Sangyeul Han

发表

Phosphorylation of Tuberin as a Novel Mechanism for Somatic Inactivation of the Tuberous Sclerosis Complex Proteins in Brain Lesions

Túlio Marcos Santos, A. Stemmer-Rachamimov, E. Thiele, 2004, Cancer Research.

Survival benefit and phenotypic improvement by hamartin gene therapy in a tuberous sclerosis mouse brain model

A. Stemmer-Rachamimov, X. Breakefield, R. Bronson, 2015, Neurobiology of Disease.

Aberrant Hyperactivation of Akt and Mammalian Target of Rapamycin Complex 1 Signaling in Sporadic Chordomas

G. Nielsen, A. Rosenberg, V. Ramesh, 2009, Clinical Cancer Research.

Amelioration of sepsis by TIE2 activation–induced vascular protection

Sung Hyun Kim, Junhao Hu, H. Augustin, 2016, Science Translational Medicine.

Regulation of mTOR Complex 2 Signaling in Neurofibromatosis 2–Deficient Target Cell Types

J. Gusella, A. Stemmer-Rachamimov, Hua Li, 2012, Molecular Cancer Research.

The E3 Ubiquitin Ligase Protein Associated with Myc (Pam) Regulates Mammalian/Mechanistic Target of Rapamycin Complex 1 (mTORC1) Signaling in Vivo through N- and C-terminal Domains*

P. Bhide, M. James, S. Bahl, 2012, The Journal of Biological Chemistry.

Calmodulin Mediates Ca2+-Dependent Inhibition of Tie2 Signaling and Acts as a Developmental Brake During Embryonic Angiogenesis

Hosung Jung, Ji Yeun Lee, Chungho Kim, 2016, Arteriosclerosis, thrombosis, and vascular biology.

Signaling and Regulation Regulation of mTOR Complex 2 Signaling in Neuro fi bromatosis 2 – De fi cient Target Cell Types

J. Gusella, A. Stemmer-Rachamimov, Hua Li, 2012 .

Reversal of learning deficits in a Tsc2+/− mouse model of tuberous sclerosis

Alcino J. Silva, D. Kwiatkowski, V. Ramesh, 2008, Nature Medicine.

NF2/Merlin Is a Novel Negative Regulator of mTOR Complex 1, and Activation of mTORC1 Is Associated with Meningioma and Schwannoma Growth

J. Gusella, A. Stemmer-Rachamimov, B. Manning, 2009, Molecular and Cellular Biology.

Normalization of Tumor Vessels by Tie2 Activation and Ang2 Inhibition Enhances Drug Delivery and Produces a Favorable Tumor Microenvironment.

Doheon Lee, H. Augustin, G. Koh, 2016, Cancer cell.

Pam (Protein associated with Myc) functions as an E3 ubiquitin ligase and regulates TSC/mTOR signaling.

Rochelle M. Witt, B. Sabatini, Túlio Marcos Santos, 2008, Cellular signalling.

Pam and Its Ortholog Highwire Interact with and May Negatively Regulate the TSC1·TSC2 Complex*

R. Beauchamp, L. A. Haddad, N. Smith, 2004, Journal of Biological Chemistry.

Alternative splicing in protein associated with Myc (Pam) influences its binding to c‐Myc

Mark J. Bowser, Túlio Marcos Santos, P. Bhide, 2006, Journal of neuroscience research.

Tie2 activation promotes choriocapillary regeneration for alleviating neovascular age-related macular degeneration

H. Augustin, Pilhan Kim, Y. Kubota, 2019, Science Advances.

Erratum: Normalization of Tumor Vessels by Tie2 Activation and Ang2 Inhibition Enhances Drug Delivery and Produces a Favorable Tumor Microenvironment (Cancer Cell (2016) 30(6) (953–967) (S1535610816305050) (10.1016/j.ccell.2016.10.018))

Doheon Lee, H. Augustin, G. Koh, 2017 .

Gln-362 of Angiopoietin-2 Mediates Migration of Tumor and Endothelial Cells through Association with α5β1 Integrin

Hyung-chan Kim, Chungho Kim, H. Lee, 2014, The Journal of Biological Chemistry.

Amelioration of sepsis by TIE2 activation–induced vascular protection

H. Augustin, Pilhan Kim, Hyung-chan Kim, 2016, Science Translational Medicine.

Aberrant Hyperactivation of Akt and Mammalian Target of Rapamycin Complex 1 Signaling in Sporadic Chordomas

G. Nielsen, A. Rosenberg, V. Ramesh, 2009, Clinical Cancer Research.

Survival benefit and phenotypic improvement by hamartin gene therapy in a tuberous sclerosis mouse brain model

A. Stemmer-Rachamimov, X. Breakefield, R. Bronson, 2015, Neurobiology of Disease.