D. Mash

MAPT H1 haplotype is a risk factor for essential tremor and multiple system atrophy

M. Farrer, D. Mash, K. Lyons, 2011, Neurology.

Neuropathological and neuropsychological changes in "normal" aging: evidence for preclinical Alzheimer disease in cognitively normal individuals.

D. Mash, K. Welsh-Bohmer, A. Saunders, 1998, Journal of neuropathology and experimental neurology.

Cyanobacterial neurotoxin BMAA in ALS and Alzheimer’s disease

D. Mash, S. Papapetropoulos, P. Cox, 2009, Acta neurologica Scandinavica.

Can Alzheimer's type pathology influence the clinical phenotype of Parkinson's disease?

S. Papapetropoulos, A. Lieberman, D. Mash, 2005, Acta neurologica Scandinavica.

Catechols in post‐mortem brain of patients with Parkinson disease

D. Goldstein, I. Kopin, C. Holmes, 2011, European journal of neurology.

Dopamine transporter‐immunoreactive neurons decrease with age in the human substantia nigra

D. Mash, G. Stebbins, E. Mufson, 1999, The Journal of comparative neurology.

Dementia in Parkinson's disease: a post‐mortem study in a population of brain donors

D. Mash, S. Papapetropoulos, S. Papapetropoulos, 2005, International journal of geriatric psychiatry.

Intraneuronal ApoE in human visual cortical areas reflects the staging of Alzheimer disease pathology.

A. Roses, A. Saunders, C. Hulette, 1998, Journal of neuropathology and experimental neurology.

LOSS OF HIGH-AFFINITY AGONIST RECEPTOR BINDING IN ALZHEIMER'S DISEASE. AUTHORS' REPLY

J. Keyser, D. Flynn, D. Mash, 1992 .