I. Park

发表

Inhibition of the receptor tyrosine kinase Axl impedes activation of the FLT3 internal tandem duplication in human acute myeloid leukemia: implications for Axl as a potential therapeutic target.

M. Caligiuri, G. Marcucci, S. Whitman, 2013, Blood.

implications for Axl as a potential therapeutic target FLT3 internal tandem duplication in human acute myeloid leukemia: Inhibition of the receptor tyrosine kinase Axl impedes activation of the

G. Marcucci, S. Whitman, J. Chandler, 2013 .

TGF-β1 inhibition of IFN-γ-induced signaling and Th1 gene expression in CD4+ T cells is Smad3 independent but MAP kinase dependent

J. Letterio, J. Gorham, I. Park, 2007 .

CD94 surface density identifies a functional intermediary between the CD56bright and CD56dim human NK-cell subsets.

M. Caligiuri, G. Marcucci, R. Trotta, 2010, Blood.

Adverse Prognostic Impact of GAS6 Expression in De Novo Cytogenetically Normal Acute Myeloid Leukemia (CN-AML) (CALGB 8461, 9665, 20202; Alliance)

M. Caligiuri, C. Bloomfield, K. Metzeler, 2012 .

Reactivity of antibodies against 10-amino acid residue and pro-domain of stromelysin-3.

Y. Kook, E. Hwang, S. Hong, 1997, Cancer letters.

Receptor tyrosine kinase Axl is required for resistance of leukemic cells to FLT3-targeted therapy in acute myeloid leukemia

M. Caligiuri, G. Marcucci, D. Bearss, 2015, Leukemia.

The Axl/Gas6 pathway is required for optimal cytokine signaling during human natural killer cell development.

M. Caligiuri, R. Trotta, J. Yu, 2009, Blood.

Axl/Gas6 pathway positively regulates FLT3 activation in human natural killer cell development

M. Caligiuri, R. Trotta, Jianhua Yu, 2013, European journal of immunology.

TGF-beta 1 inhibition of IFN-gamma-induced signaling and Th1 gene expression in CD4+ T cells is Smad3 independent but MAP kinase dependent.

J. Letterio, J. Gorham, I. Park, 2007, Molecular immunology.

CD94 Defines Phenotypically and Functionally Distinct Mouse NK Cell Subsets1

M. Caligiuri, G. Marcucci, R. Trotta, 2009, The Journal of Immunology.

TGF-β1 Inhibits T-bet Induction by IFN-γ in Murine CD4+ T Cells through the Protein Tyrosine Phosphatase Src Homology Region 2 Domain-Containing Phosphatase-11

J. Letterio, L. Shultz, J. Gorham, 2005, The Journal of Immunology.

Necroinflammatory Liver Disease in BALB/c Background, TGF-β1-Deficient Mice Requires CD4+ T Cells1

J. Cates, E. M. Duncan, Jack T. Lin, 2003, The Journal of Immunology.

GAS6 expression identifies high-risk adult AML patients: potential implications for therapy

Klaus H. Metzeler, Guido Marcucci, Stefano Volinia, 2013, Leukemia.

E3 ubiquitin ligase Cbl-b activates the p53 pathway by targeting Siva1, a negative regulator of ARF, in FLT3 inhibitor-resistant acute myeloid leukemia

M. Caligiuri, I. Park, S. Baker, 2017, Leukemia.

Co‐expression of urokinase‐type plasminogen activator and its receptor in human gastric‐cancer cell lines correlates with their invasiveness and tumorigenicity

Y. Kook, Bum-Joon Kim, Chung-Gyu Park, 1997, International journal of cancer.

TGF-beta1 inhibits T-bet induction by IFN-gamma in murine CD4+ T cells through the protein tyrosine phosphatase Src homology region 2 domain-containing phosphatase-1.

J. Letterio, L. Shultz, J. Gorham, 2005, Journal of immunology.

Multiple Ubiquitin C-terminal Hydrolases from Chick Skeletal Muscle (*)

K. Tanaka, C. Chung, S. K. Woo, 1995, The Journal of Biological Chemistry.

Over‐expression of urokinase receptor in human epidermoid‐carcinoma cell line (HEp3) increases tumorigenicity on chorio‐allantoic membrane and in severe‐combined‐immunodeficient mice

Y. Kook, Bum-Joon Kim, B. Hyun, 1998, International journal of cancer.

Sp1 mediates constitutive and transforming growth factor beta-inducible expression of urokinase type plasminogen activator receptor gene in human monocyte-like U937 cells.

Y. Kook, I. Park, Taehee Han, 2000, Biochimica et biophysica acta.