Hiroyuki Ikeda

发表

Corrigendum: IGFBP-4 is an inhibitor of canonical Wnt signalling required for cardiogenesis

I. Komuro, I. Shiojima, Yuzuru Ito, 2014, Nature.

IGFBP-4 is an inhibitor of canonical Wnt signalling required for cardiogenesis

I. Komuro, I. Shiojima, Yuzuru Ito, 2008, Nature.

Excessive cardiac insulin signaling exacerbates systolic dysfunction induced by pressure overload in rodents.

C. Kahn, I. Komuro, I. Shiojima, 2010, The Journal of clinical investigation.

Chronic doxorubicin cardiotoxicity is mediated by oxidative DNA damage-ATM-p53-apoptosis pathway and attenuated by pitavastatin through the inhibition of Rac1 activity.

I. Komuro, I. Shiojima, Masashi Yoshida, 2009, Journal of molecular and cellular cardiology.

Increased Akt-mTOR Signaling in Lung Epithelium Is Associated with Respiratory Distress Syndrome in Mice

I. Komuro, I. Shiojima, K. Walsh, 2010, Molecular and Cellular Biology.

Interaction of myocardial insulin receptor and IGF receptor signaling in exercise-induced cardiac hypertrophy.

C. Kahn, I. Komuro, I. Shiojima, 2009, Journal of molecular and cellular cardiology.

Excessive cardiac insulin signaling exacerbates systolic dysfunction induced by pressure overload in rodents.

I. Komuro, I. Shiojima, T. Minamino, 2010, The Journal of clinical investigation.

Increased Akt-mTOR Signaling in Lung Epithelium Is Associated with Respiratory Distress Syndrome in Mice

I. Komuro, I. Shiojima, K. Walsh, 2010, Molecular and Cellular Biology.

Interaction of myocardial insulin receptor and IGF receptor signaling in exercise-induced cardiac hypertrophy.

C. Kahn, K. Walsh, M. Holzenberger, 2009, Journal of molecular and cellular cardiology.