A. Chaytor

发表

cAMP facilitates EDHF-type relaxations in conduit arteries by enhancing electrotonic conduction via gap junctions

D. Edwards, T. Griffith, A. Chaytor, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Distinct hyperpolarizing and relaxant roles for gap junctions and endothelium-derived H2O2 in NO-independent relaxations of rabbit arteries

D. Edwards, T. Griffith, L. Bakker, 2003, Proceedings of the National Academy of Sciences of the United States of America.

The obligatory link: role of gap junctional communication in endothelium-dependent smooth muscle hyperpolarization.

D. Edwards, T. Griffith, A. Chaytor, 2004, Pharmacological research.

Modulation of Gap Junction-Dependent Arterial Relaxation by Ascorbic Acid

D. Edwards, T. Griffith, L. Bakker, 2007, Journal of Vascular Research.

Connexin‐mimetic peptides dissociate electrotonic EDHF‐type signalling via myoendothelial and smooth muscle gap junctions in the rabbit iliac artery

D. Edwards, T. Griffith, L. Bakker, 2005, British journal of pharmacology.

Gap junctional communication underpins EDHF-type relaxations evoked by ACh in the rat hepatic artery.

D. Edwards, T. Griffith, P. Martin, 2001, American journal of physiology. Heart and circulatory physiology.

Gap junction-dependent increases in smooth muscle cAMP underpin the EDHF phenomenon in rabbit arteries.

D. Edwards, T. Griffith, A. Chaytor, 2001, Biochemical and biophysical research communications.

5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation.

D. Edwards, T. Griffith, L. Bakker, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Gap junction-dependent and -independent EDHF-type relaxations may involve smooth muscle cAMP accumulation.

T. Griffith, A. Chaytor, H. J. Taylor, 2002, American journal of physiology. Heart and circulatory physiology.

Comparison of glycyrrhetinic acid isoforms and carbenoxolone as inhibitors of EDHF-type relaxations mediated via gap junctions.

W. L. Marsh, T. Griffith, I. R. Hutcheson, 2000, Endothelium : journal of endothelial cell research.

Nitric oxide-independent relaxations to acetylcholine and A23187 involve different routes of heterocellular communication. Role of Gap junctions and phospholipase A2.

T. Griffith, W. H. Evans, I. R. Hutcheson, 1999, Circulation research.

Essential Role of Gap Junctions in NO- and Prostanoid-Independent Relaxations Evoked by Acetylcholine in Rabbit Intracerebral Arteries

H. Ujiié, T. Griffith, L. Bakker, 2003, Stroke.

The endothelial component of cannabinoid‐induced relaxation in rabbit mesenteric artery depends on gap junctional communication

T. Griffith, P. Martin, W. H. Evans, 1999, The Journal of physiology.

Peptides Homologous to Extracellular Loop Motifs of Connexin 43 Reversibly Abolish Rhythmic Contractile Activity in Rabbit Arteries

T. Griffith, W. H. Evans, K. Thornbury, 1997, The Journal of physiology.

A study to investigate the effectiveness of SimMan® as an adjunct in teaching preclinical skills to medical students

Marina Sawdon, John McLachlan, J. McLachlan, 2014, BMC medical education.

Teaching safe prescribing to medical students: perspectives in the UK

H. Nazar, C. Rothwell, J. Portlock, 2015, Advances in medical education and practice.

Report of the substance misuse in the undergraduate medical curriculum project in England

P. Kopelman, C. Notley, H. Ghodse, 2014 .

Central role of heterocellular gap junctional communication in endothelium‐dependent relaxations of rabbit arteries

T. Griffith, W. H. Evans, A. Chaytor, 1998, The Journal of physiology.

Inhibition of the gap junctional component of endothelium‐dependent relaxations in rabbit iliac artery by 18‐α glycyrrhetinic acid

T. Griffith, A. Chaytor, H. J. Taylor, 1998, British journal of pharmacology.

Substance misuse teaching in undergraduate medical education

P. Kopelman, C. Notley, H. Ghodse, 2014, BMC Medical Education.

Role of heterocellular Gap junctional communication in endothelium-dependent smooth muscle hyperpolarization: inhibition by a connexin-mimetic peptide.

C. Garland, T. Griffith, P. Martin, 1999, Biochemical and biophysical research communications.

PHOSPHORAMIDATE DERIVATIVES OF 2′,5′-DIDEOXYADENOSINE AS POTENTIAL INHIBITORS OF THE EDHF PHENOMENON

C. Mcguigan, T. Griffith, A. Chaytor, 2005, Nucleosides, Nucleotides & Nucleic Acids.

Nitric oxide-independent relaxations to acetylcholine and A23187 involve different routes of heterocellular communication. Role of Gap junctions and phospholipase A2.

T. Griffith, A. Chaytor, I. R. Hutcheson, 1999, Circulation research.

Routes of Heterocellular Communication : Role of Gap Junctions and Phospholipase A2 Independent Relaxations to Acetylcholine and A23187 Involve Different − Nitric Oxide

T. Griffith, A. Chaytor, I. R. Hutcheson, 2022 .

Advances in Medical Education and Practice Dovepress Teaching Safe Prescribing to Medical Students: Perspectives in the Uk

H. Nazar, C. Rothwell, J. Portlock, 2022 .

Enhanced inhibition of the EDHF phenomenon by a phenyl methoxyalaninyl phosphoramidate derivative of dideoxyadenosine

T. M. Griffith, C. Mcguigan, D. Edwards, 2004, British journal of pharmacology.