. Levine, 1998, The Journal of clinical endocrinology and metabolism.

Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene.

M. Levine, H. Maheshwari, G. Baumann, 1999, The Journal of clinical endocrinology and metabolism.

Selective resistance to parathyroid hormone caused by a novel uncoupling mutation in the carboxyl terminus of G alpha(s). A cause of pseudohypoparathyroidism type Ib.

M. Levine, W. Schwindinger, Michal A. Levine, 2001, The Journal of biological chemistry.