E. Rich

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Complement receptor-mediated uptake and tumor necrosis factor-alpha-mediated growth inhibition of Mycobacterium tuberculosis by human alveolar macrophages.

J. Ellner, D. Russell, C. Hirsch, 1994, Journal of immunology.

Examining a paradox in the pathogenesis of human pulmonary tuberculosis: immune activation and suppression/anergy.

G. Vanham, Z. Toossi, J. Ellner, 1997, Tubercle and lung disease : the official journal of the International Union against Tuberculosis and Lung Disease.

Dyscoordinate expression of tumor necrosis factor-alpha by human blood monocytes and alveolar macrophages.

J. Ellner, R. Wallis, J. Panuska, 1989, The American review of respiratory disease.

Cytokine Profiles for Peripheral Blood Lymphocytes from Patients with Active Pulmonary Tuberculosis and Healthy Household Contacts in Response to the 30-Kilodalton Antigen ofMycobacterium tuberculosis

M. Torres, E. Sada, E. Rich, 1998, Infection and Immunity.

Rich tuberculosis Mycobacterium 30-Kilodalton Antigen of Household Contacts in Response to the Pulmonary Tuberculosis and Healthy Lymphocytes from Patients with Active Cytokine Profiles for Peripheral Blood

M. Torres, E. Sada, E. Rich, 1997 .

Tumor Necrosis Factor/Cachectin and Interleukin-1 Secretion by Cord Blood Monocytes from Premature and Term Neonates

E. Rich, K. Weatherstone, 1989, Pediatric Research.

T lymphocytic and immature macrophage alveolitis in active pulmonary tuberculosis.

M. Torres, E. Sada, S. Schwander, 1996, The Journal of infectious diseases.

Pulmonary Mononuclear Cell Responses to Antigens of Mycobacterium tuberculosis in Healthy Household Contacts of Patients with Active Tuberculosis and Healthy Controls from the Community1 2

Z. Toossi, J. Ellner, M. Torres, 2000, The Journal of Immunology.

from the Community Active Tuberculosis and Healthy Controls Healthy Household Contacts of Patients with in Mycobacterium tuberculosis Antigens of Pulmonary Mononuclear Cell Responses to

Z. Toossi, J. Ellner, M. Torres, 2000 .

Pulmonary immune response to Mycobacterium tuberculosis and human immunodeficiency virus.

E. Rich, 1996, Infectious agents and disease.

Dysregulation of β-chemokines in the lungs of HIV-1-infected patients

Z. Toossi, Mianda Wu, H. Aung, 2001 .

Decreased production of TGF-beta 1 by human alveolar macrophages compared with blood monocytes.

B. Hamilton, Z. Toossi, C. Hirsch, 1996, Journal of immunology.

Activation-inactivation of HIV-1 in the lung.

E. Rich, 1998, Journal of biomedical science.

Spectrum of immunoregulatory functions and properties of human alveolar macrophages.

D. Tweardy, H. Fujiwara, J. Ellner, 1987, The American review of respiratory disease.

Activation of latent HIV-1 by Mycobacterium tuberculosis and its purified protein derivative in alveolar macrophages from HIV-infected individuals in vitro.

Z. Toossi, Liang Xia, L. Xia, 1997, Journal of acquired immune deficiency syndromes and human retrovirology : official publication of the International Retrovirology Association.

Accessory function and properties of monocytes from healthy elderly humans for T lymphocyte responses to mitogen and antigen.

D. Hom, J. Ellner, M. Mincek, 1993, Gerontology.

Characterization of suppressor function of human alveolar macrophages for T lymphocyte responses to phytohemagglutinin: cellular selectivity, reversibility, and early events in T cell activation.

W. Boom, E. Rich, C. Finegan, 1993, American journal of respiratory cell and molecular biology.

Requirement for cell-to-cell contact for the immunosuppressive activity of human alveolar macrophages.

Z. Toossi, J. Ellner, M. Leonard, 1991, American journal of respiratory cell and molecular biology.

Disparate cytochemical characteristics and production of cytokines and prostaglandin E2 by human mononuclear phagocytes from the blood, lung, and peritoneal cavity.

M. Friedlander, C. Hilbert, E. Rich, 1994, The Journal of laboratory and clinical medicine.

Expression of the human NRAMP1 gene in professional primary phagocytes: studies in blood cells and in HL‐60 promyelocytic leukemia

D. Malo, W. Dalton, P. Gros, 1997, Journal of leukocyte biology.

Enhanced production of human immunodeficiency virus type 1 by in vitro-infected alveolar macrophages from otherwise healthy cigarette smokers.

L. Guay, R. Abbud, E. Rich, 1995, The Journal of infectious diseases.

Interleukin-10 contributes development of macrophage suppressor activities by macrophage colony-stimulating factor, but not by granulocyte-macrophage colony-stimulating factor.

K. Akagawa, E. Rich, K. Mochida-Nishimura, 2001, Cellular immunology.

Preliminary characterization of accessory cells in the nonadherent fraction of human blood mononuclear cells.

J. Ellner, E. Rich, 1986, Cellular immunology.

Interleukin‐1 Production by Blood Monocytes and Bronchoalveolar Cells in Sarcoidosis

H. Fujiwara, H. Fujiwara, M. Kleinhenz, 1986, Annals of the New York Academy of Sciences.

Role of dialysis modality in responses of blood monocytes and peritoneal macrophages to endotoxin stimulation.

M. Friedlander, C. Hilbert, E. Rich, 1993, American journal of kidney diseases : the official journal of the National Kidney Foundation.

Expression of tumor necrosis factor in vitro by human mononuclear phagocytes stimulated with whole Mycobacterium bovis BCG and mycobacterial antigens

J. Ellner, R. Wallis, E. Rich, 1988, Infection and immunity.

Defective accessory function of monocytes in human immunodeficiency virus-related disease syndromes.

M. Lederman, H. Fujiwara, Z. Toossi, 1988, The Journal of laboratory and clinical medicine.

Enhanced responses to Mycobacterium tuberculosis antigens by human alveolar lymphocytes during active pulmonary tuberculosis.

M. Torres, R. Wallis, E. Sada, 1998, The Journal of infectious diseases.

Selective CXCR4 antagonism by Tat: implications for in vivo expansion of coreceptor use by HIV-1.

H. Xiao, K. Jeang, M. Benkirane, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Cell membrane is a major locus for ultraviolet B-induced alterations in accessory cells.

C. Elmets, J. Ellner, J. Krutmann, 1990, The Journal of clinical investigation.

Deleterious effect of ultraviolet-B radiation on accessory function of human blood adherent mononuclear cells.

C. Elmets, H. Fujiwara, J. Ellner, 1987, Clinical and experimental immunology.

Chemokines induced by infection of mononuclear phagocytes with mycobacteria and present in lung alveoli during active pulmonary tuberculosis.

Z. Toossi, E. Sada, S. Schwander, 1998, American journal of respiratory cell and molecular biology.

Increased susceptibility of differentiated mononuclear phagocytes to productive infection with human immunodeficiency virus-1 (HIV-1).

W. O'brien, J. Zack, I. Chen, 1992, The Journal of clinical investigation.

Accessory function of human mononuclear phagocytes for lymphocyte responses to the superantigen staphylococcal enterotoxin B.

J. Fayen, M. Leonard, E. Rich, 1992, Cellular immunology.

Multifaceted Activities of the HIV-1 Transactivator of Transcription, Tat*

K. Jeang, Hua Xiao, K T Jeang, 1999, The Journal of Biological Chemistry.

Human alveolar T lymphocyte responses to Mycobacterium tuberculosis antigens: role for CD4+ and CD8+ cytotoxic T cells and relative resistance of alveolar macrophages to lysis.

W. Boom, S. Schwander, D. Canaday, 1997, Journal of immunology.

Relationship between load of virus in alveolar macrophages from human immunodeficiency virus type 1-infected persons, production of cytokines, and clinical status.

D. Hom, Z. Toossi, J. Sierra-Madero, 1994, The Journal of infectious diseases.

Differential Activation of MAP Kinase Signaling Pathways and Nuclear Factor-κB in Bronchoalveolar Cells of Smokers and Nonsmokers

Z. Toossi, D. Templeton, J. Cross, 2001, Molecular medicine.

A Macrophage-Tropic HIV-1 That Expresses Green Fluorescent Protein and Infects Alveolar and Blood Monocyte-Derived Macrophages

K. Jeang, J. Orenstein, E. Rich, 2002, Journal of Biomedical Science.

Expression of transforming growth factor-β but not tumor necrosis factor-α, interferon-γ, and interleukin-4 in granulomatous lung lesions in tuberculosis.

Z. Toossi, H. Aung, E. Sada, 2000 .

Activation-Inactivation of HIV-1 in the Lung

E. Rich, 1998, Journal of Biomedical Science.

Mediation of entry of human immunodeficiency virus-1 into alveolar macrophages by CD4 without facilitation by surfactant-associated protein-A.

L. Guay, J. Sierra-Madero, E. Rich, 1997, American journal of respiratory cell and molecular biology.

Enhanced susceptibility of blood monocytes from patients with pulmonary tuberculosis to productive infection with human immunodeficiency virus type 1

Z. Toossi, R. Blinkhorn, J. Sierra-Madero, 1993, The Journal of experimental medicine.

Quinone Reductase Inhibitors Block SAPK/JNK and NFκB Pathways and Potentiate Apoptosis*

D. Templeton, L. Parrott, J. Cross, 1999, The Journal of Biological Chemistry.

Alveolar macrophages as accessory cells for human gamma delta T cells activated by Mycobacterium tuberculosis.

W. Boom, S. Schwander, K. N. Balaji, 1995, Journal of immunology.

Identification of a type V collagen-degrading enzyme from human sputum.

J. Seyer, A. Kang, C. Mainardi, 2015, The American review of respiratory disease.

Mycobacterium tuberculosis (MTB)-stimulated production of nitric oxide by human alveolar macrophages and relationship of nitric oxide production to growth inhibition of MTB.

B. Hamilton, Z. Toossi, M. Torres, 1997, Tubercle and lung disease : the official journal of the International Union against Tuberculosis and Lung Disease.

A macrophage-tropic HIV-1 that expresses green fluorescent protein and infects alveolar and blood monocyte-derived macrophages.

K. Jeang, J. Orenstein, E. Rich, 2002, Journal of biomedical science.

Dysregulation of beta-chemokines in the lungs of HIV-1-infected patients.

Z. Toossi, H. Aung, R. Hejal, 2001, Journal of acquired immune deficiency syndromes.

Transient normalization of lymphocyte blastogenic and specific antibody responses following boosting of healthy elderly subjects with tetanus toxoid.

D. Hom, K. Edmonds, M. Mincek, 1993, Journal of gerontology.

Pulmonary surfactant as a physiologic immunosuppressive agent.

E. Rich, 1990, The Journal of laboratory and clinical medicine.