M. T. Miller

ATP depletion induces an increase in the assembly of a labile pool of polymerized actin in endothelial cells.

J. M. Burger, D. Hinshaw, J. Adams, 1993, The American journal of physiology.

Differential expression of renal nitric oxide synthase isoforms during pregnancy in rats.

J. Granger, W. C. Kruckeberg, B. Alexander, 1999, Hypertension.

Reduced Uterine Perfusion Pressure During Pregnancy in the Rat Is Associated With Increases in Arterial Pressure and Changes in Renal Nitric Oxide

S. Abram, J. Granger, B. Alexander, 2001, Hypertension.

Endothelin-A receptor antagonism attenuates the hypertension and renal injury in Dahl salt-sensitive rats.

J. Granger, J. Reckelhoff, B. Clower, 1998, Hypertension.

Systemic hemodynamics and regional blood flow during chronic nitric oxide synthesis inhibition in pregnant rats.

R. Hester, J. Granger, S. Kassab, 1998, Hypertension.

Angiotensin II stimulates synthesis of endothelial nitric oxide synthase.

J. Granger, J. Reckelhoff, B. S. Hennington, 1998, Hypertension.

Induction of anchorage independent growth by heparin-binding EGF-like growth factor (HB-EGF).

P. A. Harding, G. Besner, D. Brigstock, 1999, Growth factors.

Endothelin-A receptor antagonism attenuates the hypertension and renal injury in Dahl salt-sensitive rats.

J. Reckelhoff, S. Kassab, M. T. Miller, 1998, Hypertension.

Role of nitric oxide in modulating renal function and arterial pressure during chronic aldosterone excess.

J. Granger, J. Reckelhoff, S. Kassab, 1999, American journal of physiology. Regulatory, integrative and comparative physiology.

Role of nitric oxide in modulating renal function and arterial pressure during chronic aldosterone excess.

J. Reckelhoff, S. Kassab, B. Tucker, 1999, The American journal of physiology.