Judy Correa

发表

Cellular markers of muscle atrophy in chronic obstructive pulmonary disease.

D. Brooks, J. Batt, M. Binnie, 2010, American journal of respiratory cell and molecular biology.

Absence of caspase-3 protects against denervation-induced skeletal muscle atrophy.

M. Woo, J. Batt, James R Bain, 2009, Journal of applied physiology.

Skeletal muscle dysfunction in idiopathic pulmonary arterial hypertension.

J. Granton, J. Batt, Judy Correa, 2013, American journal of respiratory cell and molecular biology.

Evidence That the NH2 Terminus of Vph1p, an Integral Subunit of the V0 Sector of the Yeast V-ATPase, Interacts Directly with the Vma1p and Vma13p Subunits of the V1Sector*

M. Manolson, C. Landolt-Marticorena, Judy Correa, 2000, The Journal of Biological Chemistry.

The inositol phosphatase MTMR4 is a novel target of the ubiquitin ligase Nedd4

J. Batt, N. Goldenberg, James R Bain, 2009, The Biochemical journal.

Mechanisms of Chronic Muscle Wasting and Dysfunction after an Intensive Care Unit Stay. A Pilot Study.

G. Tomlinson, C. Streutker, A. Advani, 2016, American journal of respiratory and critical care medicine.

The Ubiquitin Ligase Nedd4-1 Participates in Denervation-Induced Skeletal Muscle Atrophy in Mice

D. Rotin, J. Batt, H. Kawabe, 2012, PloS one.

PDLIM7 is a novel target of the ubiquitin ligase Nedd4-1 in skeletal muscle.

J. Batt, P. Plant, Judy Correa, 2016, The Biochemical journal.

MicroRNA regulatory networks associated with abnormal muscle repair in survivors of critical illness

P. Hu, A. Bigot, S. Mathur, 2022, Journal of cachexia, sarcopenia and muscle.

Substrate- and Inhibitor-induced Conformational Changes in the Yeast V-ATPase Provide Evidence for Communication between the Catalytic and Proton-translocating Sectors*

M. Manolson, W. Kahr, C. Landolt-Marticorena, 1999, The Journal of Biological Chemistry.

Hypertrophic airway smooth muscle mass correlates with increased airway responsiveness in a murine model of asthma.

J. Batt, A. Ward, M. North, 2012, American journal of respiratory cell and molecular biology.

WW domains of Nedd4 bind to the proline‐rich PY motifs in the epithelial Na+ channel deleted in Liddle's syndrome.

O. Staub, D. Rotin, J. McGlade, 1996, The EMBO journal.