M. Said

发表

The importance of the Thr 17 residue of phospholamban as a phosphorylation site under physiological and pathological conditions

E. Kranias, A. Mattiazzi, L. Vittone, 2006 .

The importance of the Thr17 residue of phospholamban as a phosphorylation site under physiological and pathological conditions.

E. Kranias, A. Mattiazzi, L. Vittone, 2006, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.

Role of phosphorylation of Thr(17) residue of phospholamban in mechanical recovery during hypercapnic acidosis.

E. Kranias, A. Mattiazzi, P. Ferrero, 2005, Cardiovascular research.

Calcium-calmodulin dependent protein kinase II (CaMKII): a main signal responsible for early reperfusion arrhythmias.

X. Wehrens, M. Kaetzel, J. Dedman, 2011, Journal of molecular and cellular cardiology.

Chemical chaperones improve the functional recovery of stunned myocardium by attenuating the endoplasmic reticulum stress

J. I. Mariángelo, L. Vittone, M. Said, 2020, Acta physiologica.

Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart.

X. Wehrens, P. Donoso, G. Schinella, 2016, American journal of physiology. Heart and circulatory physiology.

Increased intracellular Ca2+ and SR Ca2+ load contribute to arrhythmias after acidosis in rat heart. Role of Ca2+/calmodulin-dependent protein kinase II.

G. Rinaldi, M. Kaetzel, J. Dedman, 2008, American journal of physiology. Heart and circulatory physiology.

Endoplasmic reticulum contribution to the relaxant effect of cGMP- and cAMP-elevating agents in feline aorta.

G. Rinaldi, A. Mattiazzi, L. Vittone, 2000, American journal of physiology. Heart and circulatory physiology.

CaMKII inhibition protects against necrosis and apoptosis in irreversible ischemia-reperfusion injury.

R. Hajjar, E. Kranias, E. Portiansky, 2007, Cardiovascular research.

Role of dual-site phospholamban phosphorylation in the stunned heart: insights from phospholamban site-specific mutants.

E. Kranias, A. Mattiazzi, P. Ferrero, 2003, American journal of physiology. Heart and circulatory physiology.

Phospholamban phosphorylation in ischemia-reperfused heart. Effect of pacing during ischemia and response to a β-adrenergic challenge

A. Mattiazzi, P. Ferrero, L. Vittone, 2003, Molecular and Cellular Biochemistry.

β2-Adrenergic stimulation is involved in the contractile dysfunction of the stunned heart

A. Mattiazzi, L. Vittone, M. Said, 2006, Naunyn-Schmiedeberg's Archives of Pharmacology.

Phospholamban Phosphorylation during Ischemia and Reperfusion Injury: Mechanisms and Functional Implications

A. Mattiazzi, P. Ferrero, L. Vittone, 2003 .

Time course and mechanisms of phosphorylation of phospholamban residues in ischemia-reperfused rat hearts. Dissociation of phospholamban phosphorylation pathways.

A. Mattiazzi, P. Ferrero, L. Vittone, 2002, Journal of molecular and cellular cardiology.

The relative relevance of phosphorylation of the Thr17 residue of phospholamban is different at different levels of β-adrenergic stimulation

A. Mattiazzi, L. Vittone, M. Said, 2002, Pflügers Archiv.

The signalling pathway of CaMKII-mediated apoptosis and necrosis in the ischemia/reperfusion injury.

E. Kranias, E. Portiansky, M. Kaetzel, 2010, Journal of molecular and cellular cardiology.

Phosphorylation of the Thr17 Residue of Phospholamban: New Insights into the Physiological Role of the CaMK‐II Pathway of Phospholamban Phosphorylation

A. Mattiazzi, L. Vittone, M. Said, 1998, Annals of the New York Academy of Sciences.

Dilated cardiomyopathy in Erb-b4-deficient ventricular muscle.

Julian Taranda, Cary Lai, Ju Chen, 2005, American journal of physiology. Heart and circulatory physiology.

Pharmacological inhibition of translocon is sufficient to alleviate endoplasmic reticulum stress and improve Ca2+ handling and contractile recovery of stunned myocardium.

J. I. Mariángelo, L. Vittone, M. Said, 2021, European journal of pharmacology.

Mechanisms Involved in the Acidosis Enhancement of the Isoproterenol-induced Phosphorylation of Phospholamban in the Intact Heart*

A. Mattiazzi, L. Vittone, M. Said, 1998, The Journal of Biological Chemistry.

CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia/reperfusion injury.

X. Wehrens, J. Brown, G. Rinaldi, 2014, Journal of molecular and cellular cardiology.

Frequency‐dependent acceleration of relaxation in mammalian heart: a property not relying on phospholamban and SERCA2a phosphorylation

A. Mattiazzi, P. Ferrero, J. Palomeque, 2005, The Journal of physiology.

Early effects of Epac depend on the fine-tuning of the sarcoplasmic reticulum Ca2+ handling in cardiomyocytes.

X. Wehrens, J. I. Mariángelo, L. Vittone, 2018, Journal of molecular and cellular cardiology.

Ca2+/calmodulin kinase II increases ryanodine binding and Ca2+-induced sarcoplasmic reticulum Ca2+ release kinetics during beta-adrenergic stimulation.

P. Donoso, A. Mattiazzi, P. Ferrero, 2007, Journal of molecular and cellular cardiology.

Increased intracellular Ca 2 (cid:1) and SR Ca 2 (cid:1) load contribute to arrhythmias after acidosis in rat heart. Role of Ca 2 (cid:1) /calmodulin-dependent protein kinase II

G. Rinaldi, M. Kaetzel, J. Dedman, 2008 .

induced sarcoplasmic reticulum Ca 2+ release kinetics during β- adrenergic stimulation

A. Mattiazzi, P. Ferrero, G. Sánchez, 2008 .

Increased Na+/Ca2+ Exchanger Expression/Activity Constitutes a Point of Inflection in the Progression to Heart Failure of Hypertensive Rats

E. Portiansky, G. Rinaldi, A. Mattiazzi, 2014, PloS one.

Stimulation of NOX2 in isolated hearts reversibly sensitizes RyR2 channels to activation by cytoplasmic calcium.

R. Bull, P. Donoso, G. Sánchez, 2014, Journal of molecular and cellular cardiology.

Role of CaMKII in post acidosis arrhythmias: a simulation study using a human myocyte model.

Jorge A Negroni, Elena C Lascano, Alicia Mattiazzi, 2013, Journal of molecular and cellular cardiology.

Doxorubicin and NRG-1/erbB4-Deficiency Affect Gene Expression Profile: Involving Protein Homeostasis in Mouse

H. García-Rivello, C. Hertig, H. Witt, 2012, ISRN cardiology.

Prolonged Ca2+ release refractoriness and T‐tubule disruption as determinants of increased propensity to cardiac alternans in the hypertensive heart disease

J. I. Mariángelo, M. Said, C. Mundiña-Weilenmann, 2023, Acta physiologica.

High-Fat-Diet-Induced Obesity Produces Spontaneous Ventricular Arrhythmias and Increases the Activity of Ryanodine Receptors in Mice

R. Bull, P. Donoso, Z. Pedrozo, 2018, International journal of molecular sciences.

Stimulation of NOX2 in isolated hearts reversibly sensitizes RyR2 channels to activation by cytoplasmic calcium.

R. Bull, P. Donoso, G. Sánchez, 2014, Journal of molecular and cellular cardiology.

Title CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia / reperfusion injury

X. Wehrens, J. Brown, G. Rinaldi, 2014 .

Phosphorylation of phospholamban in ischemia-reperfusion injury: Functional role of Thr17 residue

A. Mattiazzi, L. Vittone, M. Said, 2004, Molecular and Cellular Biochemistry.

Immunodetection of Phosphorylation Sites of Phospholamban in Aortic Smooth Muscle Effects of Sodium Nitroprusside

G. Rinaldi, A. Mattiazzi, L. Vittone, 1998, Annals of the New York Academy of Sciences.