Sami Gritli

Mycobacterium tuberculosis Virulent Factor ESAT-6 Drives Macrophage Differentiation Toward the Pro-inflammatory M1 Phenotype and Subsequently Switches It to the Anti-inflammatory M2 Phenotype

M. Essafi, A. Refai, M. Barbouche, 2018, Front. Cell. Infect. Microbiol..

Involvement of Different CD4+ T Cell Subsets Producing Granzyme B in the Immune Response to Leishmania major Antigens

A. Gharbi, A. Ben Salah, H. Louzir, 2014, Mediators of inflammation.

Prediction of T Cell Epitopes from Leishmania major Potentially Excreted/Secreted Proteins Inducing Granzyme B Production

A. Ben Salah, H. Louzir, M. Chenik, 2016, PloS one.

SPRED2 loss-of-function causes a recessive Noonan syndrome-like phenotype.

L. Vissers, H. Cavé, B. Gelb, 2021, American journal of human genetics.

Validation of Recombinant Salivary Protein PpSP32 as a Suitable Marker of Human Exposure to Phlebotomus papatasi, the Vector of Leishmania major in Tunisia

A. Ben Salah, J. Bettaieb, S. Kamhawi, 2015, PLoS neglected tropical diseases.