S. Lattimer

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Primary preventive and secondary interventionary effects of acetyl-L-carnitine on diabetic neuropathy in the bio-breeding Worcester rat.

D. Greene, M. Stevens, A. Sima, 1996, The Journal of clinical investigation.

Complications: Neuropathy, Pathogenetic Considerations

E. Feldman, D. Greene, M. Stevens, 1992, Diabetes Care.

Axo-glial dysjunction. A novel structural lesion that accounts for poorly reversible slowing of nerve conduction in the spontaneously diabetic bio-breeding rat.

S. Yagihashi, D. Greene, A. Sima, 1986, The Journal of clinical investigation.

Sorbitol and myo‐inositol levels and morphology of sural nerve in relation to peripheral nerve function and clinical neuropathy in men with diabetic, impaired, and normal glucose tolerance

G. Sundkvist, D. Greene, A. Sima, 2000, Diabetic medicine : a journal of the British Diabetic Association.

In vitro correction of impaired Na+-K+-ATPase in diabetic nerve by protein kinase C agonists.

D. Greene, A. Sima, S. Lattimer, 1989, The American journal of physiology.

Regeneration and repair of myelinated fibers in sural-nerve biopsy specimens from patients with diabetic neuropathy treated with sorbinil.

M. B. Brown, D. Greene, V. Bril, 1988, The New England journal of medicine.

Nerve Na+-K+-ATPase, conduction, and myo-inositol in the insulin-deficient BB rat.

S. Yagihashi, D. Greene, A. Sima, 1984, The American journal of physiology.

Impaired energy utilization and Na-K-ATPase in diabetic peripheral nerve.

D. Greene, S. Lattimer, 1984, The American journal of physiology.

Impaired rat sciatic nerve sodium-potassium adenosine triphosphatase in acute streptozocin diabetes and its correction by dietary myo-inositol supplementation.

D. Greene, S. Lattimer, 1983, The Journal of clinical investigation.

Role of sorbitol accumulation and myo-inositol depletion in paranodal swelling of large myelinated nerve fibers in the insulin-deficient spontaneously diabetic bio-breeding rat. Reversal by insulin replacement, an aldose reductase inhibitor, and myo-inositol.

D. Greene, A. Sima, S. Lattimer, 1987, The Journal of clinical investigation.

Action of Sorbinil in Diabetic Peripheral Nerve: Relationship of Polyol (Sorbitol) Pathway Inhibition to a myo-Inositol-mediated Defect in Sodium-Potassium ATPase Activity

D. Greene, S. Lattimer, 1984, Diabetes.

Polyol Pathway Activity and Myo-Inositol Metabolism: A Suggested Relationship in the Pathogenesis of Diabetic Neuropathy

D. Finegold, D. Greene, S. Lattimer, 1983, Diabetes.

Inhibition of phosphatidylinositol synthase by glucose in human retinal pigment epithelial cells.

D. Greene, J. Nakamura, D. Shewach, 1992, The American journal of physiology.

A defect in sodium-dependent amino acid uptake in diabetic rabbit peripheral nerve. Correction by an aldose reductase inhibitor or myo-inositol administration.

D. Finegold, D. Greene, J. Fernstrom, 1990, The Journal of clinical investigation.

Osmotically-induced nerve taurine depletion and the compatible osmolyte hypothesis in experimental diabetic neuropathy in the rat

D. Greene, M. Stevens, A. Sima, 1993, Diabetologia.

Recent advances in the therapy of diabetic peripheral neuropathy by means of an aldose reductase inhibitor.

D. Greene, S. Lattimer, 1985, The American journal of medicine.

The Polyol Pathway in Dysfunction of Diabetic Peripheral Nerve

D. Greene, S. Lattimer, 1985, Diabetic medicine : a journal of the British Diabetic Association.

Altered Nerve Myo-Inositol Metabolism in Experimental Diabetes and Its Relationship to Nerve Function

D. Greene, S. Lattimer, 1985 .

Sodium- and energy-dependent uptake of myo-inositol by rabbit peripheral nerve. Competitive inhibition by glucose and lack of an insulin effect.

D. Greene, S. Lattimer, 1982, The Journal of clinical investigation.

Reduced Motor Nerve Conduction Velocity and Na+-K+-ATPase Activity in Rats Maintained on L-Fucose Diet: Reversal by myo-Inositol Supplementation

J. Dunlap, E. Davidson, M. Yorek, 1993, Diabetes.

Sorbitol, phosphoinositides, and sodium-potassium-ATPase in the pathogenesis of diabetic complications.

D. Greene, A. Sima, S. Lattimer, 1987, The New England journal of medicine.

Sorbitol, myo-Inositol, and Rod Outer Segment Phagocytosis in Cultured hRPE Cells Exposed to Glucose: In Vitro Model of myo-Inositol Depletion Hypothesis of Diabetic Complications

D. Greene, J. Nakamura, R. Rabbani, 1991, Diabetes.

Pathogenesis and prevention of diabetic neuropathy.

D. Greene, A. Sima, S. Lattimer, 1988, Diabetes/metabolism reviews.

Altered Sorbitol and myo‐Inositol Metabolism as the Basis for Defective Protein Kinase C and (Na,K)‐ATPase Regulation in Diabetic Neuropathy a

D. Greene, S. Lattimer, 1986, Annals of the New York Academy of Sciences.

Glucose-induced Alterations in Nerve Metabolism: Current Perspective on the Pathogenesis of Diabetic Neuropathy and Future Directions for Research and Therapy

J. Ulbrecht, D. Greene, S. Lattimer, 1985, Diabetes Care.

Aldose reductase inhibitors: an approach to the treatment of diabetic nerve damage.

E. Feldman, D. Greene, M. Stevens, 1993, Diabetes/metabolism reviews.

Acetyl-L-carnitine deficiency as a cause of altered nerve myo-inositol content, Na,K-ATPase activity, and motor conduction velocity in the streptozotocin-diabetic rat.

E. Feldman, D. Millington, D. Greene, 1996, Metabolism: clinical and experimental.

The linked roles of nitric oxide, aldose reductase and, (Na+,K+)-ATPase in the slowing of nerve conduction in the streptozotocin diabetic rat.

E. Feldman, D. Greene, M. Stevens, 1994, The Journal of clinical investigation.

Are Disturbances of Sorbitol, Phosphoinositide, and Na+-K+-ATPase Regulation Involved in Pathogenesis of Diabetic Neuropathy?

D. Greene, A. Sima, S. Lattimer, 1988, Diabetes.

Selective Effects of Myo-inositol Administration on Sciatic and Tibial Motor Nerve Conduction Parameters in the Streptozocin-Diabetic Rat

D. Greene, M. J. Brown, S. Lattimer, 1982, Diabetes.

35th Annual meeting of the European Association for the Study of Diabetes. Brussels, Belgium, 28 September-2 October, 1999. Abstracts.

Thomas J. Smith, B. M. Rasmussen, V. C. Phan, 1999, Diabetologia.

Protein Kinase C Agonists Acutely Normalize Decreased Ouabain-inhibitable Respiration in Diabetic Rabbit Nerve: Implications for (Na,K)-ATPase Regulation and Diabetic Complications

D. Greene, S. Lattimer, 1986, Diabetes.

Transphosphatidylation of sugar alcohols and its implications for the pathogenesis of diabetic complications

D. Greene, J. Nakamura, S. Lattimer, 1994, Diabetologia.