Takaki Watanabe

发表

Impairment of methylglyoxal detoxification systems causes mitochondrial dysfunction and schizophrenia-like behavioral deficits

M. Kano, M. Itokawa, G. Konopka, 2020, bioRxiv.

AUTS2 Governs Cerebellar Development, Purkinje Cell Maturation, Motor Function and Social Communication

M. Kano, K. Sakimura, Naofumi Uesaka, 2020, bioRxiv.

mGluR5 Is Substitutable for mGluR1 in Cerebellar Purkinje Cells for Motor Coordination, Developmental Synapse Elimination, and Motor Learning

M. Kano, A. Aiba, K. Nakao, 2022, Cells.

Zebrafish relatively relaxed mutants have a ryanodine receptor defect, show slow swimming and provide a model of multi-minicore disease

Hiromi Hirata, J. Y. Kuwada, Weibin Zhou, 2007, Development.

Type-1 metabotropic glutamate receptor signaling in cerebellar Purkinje cells in health and disease

M. Kano, Takaki Watanabe, 2017, F1000Research.

Setd1a Insufficiency in Mice Attenuates Excitatory Synaptic Function and Recapitulates Schizophrenia-Related Behavioral Abnormalities.

M. Kano, Naofumi Uesaka, Y. Gotoh, 2020, Cell reports.

Ectopic positioning of Bergmann glia and impaired cerebellar wiring in Mlc1‐over‐expressing mice

Kenji F. Tanaka, Masahiko Watanabe, M. Kano, 2018, Journal of neurochemistry.

Coordinated Expression of Two Types of Low-Threshold K+ Channels Establishes Unique Single Spiking of Mauthner Cells among Segmentally Homologous Neurons in the Zebrafish Hindbrain

Y. Oda, Takaki Watanabe, Takashi Shimazaki, 2017, eNeuro.

Coexpression of auxiliary Kvβ2 subunits with Kv1.1 channels is required for developmental acquisition of unique firing properties of zebrafish Mauthner cells.

Hiromi Hirata, Y. Oda, M. Tanimoto, 2014, Journal of neurophysiology.

mGluR1 Is a Molecular “Hub” for Synapse Elimination in the Developing Cerebellum

M. Kano, Naofumi Uesaka, Takaki Watanabe, 2021, Contemporary Clinical Neuroscience.

Type-1 metabotropic glutamate receptor signaling in cerebellar Purkinje cells in health and disease

M. Kano, Takaki Watanabe, 2017, F1000Research.

Autism spectrum disorder-like behavior caused by reduced excitatory synaptic transmission in pyramidal neurons of mouse prefrontal cortex

Masahiko Watanabe, M. Kano, Naofumi Uesaka, 2020, Nature Communications.

A recurrent PJA1 variant in trigonocephaly and neurodevelopmental disorders

K. Yamakawa, M. Kano, T. Tatsukawa, 2020, Annals of clinical and translational neurology.

Combined glyoxalase 1 dysfunction and vitamin B6 deficiency in a schizophrenia model system causes mitochondrial dysfunction in the prefrontal cortex

M. Kano, M. Itokawa, G. Konopka, 2021, Redox biology.

Developmental synapse remodeling in the cerebellum and visual thalamus

M. Kano, Takaki Watanabe, 2019, F1000Research.

Multiple Phases of Climbing Fiber Synapse Elimination in the Developing Cerebellum

Masahiko Watanabe, M. Kano, Naofumi Uesaka, 2018, The Cerebellum.

Retrograde Signaling from Progranulin to Sort1 Counteracts Synapse Elimination in the Developing Cerebellum

Masahiko Watanabe, M. Kano, K. Sakimura, 2018, Neuron.

Multiple Phases of Climbing Fiber Synapse Elimination in the Developing Cerebellum

Masahiko Watanabe, M. Kano, Naofumi Uesaka, 2018, The Cerebellum.

PTPδ is a presynaptic organizer for the formation and maintenance of climbing fiber to Purkinje cell synapses in the developing cerebellum

Masahiko Watanabe, M. Kano, K. Hashimoto, 2023, Frontiers in Molecular Neuroscience.

Combining electrophysiology and optogenetics for functional screening of pyramidal neurons in the mouse prefrontal cortex

M. Kano, Naofumi Uesaka, Takaki Watanabe, 2021, STAR protocols.

Phospholipase C β3 is Required for Climbing Fiber Synapse Elimination in Aldolase C-positive Compartments of the Developing Mouse Cerebellum

M. Kano, K. Sakimura, Takaki Watanabe, 2020, Neuroscience.

Alternative splicing in the C-terminal tail of Cav2.1 is essential for preventing a neurological disease in mice

Masahiko Watanabe, M. Kano, T. Miyazaki, 2017, Journal of the Neurological Sciences.