J. A. Enyeart

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Modulation of native T-type calcium channels by ω-3 fatty acids

Sanjay Danthi, Judith A. Enyeart, John J. Enyeart, 2005 .

T-type Ca2+ channels are required for adrenocorticotropin-stimulated cortisol production by bovine adrenal zona fasciculata cells.

B. Mlinar, J. A. Enyeart, J. Enyeart, 1993, Molecular endocrinology.

Modulation of native T-type calcium channels by omega-3 fatty acids.

Sanjay J Danthi, Judith A Enyeart, John J Enyeart, 2005, Biochemical and biophysical research communications.

N6-substituted cAMP Analogs Inhibit bTREK-1 K + Channels and Stimulate Cortisol Secretion by a PKA-Independent Mechanism

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2009 .

Mibefradil potently blocks ATP-activated K(+) channels in adrenal cells.

J. Gomora, J. A. Enyeart, J. Enyeart, 1999, Molecular pharmacology.

Metabolites of an Epac-Selective cAMP Analog Induce Cortisol Synthesis by Adrenocortical Cells through a cAMP-Independent Pathway

J. A. Enyeart, J. Enyeart, J. J. Enyeart, 2009, PloS one.

Adenosine Triphosphate Activates a Noninactivating K+ Current in Adrenal Cortical Cells through Nonhydrolytic Binding

J. Gomora, J. A. Enyeart, J. Enyeart, 1997, The Journal of general physiology.

ACTH and AII differentially stimulate steroid hormone orphan receptor mRNAs in adrenal cortical cells

R. Boyd, J. A. Enyeart, J. Enyeart, 1996, Molecular and Cellular Endocrinology.

8-Phenylthio-adenines stimulate the expression of steroid hydroxylases, Cav3.2 Ca²⁺ channels, and cortisol synthesis by a cAMP-independent mechanism.

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2011, American journal of physiology. Endocrinology and metabolism.

Curcumin inhibits bTREK-1 K+ channels and stimulates cortisol secretion from adrenocortical cells.

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2008, Biochemical and biophysical research communications.

Calcium-dependent inhibition of adrenal TREK-1 channels by angiotensin II and ionomycin.

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2011, American journal of physiology. Cell physiology.

Caffeic acid esters activate TREK-1 potassium channels and inhibit depolarization-dependent secretion.

S. Danthi, J. A. Enyeart, J. Enyeart, 2004, Molecular pharmacology.

Modulation of Native TREK-1 and Kv1.4 K+ Channels by Polyunsaturated Fatty Acids and Lysophospholipids

S. Danthi, J. A. Enyeart, J. Enyeart, 2003, The Journal of Membrane Biology.

Adrenal fasciculata cells express T-type and rapidly and slowly activating L-type Ca2+ channels that regulate cortisol secretion.

J. A. Enyeart, J. Enyeart, 2015, American journal of physiology. Cell physiology.

Ca2+ and K+ channels of normal human adrenal zona fasciculata cells: Properties and modulation by ACTH and AngII

J. A. Enyeart, J. Enyeart, J. J. Enyeart, 2013, The Journal of general physiology.

Potent Inhibition of Native TREK-1 K+ Channels by Selected Dihydropyridine Ca2+ Channel Antagonists

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2007, Journal of Pharmacology and Experimental Therapeutics.

Angiotensin II Inhibits bTREK-1 K+ Channels in Adrenocortical Cells by Separate Ca2+- and ATP Hydrolysis-dependent Mechanisms*

S. Danthi, J. A. Enyeart, J. Enyeart, 2005, Journal of Biological Chemistry.

Human Adrenal Glomerulosa Cells Express K2P and GIRK Potassium Channels that are inhibited by AngII and ACTH.

J. A. Enyeart, J. Enyeart, J. J. Enyeart, 2021, American journal of physiology. Cell physiology.

ACTH Induces Cav3.2 Current and mRNA by cAMP-dependent and cAMP-independent Mechanisms*

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2010, The Journal of Biological Chemistry.

cAMP Analogs and Their Metabolites Enhance TREK-1 mRNA and K+ Current Expression in Adrenocortical Cells

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2010, Molecular Pharmacology.

Microsoft Word-17505235-file00

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2009 .

Angiotensin II inhibits native bTREK-1 K+ channels through a PLC-, kinase C-, and PIP2-independent pathway requiring ATP hydrolysis.

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2007, American journal of physiology. Cell physiology.

Adrenocorticotropic hormone and cAMP inhibit noninactivating K+ current in adrenocortical cells by an A-kinase-independent mechanism requiring ATP hydrolysis

B. Mlinar, J. A. Enyeart, J. Enyeart, 1996, The Journal of general physiology.

TREK-1 K+ channels couple angiotensin II receptors to membrane depolarization and aldosterone secretion in bovine adrenal glomerulosa cells.

S. Danthi, J. A. Enyeart, J. Enyeart, 2004, American journal of physiology. Endocrinology and metabolism.

Ca2+ channel modulation and kinase-C activation in a pituitary cell line: induction of immediate early genes and inhibition of proliferation.

A. Duchemin, D. Foster, B. Mlinar, 1992, Molecular endocrinology.

An ACTH- and ATP-regulated Background K+ Channel in Adrenocortical Cells Is TREK-1*

S. Danthi, J. A. Enyeart, J. Enyeart, 2002, The Journal of Biological Chemistry.

ACTH Inhibits bTREK-1 K+ Channels through Multiple cAMP-dependent Signaling Pathways

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2008, The Journal of general physiology.

Dual actions of lanthanides on ACTH-inhibited leak K(+) channels.

J. A. Enyeart, J. Enyeart, Lin Xu, 2002, American journal of physiology. Endocrinology and metabolism.

Activation of separate calcium and A-kinase-dependent pathways by ACTH.

J. A. Enyeart, J. Enyeart, 1998, Endocrine research.

Evidence for cAMP-independent bTREK-1 inhibition by ACTH and NPS-ACTH in adrenocortical cells

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2012, Molecular and Cellular Endocrinology.

N6-Substituted cAMP Analogs Inhibit bTREK-1 K+ Channels and Stimulate Cortisol Secretion by a Protein Kinase A-Independent Mechanism

J. A. Enyeart, J. Enyeart, Haiyan Liu, 2009, Molecular Pharmacology.

Corticotropin induces the expression of TREK-1 mRNA and K+ current in adrenocortical cells.

S. Danthi, J. A. Enyeart, J. J. Enyeart, 2003, Molecular pharmacology.

Adrenal fasciculata cells express T-type and rapidly and slowly activating L-type Ca2+ channels that regulate cortisol secretion.

J. A. Enyeart, J. J. Enyeart, 2015, American journal of physiology. Cell physiology.

A Bovine Adrenocortical Kv1.4 K+ Channel Whose Expression Is Potently Inhibited by ACTH*

J. A. Enyeart, Lin Xu, J. J. Enyeart, 2000, The Journal of Biological Chemistry.

Binding of p-nitrophenyl phosphate and other aromatic compounds by beta-lactoglobulin.

M. Behe, H. M. Farrell, J. Enyeart, 1987, Journal of dairy science.

Neuroprotective agent riluzole dramatically slows inactivation of Kv1.4 potassium channels by a voltage-dependent oxidative mechanism.

L. Xu, J. A. Enyeart, J J Enyeart, 2001, The Journal of pharmacology and experimental therapeutics.

Reciprocal modulation of voltage-gated and background K(+) channels mediated by nucleotides and corticotropin.

L. Xu, J. Gomora, J. A. Enyeart, 2001, Molecular pharmacology.

Modulation of IA Potassium Current in Adrenal Cortical Cells by a Series of Ten Lanthanide Elements

L. Xu, J. Gomora, J. A. Enyeart, 1998, The Journal of Membrane Biology.

N6-substituted cAMP Analogs Inhibit bTREK-1 K + Channels and Stimulate Cortisol Secretion by a PKA-Independent Mechanism

J. A. Enyeart, Haiyan Liu, J. J. Enyeart, 2009 .

Effect of mibefradil on voltage-dependent gating and kinetics of T-type Ca(2+) channels in cortisol-secreting cells.

L. Xu, J. Gomora, J. A. Enyeart, 2000, The Journal of pharmacology and experimental therapeutics.